Hand and Wrist Injuries

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89 Hand and Wrist Injuries

Epidemiology

Annually, more than 16 million people suffer some form of hand injury, and more than 4.8 million will seek treatment of these injuries at the emergency department (ED).1 Traumatic injuries may include lacerations, fractures, and tendon or ligamentous injuries. Most injuries are readily identified in the ED, provided that the assessment is effective.2 Because the morbidity associated with undiagnosed, misdiagnosed, or mistreated hand injuries is significant, a high level of expertise is required of the EP.

Presenting Signs and Symptoms

When assessing hand injuries, the history should focus on the mechanism of the injury, hand position at the time of the insult, perceived resultant functional impairment, and the time elapsed since the injury. Some mechanisms yield classic injury patterns, such as the jersey finger and mallet finger. Other injury patterns are known for their poor outcomes, such as fight bites or high-pressure injection injuries, and require specific managements. Some wounds are more prone to infection, such as crush injuries and grossly contaminated wounds.

During the history the patient’s hand dominance and career should also be ascertained and documented. Factors that may compromise wound healing, such as smoking, drug use, or an immunocompromised state, are important to document.2 Tetanus status should be verified.

Despite its complicated nature, the hand can be examined adequately in a short period. Developing a rapid, reproducible hand examination strategy and performing it regularly will decrease the chance of missing subtle injuries. Even when a specific injury is obvious, it is important to examine the entire hand to avoid overlooking less obvious, coincident injuries. Box 89.1 lists one approach to comprehensive assessment of the hand.

Assuming that no active bleeding is occurring and requires immediate attention, examination of the hand begins with inspection. All rings, watches, and other potentially constricting devices should be removed immediately.3 Lacerations and other disruptions in skin integrity are usually recognized easily; erythema, soft tissue swelling, and ecchymoses should also be noted. It is important to compare the general position of the hand with that of the unaffected side inasmuch as many fractures or tendon disruptions will cause characteristic deformities that are recognizable on inspection.

Vascular integrity should be determined by comparing skin temperature with that of the opposite hand, feeling for ulnar and radial pulses, and documenting intact and symmetric distal capillary refill.

Neurologic testing should be performed before local or regional anesthesia. Radial, median, and ulnar nerve function should be individually assessed and the digital nerves interrogated via both light touch and two-point discrimination. Comparison with the unaffected side can be useful. To evaluate for radial neuropathy, the dorsal aspect of the second and third web space is tested for decreased sensation. Proximal limb radial nerve lesions will cause wristdrop. However, the superficial radial nerve, as it courses through the hand, is sensory only. To test for median neuropathy, the distal, palmar surface of the second digit is assessed for decreased sensation. Placing the hand dorsal side down and abducting the fifth digit toward the ceiling tests motor function. Resistance is applied to the thenar eminence, followed by palpation, to test for contraction of the abductor pollicis brevis muscle. To test for ulnar neuropathy, the distal, palmar surface of the fifth digit is assessed for decreased sensation. Challenging the interosseous muscles best tests ulnar motor function. One method is to ask the patient to place the injured hand on a surface with the fifth digit down and the thumb pointing at the ceiling. The patient then abducts the second finger (spreads the fingers) against resistance; weakness of the first interosseous muscle verifies contraction.

Musculoskeletal assessment is targeted at identifying injuries to bones, joints, and ligaments. Bone structures should be palpated thoroughly, as should all joints, to look for signs of pain, laxity, and limited range of motion. Every digit and joint should be put through complete active and passive range of motion. All extensor and flexor tendons should be tested individually. Specific tendon function is evaluated by ranging every joint individually. Extensor tendon function is tested by extending all interphalangeal and metacarpophalangeal (MCP) joints against resistance. Flexor digitorum profundus tendon injury limits flexion at the distal interphalangeal (DIP) joint; its presence is confirmed by holding the proximal interphalangeal (PIP) and MCP joints in extension and flexing the DIP joint against resistance. Flexor digitorum sublimis tendon injury limits flexion at the PIP joint. This injury is confirmed by holding the MCP joint in extension and flexing the PIP joint against resistance. False-negative results occur if all the other digits are not held in complete extension. The uninjured thumb will have complete active range of motion without pain and should be able to oppose the fifth digit. Full flexion to a fist and full extension should be normal.

Differential Diagnosis and Medical Decision Making

Fractures of the Hand

Bennett/Reverse Bennett and Rolando Fractures

A Bennett fracture is a fracture of the proximal first metacarpal bone (Fig. 89.1). The classic mechanism is an axial load onto a flexed and adducted thumb. For example, a football quarterback strikes the helmet of an opposing player after releasing a throw. In this avulsion injury, the strong abductor pollicis longus muscle fractures the bone at the point of its insertion at the ulnar aspect of the first metacarpal bone. This causes displacement of a bony fragment, which can be seen on a plain film radiograph. It is usually an unstable fracture, and ED management should consist of referral to a hand surgeon and immobilization in a thumb spica splint. Potential long-term morbidity includes malunion, decreased function, and significant arthritis.

image

Fig. 89.1 Bennett fracture (arrows).

(From Mettler Jr FA. Essentials of radiology. 2nd ed. Philadelphia: Saunders; 2004.)

The same injury pattern and mechanism of injury can also occur in the fifth metacarpal bone and is called a reverse Bennett fracture, which is as unstable as a Bennett fracture because of the traction exerted by the extensor carpi ulnaris muscle on the distal aspect of the fifth metacarpal. Traction tends to pull the distal segment ulnarly. Closed reduction with ulnar gutter splinting may be attempted, but any articular incongruity must be recognized and referred on an emergency basis to a hand surgeon for potential immediate operative repair.

A Rolando fracture is similar to a Bennett fracture; however, it is comminuted and by definition extends into the joint space. ED management is identical to that for a Bennett fracture.

Distal Phalanx Fractures

The most common distal phalanx fracture is a tuft fracture, and nail bed injuries are the most common complication of this fracture.2 There is some controversy regarding the need to repair nail bed injuries; however, most authors recommend performing trephination only for nail bed hematomas involving 30% to 50% or greater of the nail bed surface. When the nail bed is involved, tuft fractures are considered open fractures. Although some physicians prescribe antibiotics empirically, evidence suggests that prophylactic antibiotics are not indicated.4 Proximal distal phalanx fractures are often unstable and require hand surgeon referral for percutaneous wire placement. An attempt to reduce any rotational deformity or angulation should be made before splinting. Splinting should isolate the DIP joint alone.

Ligamentous Injuries of the Hand

Gamekeeper’s/Skier’s Thumb

Gamekeeper’s thumb is also called skier’s thumb. The mechanism is hyperextension of an abducted thumb causing injury to the ulnar collateral ligament, and it is often associated with an avulsion fracture (Fig. 89.3). Historically, old-world gamekeepers sustained this injury while dispatching wounded birds during hunts. Today, this injury often occurs when a skier falls while grasping the ski pole.

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Fig. 89.3 Gamekeeper’s thumb (arrow).

(From Perron AD, Brady WJ. Evaluation and management of the high-risk orthopedic emergency. Emerg Med Clin North Am 2003;21:159–204.)

Physical examination will be remarkable for tenderness at the ulnar collateral ligament, laxity at the MCP joint, and inability to actively oppose the thumb (Fig. 89.4). Most ulnar collateral ligament ruptures occur at the distal attachment. If the injured joint demonstrates 40 degrees of radial angulation during stressing, complete ligament rupture should be assumed. An associated avulsion fracture may be present. Treatment consists of immobilization in a thumb spica splint, NSAIDs, and referral to a hand surgeon for open reduction with internal fixation.

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Fig. 89.4 Testing for ulnar collateral ligament integrity.

(From Patel D, Dean C, Baker R. The hand in sports: update on clinical anatomy and physical examination. Prim Care 2005;32:71–89.)

The initial examination may be compromised secondary to pain and spasm. In these cases, the most prudent course of action is immobilization in a thumb spica splint and referral for reevaluation.

Digital Dislocations

Dislocation of the DIP joint is a rare injury, but when it does occur, it most commonly dislocates in the dorsal direction after direct force on the finger pad. Relocation is best accomplished after a digital block with traction longitudinally and pressure directing the proximal aspect of the distal phalanx back to correct alignment. After relocation, the entire digit is splinted in extension. Some injuries are nonreducible and require operative repair because the volar plate or profundus tendon (or both) may occupy the joint space. Any indication of joint involvement should prompt referral to a hand surgeon.

PIP joint dislocations result in more complications than do DIP joint dislocations. The complex biomechanics of the joint adds a degree of intricacy that often results in the need for operative repair. The volar plate may be injured with dorsal dislocations, and the lateral collateral ligaments may be injured with ulnar or radial dislocations. It is important to assess any relocated joint for stability to better rule out the potential for ligament or volar plate injury. Patients with an irreducible or unstable joint should be referred to a hand surgeon for operative repair. If stable, the joint should be splinted in 30 degrees of flexion for 2 to 4 weeks.

MCP joint dislocations are seen less commonly than PIP dislocations but have a similar rate of complications. Dislocations may be partial or complete and may involve the volar plate. Care should be taken during the examination to not convert a partial dislocation to a complete dislocation. Injury most commonly occurs as a hyperextension mechanism. With complete dislocations and volar plate involvement, relocation is often impossible because the volar plate may become entrapped in the joint space. For the best chance of relocation, the wrist should be placed in full flexion to relieve all flexor tendon tension and exert longitudinal and volar force. The MCP joint should be splinted in full flexion.

Extensor Tendon Injuries

Open wounds on the dorsum of the hand and digits should trigger suspicion for extensor tendon injury. The Verdan extensor tendon injury classification system uses eight anatomic zones to direct treatment (Table 89.1).

Table 89.1 Verdan Classification of Extensor Tendon Injuries with Appropriate Disposition

ZONE ANATOMIC LOCATION DISPOSITION
I Distal phalanx to distal interphalangeal joint Splint, hand surgeon referral
II Middle phalanx Splint, hand surgeon referral
III Proximal interphalangeal joint Splint, hand surgeon referral
IV Proximal phalanx ED primary repair, splint
V Metacarpophalangeal joint Splint, hand surgeon referral
VI Dorsum of the hand/metacarpals ED primary repair, splint
VII Dorsum of the wrist, carpals Hand surgeon primary repair
VIII Distal forearm, proximal wrist Hand surgeon primary repair

ED, Emergency department.

Data from Verdan CE. Primary and secondary repair of flexor and extensor tendon injuries. In: Flynn JE, editor. Hand surgery. 2nd ed. Baltimore: Williams & Wilkins; 1975.

Treatment of extensor tendon injuries should usually be coordinated with a hand surgeon. Data on suture repair of partial tendon lacerations are lacking, and current treatment is based on flexor tendon treatment. Conservative treatment of injuries involving less than 50% of a cross-sectional area has been proposed.

Injuries to zones I and II occur with axial loading onto a fully extended DIP joint, which forces the DIP joint into flexion and disrupts the distal aspect of the extensor tendon. This creates a mallet injury, as described previously.

Zone III injuries are caused either by axial loading and forced flexion of the PIP joint or by direct trauma to the PIP joint. These injuries should be splinted with the joint in extension, and the patient should be referred to a hand surgeon. With complete disruption of the central slip, the lateral bands slide toward the volar surface of the digit, which causes the extensor tendons to act as flexors. Untreated injuries lead to a boutonnière deformity in which the PIP is flexed and the DIP is hyperextended.

Most zone IV injuries are caused by direct trauma. Open injuries may be treated primarily as in zone IV; by definition, no joint involvement is present. Closed injuries should be splinted with extension of the PIP joint. The extensor tendons of the phalanx are broad and flat, which allows easier primary repair.

“Fight bite” must be considered in all patients with zone V ligament injuries. Patients with open injuries should be referred to a hand surgeon for primary repair. Closed injuries can be treated by splinting the MCP joint in extension while allowing free range of motion of the PIP joint.

Zone VI injuries are usually superficial and easily repaired by the EP. Suture material should be strong, such as braided nylon, and the lacerated tendon completely apposed. After closure, the wrist should be splinted in 30 degrees of extension and the MCP joint in 15 degrees of flexion, and the PIP joint should be free. The patient should be referred to a specialist for dynamic splinting.

Zone VII and VIII injuries often involve the extensor retinaculum, and the patient should be scheduled for referral to a hand surgeon for primary closure. The affected tendon often retracts into the forearm, thus complicating the repair. Because of the density of associated anatomic structures, operative survey of the injury to identify additional injuries is indicated.

Flexor Tendon Injuries

All patients with open flexor tendon injuries should be referred to a hand surgeon for emergency evaluation (Table 89.2). However, some knowledge of the nomenclature and prognoses associated with flexor tendon injuries will aid the EP in conversations with both the consultant and the patient.

Table 89.2 Verdan Classification of Flexor Tendon Injuries with Appropriate Disposition

ZONE ANATOMIC LOCATION DISPOSITION
I Distal to insertion of the flexor digitorum sublimis tendon Hand surgeon primary repair
II Area of the flexor sheath with both the flexor digitorum sublimis and flexor digitorum profundus tendons Hand surgeon primary repair
III Carpal tunnel to the proximal aspect of the flexor sheath Hand surgeon primary repair
IV Carpal tunnel Hand surgeon primary repair
V Forearm proximal to the carpal tunnel Hand surgeon primary repair

Data from Verdan CE. Primary and secondary repair of flexor and extensor tendon injuries. In: Flynn JE, editor. Hand surgery. 2nd ed. Baltimore: Williams & Wilkins; 1975.

Repair of complete lacerations within 24 hours is most commonly recommended. Operative repair is usually limited to injuries involving greater than 50% of a cross-sectional area. Injuries involving less than 50% are frequently treated conservatively with splinting. Newer data suggest that conservative management is adequate for injuries occupying less than 75% of a cross-sectional area; however, this decision should be deferred to the consulting hand surgeon.1 When splinting flexor tendon injuries, the wrist should be placed in 30 degrees of flexion, MCP joint injuries in 70 degrees of flexion, and DIP or PIP joint injuries in 10 degrees of flexion. Classification of flexor tendon injuries is based on anatomic location, treatment, and prognosis. All patients with flexor tendon injuries should be referred to a hand surgeon for operative exploration and repair.

Fractures of the Wrist

Carpal bone fractures can result in significant long-term morbidity and are easily missed during the physical examination. The two most common carpal fractures are fractures of the scaphoid and triquetrum. Scapholunate, perilunate, and lunate dislocations are the most common dislocations.

Dislocations of the Wrist

Scapholunate, perilunate, and lunate dislocations are varying degrees of the same disease process. The mechanism is one of hyperextension. In cadaver work it was shown that progressive force applied to the wrist in a hyperextension mechanism will reliably re-create these injuries in a persistent pattern.6 The reason is anatomically based and the result of progressive ligament injuries.

Scapholunate Dislocation

Scapholunate dislocation is the most common of these injuries and occurs with the least amount of force. It can be diagnosed on plain film radiography. Scapholunate dislocation results in a classic radiologic finding—the Terry-Thomas sign (Fig 89.7). Terry-Thomas was a 20th-century British comedian who possessed a noticeable gap between his two front teeth, reminiscent of the wide space (2 mm when measured on an anteroposterior view) seen between the scaphoid and lunate bones when they are dislocated.3 Stress views accentuate this finding. Additionally, the scaphoid may twist on its axis and cause a ringlike shadow known as the signet ring sign. This artifact is caused by the x-rays traveling longitudinally down the twisted scaphoid, unlike the normal crosswise orientation. Mayfield et al. classified this injury as a stage I injury.6

image

Fig. 89.7 Scapholunate dislocation.

A, Terry-Thomas sign (arrows). B, Normal wrist. L, Lunate; N, navicular.

(From Mettler Jr FA. Essentials of radiology. 2nd ed. Philadelphia: Saunders; 2004.)

Perilunate Dislocation

Stage II injury is associated with progressively more force (e.g., an automobile accident versus a slip and fall) and results in perilunate dislocation (Fig. 89.8). Perilunate dislocations may be a difficult concept because there is no “perilunate” bone. Perilunate dislocation is a disruption of the ligamentous structures around (“peri”) the lunate bone. One of these structures is the capitate, which most often dislocates dorsally. Perilunate dislocation may perhaps better be called capitate dislocation; however, perilunate dislocation is actually a more accurate description of the stepwise disease process as outlined by Mayfield et al.6 Dislocation of the capitate can be associated with a scaphoid fracture. The EP should be diligent in assessing for one in the setting of the other. Perilunate dislocation is frequently overlooked despite the classic plain film finding of the capitate and the remainder of the distal part of the hand lying dorsal to the lunate on the lateral projection.

image

Fig. 89.8 Perilunate dislocation.

C, Capitate; L, lunate; R, radius; U, ulna.

(From Mettler Jr FA. Essentials of radiology. 2nd ed. Philadelphia: Saunders; 2004.)

Lunate Dislocation

Stage IV injury is defined by the presence of a lunate dislocation. It is a complete disruption of the ligamentous structures of the wrist. In stage I the scaphoid dislocates from the lunate, in stages II and III the capitate and triquetrum dislocate from the lunate, and in stage IV the lunate dislocates from its articulation with the distal end of the radius. The dislocated capitate, which lies dorsal to the lunate, will often collapse onto the distal end of the radius as a result of muscular spasm because the lunate is no longer present to prevent such spasm.

The result is a distal radius and capitate pseudoarticulation, with the lunate lying palmar to the “new” wrist articulation. This is best viewed on a lateral plain film projection and causes the “spilled teacup” sign (Fig. 89.9). When teaching this concept, the author has asked students to imagine a watermelon seed being squeezed between two fingers and then popping forward with force. This is essentially what happens to the lunate as it is “squeezed” by the radius and distal wrist structures, including the capitate, in an extreme hyperextension mechanism. Lunate dislocation often compresses the carpal tunnel and can cause median neuropathy.

image

Fig. 89.9 Lunate dislocation: spilled teacup sign.

C, Capitate; L, lunate; R, radius; U, ulna.

(From Mettler Jr FA. Essentials of radiology. 2nd ed. Philadelphia: Saunders; 2004.)

Plain film radiography is in general adequate to diagnose carpal bone dislocations, although computed tomography can be helpful in ambiguous cases. The EP may attempt closed reduction; however, many of these injuries are unstable. All injuries should be splinted in a long arm splint and patients should be referred to a hand surgeon. Many of these injuries will require internal fixation.

Bites to the Hand

Because of the potential for injury and morbidity, all open injuries of the MCP joint should be treated as closed-fist bite wounds, or “fight bites,” until proved otherwise. These often minor-appearing injuries are by definition caused by a clenched fist versus human teeth and are well known for poor outcomes. Potential complications include violation of the joint capsule, extensor tendon injury, and contamination of the deep fascial space.7 The potential for infection is great because of the poor vascular supply of the extensor tendon and joint capsule. Treatment of these injuries is threefold: surgical decontamination, antibiotics, and dynamic splinting.8 These injuries are not limited to fist fights and also commonly occur during sporting events.5

Delayed manifestations most commonly occur 2 to 3 days after the inciting event and consist of signs and symptoms of local or significantly advanced infection. Any indication of infection or joint space or tendon sheath involvement should prompt referral to a hand surgeon for irrigation and débridement. The timing of initiation of intravenous antibiotics should be determined in consultation with the hand surgeon, who may wish to delay antibiotic treatment until after tissue for culture has been obtained intraoperatively. Antimicrobial therapy should cover common pathogens found in the human oral and skin flora, including aerobic and anaerobic pathogens. Staphylococcus aureus is the most common pathogen, followed by Streptococcus species, Corynebacterium species, and Eikenella corrodens.9

If the signs and symptoms are acute with no indication of fracture, joint space involvement, or extensor tendon injury, antibiotic therapy and local wound care are sufficient. In this nonoperative patient group, wounds should be treated with high-volume irrigation, and they should be left open to heal by secondary intention. The injured hand should be splinted in the position of function, and the patient should be instructed to elevate the affected limb and to return if any evidence of infection is seen.

Prophylactic antibiotics for clenched-fist bite wounds should be initiated for all but the most superficial injuries. Recommended regimens include amoxicillin–clavulanic acid, a combination of penicillin and dicloxacillin, and a combination of penicillin and a first-generation cephalosporin.9

High-Pressure Injection Injuries

High-pressure injection injuries occur when substances such as paint, oil, grease, solvents, and water are sprayed under high pressure. These substances can penetrate deeply into the soft tissues of the hand and cause inflammation, infection, fibrosis, and severe disability. In one series the rate of amputation approximated 50%, and when patients are initially seen more than 6 hours after the injury, amputations are the rule rather than the exception.10,11 Even a small puncture wound with a history of high-pressure injection should be considered a high-priority emergency.

Historical information, including time since the injury, the material injected, the amount injected, the temperature of the material, and the velocity and pressure of the injection may be helpful in determining the prognosis. For example, the amputation rate is considerably lower with grease injection than with injection of paint or solvent-based material. Thinner and less viscous material is more apt to lead to amputation because of easier spread and a subsequently larger extent of injury.

Plain film radiographs should be obtained to look for subcutaneous air and radiopaque material. The hand should be elevated and maintained in a position of comfort. Parenteral analgesics are often required. Infiltration of anesthetics directly into the area of injury may worsen local inflammation and is therefore contraindicated. All these injuries require immediate consultation with a hand surgeon. Even though several authors recommend prophylactic antibiotics and some suggest systemic corticosteroids to decrease inflammation, few data support either recommendation.

Treatment

Although individual hand and wrist injuries require specific treatment, several generalizable steps should be taken to optimize management in the ED.

Follow-Up, Next Steps in Care, and Patient Education

The vast majority of hand injuries are amenable to outpatient follow-up. Care pathways should be established between the ED and hand surgeons to ensure that patients are not lost to follow-up when follow-up is vital. A relatively small subset of acute hand injuries will require immediate operative repair (e.g., vascular injuries with compromised perfusion, major crush injuries, amputations) or washout in the operating room (e.g., open fractures and joint injuries, grossly contaminated soft tissue injuries, high-pressure injection injuries, bite wounds at high risk for infection). When in doubt, the EP should not hesitate to obtain phone consultation or call a colleague to the bedside.