Adult Reconstruction

Published on 17/03/2015 by admin

Filed under Orthopaedics

Last modified 22/04/2025

Print this page

rate 1 star rate 2 star rate 3 star rate 4 star rate 5 star
Your rating: none, Average: 0 (0 votes)

This article have been viewed 3689 times

Chapter 5

Adult Reconstruction

Contents

SECTION 1 HIP DYSPLASIA—ADULT PRESENTATION

SECTION 2 HIP ARTHRITIS ASSESSMENT

SECTION 3 HIP ARTHRITIS TREATMENT

SECTION 4 OSTEONECROSIS OF THE HIP

SECTION 5 TOTAL HIP ARTHROPLASTY

SECTION 6 REVISION THA

SECTION 7 OSTEOLYSIS IN THA

SECTION 8 PERIPROSTHETIC THA FRACTURE

SECTION 9 TOTAL ARTICULAR RESURFACING

SECTION 10 THA—MISCELLANEOUS

SECTION 11 THA—JOINT STABILITY

SECTION 12 THA—ARTICULAR BEARING TECHNOLOGY

SECTION 13 KNEE ARTHRITIS ASSESSMENT

SECTION 14 KNEE ARTHRITIS TREATMENT

SECTION 15 TOTAL KNEE ARTHROPLASTY

SECTION 16 TKA DESIGN

SECTION 17 REVISION TKA

SECTION 18 PATELLAR TRACKING IN TKA

SECTION 19 CATASTROPHIC WEAR IN TKA

SECTION 20 SHOULDER ARTHROPLASTY

SECTION 21 PERIPROSTHETIC JOINT INFECTION

TESTABLE CONCEPTS

section 1 Hip Dysplasia—Adult Presentation

NATURAL HISTORY

II SPECTRUM OF PRESENTATION

III CLASSIFICATION OF ADULT HIP DYSPLASIA

IV ACETABULAR DYSPLASIA (Box 5-1 and Figure 5-1)

Classical (too little coverage)

Acetabular retroversion

1. Acetabular socket faces backward. (Normal socket faces forward, i.e., anteversion.)

2. Radiographic findings (Figure 5-2)

Acetabular overcoverage

PROXIMAL FEMORAL DYSPLASIA

Head-neck dysplasia (Figure 5-3)

Altered neck version

VI CLINICAL SYNDROME ASSOCIATED WITH DYSPLASIA

Femoral acetabular impingement

Clinical progression

Clinical presentation

Causes of femoral acetabular impingement

Femoral acetabular impingement—two types

1. Pincer impingement (Figure 5-5)

2. Cam impingement (Figure 5-6)

VII DYSPLASIA TREATMENT

Treatment depends upon the extent of deformity and location.

Surgical correction goals are to relieve pain and to correct anatomic deformity. Long-term goal is to reduce the occurrence of degenerative joint disease.

Surgical correction addresses the main anatomic deformity: shallow socket, retroverted socket, reduced femoral neck offset (i.e., fat neck), abnormal femoral neck version.

Main surgical options

Periacetabular osteotomy (Figure 5-7)

Anterior hip decompression (Figure 5-8)

Proximal hip osteotomy

section 2 Hip Arthritis Assessment

Patient assessment of hip pain includes a physical examination and diagnostic radiographic modalities.

PHYSICAL EXAMINATION TESTS FOR HIP IRRITABILITY

Impingement test

Roll test

Stinchfield test

Patrick test

II STUDIES

section 3 Hip Arthritis Treatment

NONOPERATIVE

II OPERATIVE

Arthroscopy

1. Best indication

2. Beware of labral resection in dysplasia.

3. Other indications

4. Arthroscopy technique

5. Arthroscopy complications

Osteotomy (adult)

THA

Hip fusion

1. Less frequently used as THA technology advances

2. Classical indication

3. Energy expenditure

4. Collateral arthritis

5. Hip fusion technique

6. Fusion position

7. Fusion conversion to THA

8. Function after conversion to THA

Resection arthroplasty

1. Usually last step before hip disarticulation in a frustrating downward clinical course

2. Indications

Hemiarthroplasty

1. Relegated to specific limited role

2. Hemiarthroplasty advantage

3. Hemiarthroplasty disadvantage

section 4 Osteonecrosis of the Hip

OCCURRENCE

II ETIOLOGY (see Chapter 1, Basic Science)

III CLINICAL PRESENTATION

IV IMAGING

STAGING (Table 5-1)

VI TREATMENT

Nonsurgical

Surgical treatment

1. Surgical treatment depends on these major variables.

2. Younger age and crescent (or worse)

3. Younger age and no crescent

image Common treatments

image Core decompression

image Vascularized fibular strut (Figure 5-10)

image Less-common treatments

4. Age 40 years or older and medium (B) or large (C) lesion

5. Older than 40 years and small (A) lesion

section 5 Total Hip Arthroplasty

INDICATIONS

II IMPLANT FIXATION

Methods of fixation

Cement fixation

Cemented stem failure

Cement technique—success

Biologic fixation

1. Bone ingrowth (Figure 5-12)

image Prosthesis is fabricated with metal pores into metallic alloy. Bone grows into the porous structure, stabilizing the prosthesis to bone.

image Successful bone ingrowth is based upon following factors.

image Optimal pore size

image Pore depth (i.e., deeper distance into metal) is directly related to increased fixation strength.

image Optimal metal porosity

image Minimize gap distance between prosthesis and bone.

image Minimal implant micromotion

image Cortical contact with bone

image Viable bone

Initial rigid fixation for cementless hip implants

1. Initial rigid implant fixation to host bone is required for long-term osteointegration. There are two techniques used. They are the press fit technique and the line-to-line technique.

2. Press fit technique (Figure 5-13)

3. Complication of press fit technique

4. Line-to-line technique

5. Complication of line-to-line technique

III BONE ONGROWTH FIXATION

Description

Surface roughness (Ra) (Figure 5-14)

Technique

Complication of bone ongrowth

IV HYDROXYAPATITE

PRIMARY THA—FIXATION SELECTION

VI FEMORAL STEM LOADING

Proximal porous coating (Figure 5-16)

Extensive porous coating (Figure 5-17)

Cemented stem

VII FEMORAL STRESS SHIELDING

Description

Etiology

Factors affecting stem stiffness

Archetypical scenario creating stress shielding

VIII FEMORAL STEM BREAKAGE (Figure 5-19)

section 6 Revision THA

PRESENTATION

II ACETABULAR SIDE (Figure 5-20)

Identify bone defects in acetabulum and pelvis.

Well-fixed cementless implant with osteolytic defect

Cementing a polyethylene (PE) liner into a damaged cup is associated with an increased rate of dislocation.

Significance of bone defects

Fixation revision of acetabulum

1. Cementless porous biologic fixation is preferred.

2. Hemispheric porous cup with screw is standard.

3. Reconstruction cage (Figure 5-21)

4. Acetabular screw placement (Figure 5-22)

5. Pelvic dissociation

III FEMORAL SIDE (Figure 5-23)

Identify bone defects in femur.

Significance of bone defects

Fixation revision of femur

1. Cementless porous biologic fixation is preferred.

2. Extensive porous-coated long-stem prosthesis is standard.

3. Longer stem than previous stem is recommended.

4. Impaction grafting technique

image Acceptable revision technique with limited indication

image Surgical technique

image Indication

image Complication of impaction grafting

5. Segmental bone deficiency of femur

Modular bearing change

1. Indications

2. Complications

3. Technique

4. Cementing PE bearing into fixed porous cup

section 7 Osteolysis in THA

INTRODUCTION

PE wear debris is the main culprit (when using traditional PE cup bearing). PE wear comes from two sources.

Submicron-sized particles shed by the PE bearing are responsible for eliciting the osteolysis reaction.

Adhesive bearing wear is the most important process that generates submicron-sized PE particles.

1. Types of PE bearing wear

image Adhesive wear (Figure 5-24)—This is most important mechanism in osteolysis process.

image Abrasive wear—A rough femoral head surface causes mechanical scratching of PE surface with loss of PE material (cheese grater effect).

image Third-body particles—Particles within the joint space get between head and PE cup, causing abrasion. These particles cause PE to be removed from cup surface. Third-body particle sources include

II OSTEOLYSIS PROCESS

Phagocytes of submicron-sized PE particles by macrophage; macrophage becomes activated

Additional macrophage recruitment via cytokines released by activated macrophage

Release of osteolytic factors (cytokines) by the activated macrophage; these factors include

Bone resorption mediated via RANKL

Mechanism of bone resorption mediated by osteoblast/RANKL (Figure 5-25)

III OSTEOLYSIS AROUND THA PROSTHESIS—EFFECTIVE JOINT SPACE

The intraarticular generation by PE particles elicits an inflammatory response that results in a hydrostatic pressure buildup within the joint.

PE particles are then disseminated throughout the effective joint space (Figure 5-26).

Osteolysis can occur anywhere within the effective joint space (Figure 5-27).

IV PARTICLE DEBRIS FORMATION—LINEAR VERSUS VOLUMETRIC WEAR

Volumetric wear is main determinant of the number of PE particles generated.

Volumetric wear is directly related to the square of the radius of the head (Figure 5-28).

Volumetric wear versus linear—the trade off

OSTEOLYSIS—RADIOGRAPHIC FINDINGS IN THA

VI OSTEOLYSIS REDUCTION

section 8 Periprosthetic THA Fracture

TIME OF FRACTURE

II PERIOPERATIVE FRACTURE

III LATE FRACTURE

Most late fractures occur at stem tip.

Treatment

1. Rule 1

2. Greater trochanter

3. Proximal metaphysis or diaphysis

4. Stem tip fracture

image For stem tip fracture that involves less than 25% of stem (cement and cementless stems) treatment options allowed are

image For stem tip fracture that involves more than 25% of cemented stem (cement mantle is compromised), there is only one recommended treatment.

image For stem tip fracture that involves more than 25% of cementless stem (stem fixation biologically remains intact) treatment options allowed are

5. Supracondylar fracture distant to stem tip

6. Supracondylar fracture at tip of long-stem revision prosthesis

section 10 THA—Miscellaneous

THA—NERVE INJURY

Eighty percent of injuries are to sciatic nerve; 20% involve femoral nerve.

Compression is most common pathologic mechanism of injury.

Sciatic nerve travels closest to acetabulum at the level of ischium.

Risk factors for nerve injury

Developmental dysplasia of the hip

Postoperative functional footdrop

Postoperative hematoma

II THA—ANATOMY

III THA—SPECIFIC COMPLICATIONS

Sickle cell disease

Psoriatic arthritis

Ankylosing spondylitis

Parkinson disease

Fat emboli syndrome

IV VENOUS THROMBOSIS IN THA

THA—SURGICAL APPROACH

Posterolateral

Direct lateral (Hardinge)

Anterior (Smith-Peterson)

VI THA—IMPLANT FACTS

section 11 THA—Joint Stability

Dislocation in THA frequently is a multifactorial issue. Treatment is patient specific, and the solution depends on the problem.

INCIDENCE OF THA DISLOCATION

II RISK FACTORS FOR DISLOCATION

III DISLOCATING THA—ASSESSMENT

IV COMPONENT DESIGN

Prosthetic range of motion consists of two parts.

1. Primary arc range (Figure 5-30)

2. Lever range (Figure 5-31)

PRIMARY ARC RANGE

Primary arc range is controlled by the head/neck ratio (Figure 5-32).

Additions to acetabulum and/or femoral neck decrease primary arc range.

VI LEVER RANGE

VII COMPONENT DESIGN—BEST RANGE IN THA

VIII COMPONENT ALIGNMENT

Primary arc range must be centered within patient’s functional hip range (Figure 5-37).

Component malalignment does not decrease primary arc range.

Placement of components in a malaligned position results in a stable side and unstable side of the functional hip range.

Implant positioning in THA

1. Cup anteversion20 to 30 degrees (Figure 5-38)

2. Cup theta (θ)-angle (also known as coronal tilt)35 to 40 degrees (Figure 5-39)

3. Stem anteversion10 to 15 degrees (Figure 5-40)

Cup malposition

Stem malposition

IX SOFT TISSUE TENSION

Abductor complex is key to hip stability.

Restoration of abductor tension achieved by the following (Figure 5-41):

Reduced hip offset—problems

Short neck length—problems

Restored neck length using long head—problems

1. A short femoral neck cut can be compensated with an extralong prosthetic neck length. However, a long neck length requires a skirt (Figure 5-43).

Narrow-offset femoral stem design

Greater trochanteric escape (Figure 5-45)

1. Greater trochanteric escape occurs when the greater trochanter pulls away from the proximal femur.

1. Treatment

SOFT TISSUE FUNCTION

The soft tissues about the hip are controlled by several body systems. All are integrated together to provide hip stability. The three main factors controlling soft tissue function include

CNS mechanisms causing disruption to hip function and increasing risk for dislocation

CNS conditions affecting hip function

Peripheral nervous system mechanisms causing disruption to hip function, increasing risk for dislocation

Peripheral nervous system conditions affecting hip

Local soft tissue integrity mechanisms causing disruption to hip function and increasing risk for dislocation

Local soft tissue conditions affecting hip function

XI DISLOCATING THA—TREATMENT

Each case of hip dislocation is unique. There is not one common treatment.

In each case assess

Clinical review of dislocating event important

Radiographic review

Initial treatment for dislocated THA

1. Two thirds of patients with a first-time THA dislocation can be successfully treated with closed measures.

2. Closed reduction

image Sedation or anesthesia preferred to minimize soft tissue trauma

image During closed reduction, take hip through full range and assess position of dislocation.

image Posterior hip dislocation

image Anterior hip dislocation

image Postreduction treatment

Surgical treatment

1. Surgical options

2. Rule 1surgical treatment

3. Component revision—goals

4. Greater trochanter advancement (also known as Charnley tensioning) (Figure 5-46)

5. Constrained PE socket

image A constrained PE socket encloses the femoral head and mechanically prevents hip from distracting out of socket.

image Reserved for the multiple dislocator with soft tissue dysfunction

image Best indications

image Contraindication

image Constrained cup—failure mechanisms

6. Bipolar hemiarthroplasty conversion

image Technique—Remove acetabular component. Ream remaining bone to a hemisphere. Press fit bipolar ball to rim of acetabulum (minimizes risk for medial migration of head).

image Requirements

image Advantage

image Disadvantages

7. Resection arthroplasty

Share this: