SHOULDER

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CHAPTER 4

SHOULDER

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Précis of the Shoulder Assessment*

History (sitting)

Observation (sitting or standing)

Examination

Active movements (sitting or standing)

Passive movements (sitting)

Special tests (sitting or standing)

Instability tests

Impingement tests

Labral tears

Scapular stability

Other shoulder joint tests

Muscle/tendon pathological conditions

Thoracic outlet test

Reflexes and cutaneous distribution (sitting)

Palpation (sitting)

Resisted isometric movements (supine lying)

Special tests (supine lying)

Joint play movements (supine lying)

Diagnostic imaging


*This assessment is shown in an order that limits the amount of movement the patient must do but ensures that all necessary structures are tested. After any examination, the patient should be warned that symptoms may be exacerbated as a result of the assessment.

SELECTED MOVEMENTS

ACTIVE MOVEMENTS121 image

Elevation Through Scaption

Elevation Through Abduction

INDICATIONS OF A POSITIVE TEST

Active abduction normally is 160° to 180°. Inability to attain this ROM and/or pain during the performance of this test is considered a positive test result. Altered mechanics during motion also may be an indication of a pathological condition.

When examining the movement of elevation through abduction, the examiner must take time to observe the scapulohumeral rhythm of the shoulder complex both anteriorly and posteriorly. During 180° of abduction, there is roughly a 2:1 ratio of movement of the humerus to the scapula, with 120° of movement occurring at the glenohumeral joint and 60° at the scapulothoracic joint. However, the examiner must keep in mind that a great deal of variability exists among individuals and may depend on the speed of movement; also, authors do not completely agree on the exact amounts of each movement.1820

In the unstable shoulder, the scapulohumeral rhythm commonly is altered because of incorrect dynamic functioning of the scapular or humeral stabilizers or both. This may be related to incorrect arthrokinematics at the glenohumeral joint. Kibler21 pointed out that watching the movement of the scapula in both the ascending and descending phases of abduction is especially important. Commonly, weakness of the scapular control muscles is more evident during descent as many of the muscles are required to work eccentrically. An instability jog, hitch, or jump may occur when the patient loses control of the scapula.

CLINICAL NOTES

This movement occurring simultaneously at the four joints involves three phases. There is variability regarding the amount of motion and the timing of motion that occurs at each phase of movement. Other authors will give values for the amount of each movement that differ from those noted here.

• In the first phase of 30° of elevation through abduction, the scapula is said to be “setting.” This means that the scapula moves minimally during this stage—rotating slightly in, rotating slightly out, or not moving at all. Therefore, there is no 2:1 ratio of movement during this phase. The angle between the scapular spine and the clavicle also may increase up to 5° at the sternoclavicular and acromioclavicular joints when elevating the arm; however, this depends on whether the scapula moves during this phase.

• During the next 60° of upper extremity elevation (second phase), the scapula rotates about 20°, and the humerus elevates 40° with minimal protraction or elevation of the scapula. Therefore, there is a 2:1 ratio of scapulohumeral movement. During phase 2, the clavicle elevates because of the scapular rotation, but the clavicle still does not rotate or does so minimally.

• During the final 90° of motion (third phase), the 2:1 ratio of scapulohumeral movement continues and the angle between the scapular spine and the clavicle increases an additional 10°. Therefore, the scapula continues to rotate and now begins to elevate. The amount of protraction continues to be minimal when the abduction movement is performed. During this stage, the clavicle rotates posteriorly 30° to 50° on a long axis and elevates up to a further 15°. Also during this final stage, the humerus laterally rotates 90° so that the greater tuberosity of the humerus avoids the acromion process.

• During the second and third phases, rotation of the scapula (total, 60°) is possible because of the 20° of motion at the acromioclavicular joint and 40° at the sternoclavicular joint.

Medial Rotation

CLINICAL NOTES

Lateral Rotation

CLINICAL NOTES/CAUTIONS

Extension

Horizontal Adduction/Cross Flexion

Scapular Retraction and Protraction

APLEY’S SCRATCH TEST22 image

CLINICAL NOTES

• Often the dominant shoulder shows greater restriction than the nondominant shoulder, even in the absence of a pathological condition. An exception would be individuals who continually use their arms at the extremes of motion (e.g., baseball pitchers). Because of the extra ROM developed over time doing the activity, the dominant arm may show greater ROM. However, the examiner must always be aware that shoulder movements include movements of the scapula and clavicle, as well as the glenohumeral joint. Many glenohumeral joint problems actually are scapular muscle control problems, which may secondarily lead to glenohumeral joint problems, especially in people under 40 years of age.

• The scapular reach test (neck and back) is a similar test in which the patient does medial rotation and adduction (back reach) of both arms at the same time, then lateral rotation and adduction (neck reach) of both arms at the same time. By having the patient do the combined movements, the examiner gets some idea of the individual’s functional capacity and can easily see differences between the two sides. (See Figure 5-29 in Magee DJ: Orthopedic Physical Assessment, ed. 5.)

PASSIVE MOVEMENTS2226 image

TEST PROCEDURE

Starting with the unaffected side, the examiner grasps the patient’s forearm with one hand and places the other hand on the patient’s shoulder to monitor shoulder compensation (i.e., movement at the glenohumeral joint, scapulothoracic joint, and acromioclavicular joint) while watching the sternoclavicular joint. While palpating the shoulder, the examiner passively lifts the patient’s arm sequentially into flexion, then abduction, then scaption or brings it backward to assess shoulder extension.

The examiner then can assess passive medial and lateral rotation by bringing the patient’s elbow to the side and medially or laterally rotating the arm. Rotation also can be assessed at varying degrees of shoulder abduction (most commonly, 90º).

Finally, the shoulder can be brought to 90º of shoulder abduction, provided the patient can achieve 90°. From this starting point, the shoulder can be brought across the body to assess shoulder horizontal adduction or extended backward to assess shoulder horizontal abduction.

POSTERIOR CAPSULAR TIGHTNESS TEST image

RESISTED ISOMETRIC MOVEMENTS image

TEST PROCEDURE

The muscles of the shoulder are tested isometrically, with the examiner positioning the patient and saying, “Don’t let me move you.” Pressure and force should be increased slowly and gradually. From this position, the examiner tests shoulder flexion, extension, abduction, adduction, medial rotation, and lateral rotation, as well as elbow flexion (biceps) and extension (triceps).

Flexion. The examiner places the palm of one hand on the anterior distal humerus to provide resistance near the elbow and uses the other hand to support the patient’s hand at the wrist.

Extension. The examiner places the palm of one hand on the posterior distal humerus to provide resistance near the elbow and uses the other hand to support the patient’s hand at the wrist.

Abduction. The examiner places the palm of one hand on the lateral distal humerus to provide resistance near the elbow and uses the other hand to support the patient’s hand at the wrist.

Adduction. The examiner places the palm of one hand on the medial distal humerus to provide resistance near the elbow and uses the other hand to support the patient’s hand at the wrist.

Medial rotation. The examiner places the palm of one hand on the distal forearm at the palmar aspect of the wrist to provide resistance, and the other hand just above the elbow.

Lateral rotation. The examiner places the palm of one hand on the distal forearm at the posterior aspect of the wrist to provide resistance and the other hand just above the elbow.

Elbow flexion. The examiner places the palm of one hand near the anterior wrist to provide resistance and uses the other hand to support the elbow.

Elbow extension. The examiner places the palm of one hand near the posterior wrist to provide resistance and uses the other hand to support the elbow.

CLINICAL NOTES

• The disadvantage of testing shoulder isometrics with the patient in the supine position is that the examiner cannot observe the stabilization of the scapula during the testing. Normally, the scapula should not move during isometric testing. Scapular protraction, winging, or tilting during isometric testing indicates weakness of the scapular control muscles.

• Although all the muscles around the shoulder can be tested with the patient in the supine lying position, some recommend testing the muscles in more than one position (e.g., different amounts of abduction or forward flexion) to determine the mechanical effect of the contraction in different situations.

• If the patient history includes a complaint of pain in one or more positions, these positions also should be tested. If the initial position causes pain, other positions (e.g., position of injury, position of mechanical advantage) may be tried to further differentiate the specific contractile tissue that has been injured.

• The relative percentages for isometric testing will be altered if tests are performed at faster speeds and in different planes.

• If the patient history includes a complaint that concentric, eccentric, or econcentric (biceps and triceps) movements are painful or cause symptoms, these movements should be tested with loading or no loading as required.

• When testing isometric elbow flexion, the examiner should watch for the possibility of a third-degree strain (rupture) of the long head of the biceps tendon (“Popeye muscle”).

SPECIAL TESTS FOR ANTERIOR GLENOHUMERAL INSTABILITY2737

General Information

Two types of anterior instability may be found in the shoulder. Type I, which is more closely related to muscle weakness and labral tears, can be found in any part of the ROM (translational instability). Type II, which is related to end-range instability and trauma will typically present with apprehension when tested at end ROM. Type II instability is often associated with tearing of the labrum and/or capsule.

Suspected Injury

• Unidirectional anterior instability.

• If the instability is the result of anterior dislocation or subluxation, injuries that may be present include a Hill-Sachs lesion, fracture of the anterior glenoid rim, or a Bankart lesion.

• Incorrect forward head posture, with rounded shoulders and protracted scapulae.

• Any of the peripheral nerves arising from the brachial plexus could be compromised. Damage can range from minor numbness or tingling to complete palsy of the nerve. The axillary nerve is especially susceptible.

• Anterior laxity often is accompanied by damage to the labrum, glenoid, anterior capsule, and/or brachial plexus.

• Vascular damage can occur with a shoulder dislocation. The brachial artery may be injured as it runs through the anterior shoulder complex.

Epidemiology and Demographics

Athletes who play overhead sports (e.g., tennis, volleyball, baseball) and patients with a history of dislocations or subluxations are more likely to have anterior shoulder instability. Several factors make the younger athlete more susceptible to anterior shoulder instability, such as poor technique and inadequate strength. Some researchers speculate that, in addition, adolescents’ immature collagen makeup makes them more susceptible to anterior shoulder instability and laxity.37

A wide array of incidences has been proposed in studies on anterior shoulder instability. Most patients initially dislocate the shoulder in their 20s or 30s, and 85% to 95% of dislocations in the shoulder are anterior. One fourth of all patients with dislocations have a family history of the same problem. The patient’s age at the time of dislocation has a significant impact on the recurrence rate. Reported recurrence rates in patients younger than age 20 vary from 70% to 100%.37

Relevant Signs and Symptoms

Translational Instability

• The patient may complain of generalized or anterior shoulder pain that can radiate down into the deltoid region of the shoulder, especially when the arm is above shoulder height.

• The patient may also complain of weakness in the shoulder that may be accompanied by clicking or grinding with shoulder motion.

• The patient often has a subjective feeling of instability, dislocation, or apprehension.

• The patient may complain that the shoulder does not “feel right,” especially when it is loaded above shoulder height.

• The results of unloaded active and passive movement and/or resisted isometric testing in neutral may be normal.

• Sensory loss, numbness or tingling, weakness, or complete palsy within a peripheral nerve distribution may be present in some cases.

• Coldness or weakness may be present if the vascular structures are compromised through the brachial region.

Mechanism of Injury

Unidirectional anterior instability usually is the result of repetitive overhead use, such as from throwing, swimming, or playing tennis (type I), or of trauma (type II). For type II injuries, the most common position of injury is with the arm abducted to 90º and laterally rotated to end range. Trauma that involves forced lateral rotation/abduction with a posterior force at the glenohumeral joint results in shoulder anterior dislocation or subluxation. Other mechanisms of injury include traction on the arm in an anterior direction and a fall onto an outstretched hand (FOOSH) with the arm abducted and laterally rotated.

RELIABILITY/SPECIFICITY/SENSITIVITY/COMPARISON2730,32,33,36

  Validity Interrater Reliability Intrarater Reliability Specificity Sensitivity Positive Likelihood Negative Likelihood Ratio
Anterior Crank Test Unknown Unknown Unknown 56% to 100% 35% to 91%

Crank/Relocation Test Unknown Unknown 98.91 52.78 48.42 0.48 Fulcrum Test Unknown Unknown Unknown Unknown Unknown Unknown Unknown Load and Shift Test Unknown Unknown Unknown Unknown Unknown Unknown Rockwood Test Unknown Unknown Unknown Unknown Unknown Unknown Unknown

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LOAD AND SHIFT TEST—ANTERIOR22,3849 image

MECHANISM OF INJURY

Either trauma (e.g., injury to the labrum or capsule) or lack of muscular control can produce shoulder instability. Unidirectional instability of the shoulder typically occurs after shoulder dislocation or subluxation as a result of trauma (type II instability), or it may be the result of poor muscle function (type I instability). In most cases, laxity is felt in the anterior aspect of the joint. Multidirectional shoulder instability often is the result of a genetic predisposition to joint laxity combined with overuse of the shoulder, especially in the elevated position. Generally with multidirectional shoulder instability, laxity may be experienced in both shoulders and in other joints in the body.

TEST PROCEDURE

The humerus is gently pushed into the glenoid to seat it properly in the glenoid fossa so that the humeral head sits in neutral. This is the “load” portion of the test, and this seating of the humerus allows true translation to occur. If the load is not applied to put the head in neutral, the amount of movement found will not indicate the true amount of translation, and the end feel will be altered.

Next, the humeral head is pushed anteriorly to test for anterior instability or posteriorly to test for posterior instability, and the amount of translation is noted. This is the “shift” portion of the test. The affected side and the normal side should be compared for differences. Differences between the two sides and reproduction of symptoms often are considered more important than the amount of movement obtained.

INDICATIONS OF A POSITIVE TEST

Translation of 25% of the humeral head diameter or less anteriorly from the neutral position is considered normal. Hawkins and Mohtadi39 proposed a three-grade system of anterior translation. Normally, the head translates 0 to 25% of the diameter of the humeral head. Up to 50% of humeral head translation, with the head riding up to the glenoid rim and spontaneous reduction, is considered a grade I anterior translation. In a grade II anterior translation, the humeral head has more than 50% translation, and the head feels as though it is riding over the glenoid rim, but it spontaneously reduces. Grade III is a dislocation with no spontaneous reduction.

The affected side and the normal side should be compared for the amount of translation and the ease with which it occurs. This comparison, along with reproduction of symptoms, often is considered more important than the amount of movement obtained. If the patient has multidirectional instability, both anterior and posterior translation may be excessive on the affected side compared with the normal side.

CLINICAL NOTES/CAUTIONS

• Normally, the head of the humerus should translate anteriorly 0% to 25% of the diameter of the humeral head. The head of the humerus should translate posteriorly equal if not more than the anterior translation (25% to 50% of the diameter of the humerus). (Authors vary with regard to the amount of movement possible.)

• The load and shift test is designed to test for clinical symptoms more than any particular pathological condition. Many types of pathological conditions can result in instability of the shoulder. The load and shift test can be used as an assessment tool for both multidirectional and unidirectional instability. Multidirectional instability usually is the result of a genetic predisposition to laxity in the joints. Unidirectional instability is the result of trauma or repetitive use. Shoulder dislocations/subluxations/labral tears (type II instability) and general joint laxity (type I instability) are examples of pathological conditions that can be detected with this test.

• It is important to note that a positive test result is not specific for any one pathological condition; rather, it helps to guide the clinician in the reasoning process. Instability may be a contributing factor in the development of the ultimate pathological condition. Other factors that may contribute to instability include muscle weakness of the rotator cuff and/or weakness of the scapular control muscles.

CRANK APPREHENSION TEST (RELOCATION TEST)2730,36,41,5061 image

RELEVANT SIGNS AND SYMPTOMS

• If the shoulder has not been reduced, the patient will be in pain, will be supporting the arm, and will be hesitant to move it.

• If the shoulder has been reduced, the patient may have a generalized ache or pain that may radiate down into the deltoid region of the shoulder.

• Weakness in the shoulder may be noted, and the patient may complain of clicking or grinding with shoulder motion.

• Neural irritation is not uncommon in conjunction with shoulder instability. Sensory loss, numbness or tingling, weakness, or complete palsy within a peripheral nerve distribution (usually the axillary nerve) may be seen.

• If brachial vessels have been compromised, the patient may complain of weakness, coldness, or heaviness in the hand and arm.

• The patient often has a subjective feeling of instability, dislocation, or apprehension in the shoulder (especially with lateral rotation).

TEST PROCEDURE

Step 1—the crank test. The examiner places one hand beneath the elbow to support the upper extremity. The other hand grasps the wrist and is responsible for movement of the shoulder into lateral rotation. The examiner flexes the elbow to 90°, abducts the arm to 90°, and laterally rotates the shoulder slowly, watching for apprehension. The shoulder is laterally rotated as far as possible. The hand supporting the elbow then is moved to the anterior aspect of the humeral head. The examiner should maintain the amount of lateral rotation without releasing pressure.

Step 2—the relocation test. The examiner applies a posterior stress to the humeral head and assesses whether the patient loses the apprehension, the pain decreases, and further lateral rotation is possible before the apprehension returns. This relocation sometimes is referred to as the Fowler sign or Fowler test or the Jobe relocation test.39,56 The hand pressure on the humeral head is removed, and symptoms are reassessed. For most patients, lateral rotation should be released before the posterior stress is released.

INDICATIONS OF A POSITIVE TEST

Apprehension test. A positive test result is indicated if the patient becomes apprehensive as the arm is laterally rotated and begins to contract the muscles to stop the lateral rotation (apprehension predominates). The patient may say that the feeling resembles what it felt like when the shoulder was dislocated.

Relocation test. The patient’s apprehension in the laterally rotated position disappears with the posterior translation. The examiner may find that lateral rotation will increase and apprehension will return as the lateral rotation increases. The test result is considered positive if pain decreases during the relocation maneuver, even if the patient felt no apprehension. If the arm is released (“surprise” test) in the new acquired range (step 3), pain and forward translation of the head are noted as positive test results.40,56,57

CLINICAL NOTES

• If pain rather than apprehension increases on lateral rotation, the problem is more likely to be impingement, and impingement tests should be performed.

• Hawkins and Bokor58 state that the examiner should note the amount of lateral rotation present when the patient becomes apprehensive.

• With the relocation test, lateral rotation should be released before the posterior stress is released.

• If the patient’s symptoms decrease or are eliminated during the relocation test, the diagnosis is glenohumeral instability, subluxation, dislocation, or impingement.

• If apprehension predominates during the crank test and disappears with the relocation test, the diagnosis is glenohumeral instability, subluxation, or dislocation.

• If pain predominates during the crank test and disappears with the relocation test, the diagnosis is pseudolaxity or anterior instability at either the glenohumeral joint or the scapulothoracic joint, with secondary impingement, or a posterior labral lesion.

• Kvitne and Jobe59 advocate applying a mild, anteriorly directed force to the posterior humeral head when it is in the test position to see whether apprehension or pain increases. An increase in posterior pain may indicate a posterior internal impingement.

• Hamner et al60 have suggested that if a posterior internal impingement is suspected, the relocation test should be done in 100° to 120° of abduction.

• In patients with a primary impingement, the relocation test does not alter the pain. A decrease in posterior pain when the relocation test is done posteriorly is a positive test result for posterior internal impingement.

• If the joint is normal, translation of the humeral head in the glenoid is less than with other tests, because the crank test takes the joint into the close packed position.

• If the arm is released (anterior release, or “surprise,” test) in the newly acquired range of the relocation test, a positive test result is indicated by pain and forward translation of the head.

• The release maneuver (surprise test) should be done with care, because it often causes apprehension and distrust in the patient, and it could cause a dislocation.40,56,57

• The pain that results from the release maneuver (surprise test) may be caused by anterior shoulder instability, a labral lesion (Bankart or SLAP lesion), or bicipital peritenonitis or tendinosus.40,56,57 Most often this pain is related to anterior instability, because it is temporarily produced by the anterior translation. The surprise test also has been reported to cause pain in older patients with a pathological condition of the rotator cuff and no instability.61

ROCKWOOD TEST (MODIFICATION OF THE CRANK TEST)62 image

SPECIAL TESTS FOR POSTERIOR GLENOHUMERAL INSTABILITY

Relevant Special Tests

Clinical Note/Caution

LOAD AND SHIFT TEST—POSTERIOR3844,4749,65 image

NORWOOD STRESS TEST66,67 image

TEST PROCEDURE

The examiner places the left hand as illustrated on the shoulder joint to support and stabilize the scapula. With the right hand, the examiner abducts the shoulder 60º to 100º and laterally rotates it 90º, with the elbow flexed to 90º so that the arm is horizontal. The examiner palpates the posterior humeral head with the fingers of the left hand and stabilizes the upper limb by holding the forearm and elbow at the elbow with the right hand. The examiner then brings the arm into 90° of forward flexion with the right hand.

To perform the test, the examiner horizontally adducts the abducted arm across the patient’s body with the right arm and at the same time pushes the humeral head posteriorly with the thumb of the left hand. As the humeral head is pushed posteriorly with the thumb, the examiner palpates the posterior humeral head with the fingers of the left hand to feel how far it slides posteriorly. The test first is performed on the unaffected shoulder and then the affected one, and the two sides are compared.

CLINICAL NOTES/CAUTIONS

• Cofield and Irving67 recommend medially rotating the forearm approximately 20º after the forward flexion and then pushing the elbow posteriorly to enhance the effect of the test.

• Care must be taken with this test, because it does not cause apprehension in the patient, but it may cause subluxation or dislocation.

POSTERIOR APPREHENSION OR STRESS TEST41,49,63,68,69 image

SPECIAL TESTS FOR INFERIOR GLENOHUMERAL INSTABILITY

Relevant Special Tests

Mechanism of Injury

Shoulder instability may be the result of trauma, repetitive use, or a genetic joint laxity. Multidirectional instability may be the result of a genetic predisposition to joint laxity. Generally, genetic laxity occurs in both shoulders and in other joints. It is believed that if a patient demonstrates inferior instability, multidirectional instability also is present; therefore, a patient with inferior instability also will demonstrate anterior or posterior instability, or both.

SPECIAL TESTS FOR IMPINGEMENT

Relevant Special Tests

Epidemiology and Demographics83

Neer has described three stages of impingement (Table 4-2).

Table 4-2

Neer’s Grades of Shoulder Impingement83

Grade Pathology Symptoms Range of Motion and Strength
Grade I Subacromial bursitis/tendonitis

Minimal loss of ROM and strength Grade II Partial rotator cuff tear Symptoms similar to grade I but symptoms more pronounced Moderate loss of ROM and strength Grade III Full-thickness rotator cuff tear Significant swelling and ecchy-mosis Severe loss of ROM and strength

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Mechanism of Injury8488

Anterior shoulder impingement. Regardless of its cause (e.g., pathological condition of the rotator cuff, bicipital paratenonitis/tendinosis, scapular or humeral instability, labral pathology), anterior shoulder impingement most commonly results when structures are compressed in the anterior aspect of the humerus between the head of the humerus and the coracoid process, under the acromion process and under the coracoacromial ligament. These injuries may be acute or degenerative. An acute injury produces a sudden onset of shoulder pain after a specific activity. The aggravating activity commonly is an overhead maneuver, such as throwing or painting. Trauma, such as a fall onto an outstretched hand (FOOSH injury), also can result in shoulder impingement. Because of the progressive nature of subacromial impingement, patients with degenerative-type impingement often complain of several bouts of shoulder pain over a number of years. Each subsequent episode generally is worse than the previous one. The patient often reports that the latest episode did not resolve and has increasing loss of ROM.

Posterior shoulder impingement. Posterior impingement occurs when the rotator cuff impinges against the posterosuperior edge of the glenoid when the arm is abducted, extended, and laterally rotated. The result is a “kissing” labral lesion posteriorly. The resulting impingement occurs between the rotator cuff and greater tuberosity on one side and the posterior glenoid and labrum on the other side. This type of impingement often accompanies anterior instability or pseudolaxity, and deltoid activity increases to compensate for weakened rotator cuff muscles.

RELIABILITY/SPECIFICITY/SENSITIVITY/COMPARISON89

  Validity Interrater Reliability Intrarater Reliability Specificity Sensitivity

Unknown Unknown Unknown 25% 92% Neer Impingement Test Unknown Unknown Unknown 30.5% 88.7% Unknown Unknown Unknown Unknown Unknown

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HAWKINS-KENNEDY IMPINGEMENT TEST87,8999 image

CLINICAL NOTES/CAUTIONS

• The test also may be performed in different degrees of forward flexion (vertically “circling the shoulder”) or horizontal adduction (horizontally circling the shoulder).

• McFarland et al.96 described the coracoid impingement sign, which is the same as the Hawkins-Kennedy test but involves horizontally adducting the arm across the body 10° to 20° before doing the medial rotation. This is more likely to approximate the lesser tuberosity of the humerus and the coracoid process.

• The Yocum test is a modification of the Hawkins-Kennedy test in which the patient’s hand is placed on the opposite shoulder and the examiner elevates the elbow.97,98

• Park et al.99 found that combining tests gave better results. They found that the Hawkins-Kennedy test, the painful arc sign, and a positive result on the infraspinatus test gave the best probability of detecting impingement, whereas the painful arc sign, drop arm test, and infraspinatus test were best for detecting full-thickness rotator cuff tears.

NEER IMPINGEMENT TEST89,90,95,99102 image

POSTERIOR INTERNAL IMPINGEMENT TEST55,103106 image

SPECIAL TESTS FOR LABRAL TEARS OF THE GLENOHUMERAL JOINT

Relevant Special Tests

Note: Parentis et al.107 concluded that no one test can provide a definitive diagnosis of a labral lesion.

Mechanism of Injury

Repetitive overhead activities, such as serving a tennis ball or throwing, can result in labral injury. Traumatic injuries also can damage the labrum, such as a FOOSH injury, catching a heavy object with forced biceps contraction, shoulder dislocation, and shoulder subluxation.

A Bankart lesion occurs most commonly with a traumatic anterior dislocation that leads to anterior instability. For example, if this type of injury occurs in the right shoulder, the labrum is detached anywhere from the 3 o’clock to the 7 o’clock position, resulting in both anterior and posterior structural injury. Not only is the labrum torn; the stability of the inferior glenohumeral ligament also is lost.

With a SLAP lesion, the labrum is detached from the glenoid (pulled or peeled, depending on the mechanism). This detachment can occur anywhere from the 10 o’clock to the 2 o’clock position on the glenoid. This lesion often results from a FOOSH injury, occurs during deceleration when throwing, or arises when sudden traction is applied to the biceps. If the biceps tendon also detaches, the shoulder becomes unstable and the support of the superior glenohumeral ligament is lost.

Clinical Note/Caution

RELIABILITY/SPECIFICITY/SENSITIVITY COMPARISON2729,110112

  Active Compression Test of O’Brien Anterior Slide Test Biceps Load Test Biceps Tension Test Clunk Test Labral Crank Test SLAP Prehension Test
Specificity 31% to 55% 84% to 91.5% 96.6% Unknown Unknown 56% to 100% Unknown
Sensitivity 47% to 54% 8% to 78.4% 89.7% Unknown Unknown 46% to 91% Unknown

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ACTIVE COMPRESSION TEST OF O’BRIEN29,30,32,53,102,103,110,111,113,114 image