Knee and Lower Leg Injuries

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84 Knee and Lower Leg Injuries

Pathophysiology

Knee

Anatomy

The knee is the largest and most complex joint in the body. Injuries to this joint are common, so a clear understanding of the anatomy and pathophysiology of the knee is essential for appropriate evaluation, diagnosis, and treatment of disorders in this area.

The knee has a wide range of motion, including flexion, extension, abduction, adduction, and internal and external rotation. Three different articulations are present in the knee: the patellofemoral articulation (anterior) and articulations between the lateral and medial tibial and femoral condyles. In full extension, the stabilizing ligaments of the knee are tight and prevent rotary motion of the knee. Beyond 20 degrees of flexion, the ligaments are relaxed and allow axial rotation of the joint.

Knee stability is provided solely by ligaments and tendons in and around the joint (Fig. 84.1). The knee joint is encapsulated by fibrous connective tissue lined by a synovial membrane. The knee capsule is continuous with the suprapatellar bursa, which expands when a joint effusion is present.

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Fig. 84.1 Knee anatomy.

ACL, Anterior cruciate ligament; LCL, lateral collateral ligament; MCL, medial collateral ligament; PCL, posterior cruciate ligament.

(From Brown JR, Trojian TH. Anterior and posterior cruciate ligament injuries. Prim Care 2004;31:925–56.)

The popliteal fossa contains the popliteal artery and vein and the peroneal and tibial nerves. The popliteal fossa is delineated laterally by the biceps femoris muscle, medially by the semimembranosus and semitendinosus muscles, and inferiorly by the gastrocnemius muscle.

The popliteal artery is a continuation of the femoral artery after it leaves the adductor hiatus. It gives rise to the geniculate arteries, which form a rich vascular anastomosis around the knee, and divides to form the anterior and posterior tibial arteries at the level of the tibial tubercle. The popliteal artery is immobilized proximally and distally within the popliteal fossa, which predisposes it to vascular injury in the setting of traumatic knee injuries.

The tibial nerve and common peroneal nerve (a branch of the tibial nerve) innervate the knee. Because the tibial nerve is not immobilized proximally, it is less likely than the popliteal artery to be injured in the setting of joint disruption. The common peroneal nerve travels around the head of the fibula and divides into the deep and superficial peroneal nerves.

Evaluation

Key aspects of evaluation of knee are listed in Box 84.1. In general, evaluation of knee complaints should also include an examination of the hip and back to prevent overlooking a source of pain referred to the lower extremity.

During evaluation, the point of maximal tenderness should be assessed last. Specific tests for evaluating ligamentous and meniscal injuries are detailed in Table 84.1. Comparison with the uninjured or normal knee is helpful, especially for assessment of ligamentous laxity.

Joint pain or swelling may limit full evaluation of the knee in the acute setting. Patients with limited evaluations should undergo immobilization and follow-up examination within 7 days. Key physical findings are listed in discussions of specific disorders later in the chapter.

Diagnostic Testing

Because acute injuries to the knee commonly involve soft tissue, plain radiographic examination is not always indicated. The Ottawa Knee Rules1 (Box 84.2) and the Pittsburgh Knee Rules2 (Box 84.3) are useful guides to aid in the decision of whether to order plain radiographs. Both criteria are sensitive for fractures, but the Pittsburgh criteria are more specific and can be applied to both children and adults.

If plain radiographs are indicated, a minimum of an anteroposterior (AP) and a lateral view should be obtained. Oblique radiographs are helpful for detecting subtle tibial plateau fractures. The intercondylar or tunnel view is helpful in evaluating for tibial spine fractures and osteochondral defects (Fig. 84.2). Assessment of the patellofemoral joint and evaluation for the presence of patellar tilt (increased propensity for patellar subluxation or dislocation) can be done with the Merchant or sunrise view (Fig. 84.3). Comparison radiographs of the unaffected extremity are helpful in discerning problems in skeletally immature patients.

When describing the knee radiograph, the examiner should note the alignment and joint spacing of the femoral condyles in relation to the tibial plateau. Narrowing of the joint space (particularly in weight-bearing views) indicates articular cartilaginous and meniscal degeneration. The patella should be examined for possible fractures (in the event of a direct blow to the anterior aspect of the knee) and the presence of a bipartite or tripartite patella. Significant joint effusions are evident as a water-density radiolucency on the lateral view, anterior to the distal end of the femur.3 Effusions seen shortly after injury are suggestive of anterior cruciate ligament (ACL) or posterior cruciate ligament (PCL) tears, tibial plateau fractures, femoral condyle fractures, or patellar fractures.4

In the setting of acute injuries, radiographs should be examined for the presence of fractures involving the tibial plateau (depression fracture) or tibial spine (suggesting rupture of the ACL). Segond fractures are avulsion fractures of the lateral tibial plateau at the site of attachment of the lateral capsular ligament. These fractures are associated with ACL and meniscal injuries. The presence of posterior opaque bodies should also be noted. These may be fabellae (congenital sesamoid) or loose bodies. More than 75% of loose bodies originate from osteochondral lesions.3 Occult fractures that are commonly missed on plain radiographs include patellar, tibial plateau, fibular head, and Segond fractures.5

Musculoskeletal ultrasound techniques can be used to diagnose ACL and PCL tears and may be helpful in the diagnosis of meniscal injuries.4 Magnetic resonance imaging (MRI) can be used to confirm suspected meniscal injuries, ligamentous disruptions, osteochondral lesions, and occult fractures; however, it is rarely indicated in the acute setting. Computed tomography (CT) is helpful for establishing the extent of certain fractures (e.g., tibial plateau fractures) and is often more readily available than MRI.

Lower Leg

Dislocations and Fractures

Dislocations

Knee Dislocations

Knee dislocations are relatively uncommon and represent, at most, 0.2% of all orthopedic injuries. They require disruption of at least three of the four major ligaments of the knee. Common mechanisms include traffic accidents, sporting injuries, or simple mechanical falls. Dislocations are classified on the basis of the direction of tibial movement in relation to the femur. Anterior and posterior dislocations account for approximately 70% of all knee dislocations. Knee dislocations may also have associated intraarticular fractures involving the tibial plateau or femoral condyles.6

Knee dislocations are associated with a high risk for neurovascular injury and should be considered an orthopedic emergency. The neurovascular bundle runs posterior to the bony and ligamentous structures in the popliteal fossa. The popliteal artery and nerve are fixed in the fibrous tunnel of the adductor magnus muscle proximally and traverse the fibrous arch of the soleus muscle and interosseous membrane distally. The relative immobility of the neurovascular bundle makes it susceptible to injury. The popliteal artery may be injured in up to 14% of all knee dislocations (Fig. 84.5). Traction injuries to the common peroneal and, less commonly, to the tibial nerve may also be present.6

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Fig. 84.5 A, Radiograph demonstrating posterior knee dislocation from a dashboard injury. B, Arteriogram showing an injury to the popliteal artery.

(From Browner BD, Jupiter JB, Levine AM, et al. Skeletal trauma; basic science, management, and reconstruction. 3rd ed. Philadelphia: Saunders; 2003.)

A grossly unstable knee after a traumatic injury should be assumed to be a reduced dislocation until proved otherwise. Any patient suspected of having sustained a knee dislocation should undergo a careful neurovascular examination. Anterior and posterior dislocations have a higher incidence of vascular injury. Vascular compromise in a dislocated knee requires immediate reduction.

Neurovascular status should be documented before and after reduction. All patients with a suspected or confirmed knee dislocation should have the ankle-brachial index (ABI) calculated. An ABI of less than 0.9 has high predictive value for a vascular injury, and such patients should undergo either CT or traditional angiography. Patients with normal findings on vascular examination and an ABI lower than 0.9 should be observed for at least 24 hours with neurovascular checks every 2 to 3 hours.4 Prompt diagnosis of vascular injury is essential given the chance of development of progressive distal ischemia. When injury to the popliteal artery has occurred, patient outcome is directly related to the duration of ischemia.

Standard AP and lateral radiographs are adequate for initial evaluation of knee dislocations. After appropriate analgesia and sedation, emergency reduction of the dislocated knee should be attempted. Longitudinal traction on the tibia (to free it from the femur) should be followed with a force in the opposite direction of the dislocation. The rotary components should also be corrected to restore normal leg alignment. After reduction, the knee should be immobilized in 15 to 20 degrees of flexion.4 Orthopedic consultation in the ED is mandatory for all suspected and confirmed knee dislocations.

Patellar Dislocation

The patella normally articulates in the groove between the femoral condyles. The vastus medialis, medial and lateral patellofemoral, and patellotibial ligaments and the medial retinaculum all stabilize the patella (Fig. 84.6).

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Fig. 84.6 Patellofemoral and patellotibial ligaments.

These structures act as static stabilizers of the patella.

(From DeLee JC, Drez Jr D, Miller MD. DeLee & Drez’s orthopaedic sports medicine: principles and practice. 2nd ed. Philadelphia: Saunders; 2003.)

The overall incidence of patellar dislocations is estimated to be 7 per 100,000 per year and as high as 31 per 100,000 per year in patients between the age of 10 and 19 years.7 Patellar dislocations most commonly occur when a varus force is applied to a flexed knee or after forced contraction of a flexed quadriceps. Dislocations may be associated with meniscal tears, disruption of the medial collateral ligament, and osteochondral fractures.4

The patient may report that the knee “gave out,” followed by pain and swelling. Patients may not be able to bear weight on or flex the knee. An acute hemarthrosis is most commonly seen if an associated osteochondral fracture is present. A patellar apprehension test may be useful in a patient who reports a dislocation that resolved spontaneously. This test is performed by moving a nondisplaced patella laterally. The result is positive if the patient shows apprehension, senses pain, or feels a sensation of impending dislocation when the patella is moved laterally (Fig. 84.7). AP, lateral, and sunrise radiographs are adequate to evaluate for acute dislocations and associated fractures.

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Fig. 84.7 Positive apprehension test.

As the patella is displaced laterally over the lateral femoral condyle, the patient experiences apprehension and discomfort.

(From DeLee JC, Drez Jr D, Miller MD. DeLee & Drez’s orthopaedic sports medicine: principles and practice. 2nd ed. Philadelphia: Saunders; 2003.)

After proper sedation, the emergency physician (EP) should reduce a lateral patellar dislocation by flexing the hip and pushing medially on the patella while extending the knee. Postreduction radiographs are mandatory to rule out osteochondral fractures. Intraarticular, horizontal, and superior dislocations typically require open reduction. Dislocations associated with osteochondral fractures are generally treated surgically.

After reduction, the patient should be told to use conservative therapeutic measures, such as ice, elevation, and pain control. The knee should be placed in a straight leg immobilizer, and the patient can start progressive weight bearing when comfortable. Follow-up should be arranged within 1 to 2 weeks. Complications include persistent instability, subluxation, repeated dislocation, and osteoarthritis.4

Fractures

Patellar Fracture

The patella, the largest sesamoid bone in the body, is enveloped within the quadriceps tendon and articulates with the trochlear groove of the distal end of the femur. The superficial location of the patella makes it more susceptible to injury.

Patellar fractures account for approximately 1% of skeletal injuries and are the result of either a direct injury (dashboard injury) or an indirect injury (violent flexion). Indirect injuries result in an avulsion injury of the patella as a result of pull of the quadriceps muscle against resistance. Transverse patellar fractures are the most common type of fracture and are more likely to be displaced and be manifested as a disrupted extensor mechanism.

The patient usually has a swollen, painful knee. Patellar evaluation includes palpation for pain and bony disruption and assessment for extensor weakness. AP, lateral, and sunrise views of the knee should be obtained. A bipartite patella may be difficult to distinguish from a patellar fracture and is most often seen in the superolateral part of the patella. A high-riding patella, or a patella alta position, may signify disruption of the distal extensor mechanism; this is best visualized on AP and lateral views.8

Acute treatment of patellar fractures consists of ice, elevation, pain control, and a straight leg knee immobilizer. Nonoperative intervention is considered for nondisplaced fractures (<3 to 4 mm) when the extensor mechanism is intact. Patients with a disrupted extensor mechanism should have immediate orthopedic consultation because these injuries are often repaired within 24 hours.4

Proximal Tibial Fractures

Proximal tibial fractures include fractures above the tibial tuberosity.

Tibial Plateau Fractures

The proximal end of the tibia comprises the medial and lateral condyles, which make up approximately three fourths of the proximal tibial surface. The condyles ensure appropriate knee alignment, stability, and motion. Tibial plateau fractures account for about 1% of all proximal tibial fractures.9

Tibial plateau fractures are caused by side loading secondary to either a varus or a valgus force combined with axial compression, which results in the femoral condyle impacting on the tibia. Common mechanisms are motor vehicle crashes, falls, and athletic activities such as skiing (Fig. 84.8). A Segond fracture is bony avulsion of the lateral tibial plateau at the site of attachment of the lateral capsular ligament. This fracture is an important marker for ACL disruption and anterolateral rotary instability. Associated soft tissue injuries to the collateral ligaments, menisci, and neurovascular structures are common, although low-energy injuries (from athletic activities) usually result in less soft tissue damage.9

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Fig. 84.8 Lateral tibial plateau fracture.

(From Browner BD, Jupiter JB, Levine AM, et al. Skeletal trauma: basic science, management, and reconstruction. 3rd ed. Philadelphia: Saunders; 2003.)

Patients with tibial plateau fractures exhibit pain and swelling of the knee and hold their knee in a slightly flexed position. A valgus or varus deformity of the knee generally indicates a depressed fracture. Careful assessment of associated ipsilateral bony, soft tissue, and neurovascular status is essential given the high rate of association of such injuries with tibial plateau fractures.9

AP, lateral, and oblique radiographs are necessary to evaluate tibial plateau fractures. In addition, a tibial plateau view is helpful in assessing the amount of depression present. CT with 2-mm cuts and three-dimensional reconstruction is useful to further investigate indeterminate plain films, evaluate fracture patterns, show the precise extent of articular depression, and assist in planning for optimal operative treatment.4

Nonoperative treatment is indicated for minimal or nondisplaced fractures (<2 to 3 mm of articular incongruity), peripheral (submeniscal) fractures, and fractures in elderly, low-demand, or osteoporotic patients. Patients should not bear weight on the affected leg for 4 to 6 weeks.

Absolute indications for surgery are open fractures, arterial injuries, and compartment syndrome. Relative indications for surgery are displaced fractures leading to joint instability and depression of the plateau. The amount of depression that requires operative intervention is controversial and ranges from 3 to 10 mm; however, 3 mm is the usual cutoff in athletic patients.9

Tibial Spine Fractures

Tibial eminence avulsion fractures occur most often in children 8 to 14 years of age but can be seen in skeletally mature patients as well. A fracture of the anterior tibial spine in children is equivalent to an ACL rupture in adults.

The intercondylar eminence, or tibial spine, is the central portion of the proximal tibial surface. Tibial spine injuries usually result from a hyperextension force with or without a valgus or rotational moment about the knee. The fracture may also occur after a direct blow to the distal end of the femur while the knee is flexed (Fig. 84.9).

Affected patients have a suggestive history and a painful, swollen knee. In most cases, patients are unable to fully extend the knee and exhibit an effusion, and findings on stability tests (Lachman, anterior drawer) are abnormal. The examiner should carefully evaluate such patients for associated ligamentous injuries.

Routine AP and lateral radiographs are adequate to define these fractures. CT is helpful for evaluating displacement, whereas MRI is superior in assessing any accompanying soft tissue injuries.

Fractures with little or no displacement should be immobilized in a long leg splint with the knee flexed at approximately 10 to 20 degrees. Displaced fractures necessitate orthopedic consultation because they may require closed reduction (if no ligamentous damage is present) or open or arthroscopic reduction with fixation of the fragments.10

Tibial Shaft Fractures

Tibial shaft fractures are the most common long-bone fractures, as well as the most common open long-bone fracture. They are commonly associated with a fibular fracture or ligamentous injury. The fibula remains intact in only 15% to 25% of tibial shaft fractures. These fractures are associated with a high incidence of infection, delayed union, nonunion, and malunion.

Tibial shaft fractures result from either direct (motor vehicle accidents) or indirect (rotary or compressive forces) trauma. High-energy direct injuries usually cause transverse or comminuted fractures (most common). Indirect trauma commonly results in spiral or oblique fractures (Fig. 84.10).

A good neurovascular examination is essential. Skin integrity should be noted, and documentation of the integrity of the peroneal nerve is mandatory, as is a thorough examination of the knee and ankle.

AP and lateral radiographs should be obtained and must include the knee and ankle in both views. Closed tibial shaft fractures are immobilized in a long leg posterior splint with 10 to 20 degrees of knee flexion. Closed tibial and fibula shaft fractures, especially if displaced, are at risk for the development of compartment syndrome. Such injuries may necessitate observation in the hospital. It is estimated that compartment syndrome may develop in approximately 8% of tibial diaphyseal fractures. Any patient discharged home from the ED with a closed tibial fracture should be educated about the signs of compartment syndrome.

Open fractures should be gently cleaned, dressed with sterile dressings, and placed in a splint while awaiting orthopedic consultation. Patients should receive prophylactic antibiotics and tetanus prophylaxis. Emergency reduction is necessary for injuries accompanied by neurovascular compromise. Nonunion or delayed union is more likely with fractures that are open, severely displaced, or comminuted or with fractures associated with severe soft tissue injuries or infections.11

Tibial Tuberosity Fractures

The tibial tubercle is a bony prominence that is found approximately 3 cm distal to the proximal articular surface of the tibia and in line with the medial half of the patella. It is the insertion point of the extensor mechanism, and thus accurate reduction and healing of this structure are essential.

Tibial tubercle avulsion fractures are rare injuries. Although they can occur in adults, these fractures are more commonly seen in adolescents undergoing a growth spurt. Most fractures are the result of an indirect force delivered by an eccentric load.12 A sudden flexion force is applied while the knee is in flexion and the quadriceps is tightly contracted. The quadriceps resists the force, which causes avulsion of the tibial tubercle.

The physical findings depend on the extent of the injury. Swelling and tenderness are present over the anterior aspect of the tibia. A joint effusion may result from associated intraarticular injuries. The injured knee is usually held in 20 to 40 degrees of flexion secondary to hamstring spasm. In addition, the patient may not be able to extend the knee because of either pain or loss of the extensor mechanism.13

Routine AP and lateral radiographs are useful for ruling out associated fractures (Fig. 84.11). Initial treatment is similar to that for both quadriceps and patellar tendon injuries. Minimally displaced fractures are treated conservatively. Displaced fractures frequently require open reduction and internal fixation.12

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Fig. 84.11 Lateral knee radiograph in a 14-year-old boy with a displaced fracture of the tibial tubercle.

(From Green NE, Swiontkowski MF. Skeletal trauma in children, vol 3. 3rd ed. Philadelphia: Saunders; 2003.)

Soft Tissue and Cartilaginous Injuries

Quadriceps and Patellar Tendon Injuries

The quadriceps tendon represents the convergence of the rectus femoris, vastus intermedius, vastus lateralis, and vastus medialis muscles. It inserts on the superior pole of the patella. The patellar tendon travels from the inferior pole of the patella to the tibial tubercle.

Quadriceps ruptures typically occur in patients older than 40 years. Rupture is usually the result of forced quadriceps contraction with a flexed knee, which loads the tendon. Direct blows and lacerations can also cause disruption of the tendon. The most common site of rupture is at or near its insertion on the patella. Predisposing factors are listed in Box 84.4.

Patellar tendon ruptures are less common. Risk factors for patellar tendon rupture are similar to those for quadriceps rupture, with the exception that they generally occur in patients younger than 40 years. Most patellar tendon ruptures occur along the inferior pole of the patella.

Pain, swelling, and ecchymosis are usually localized to the superior pole (quadriceps tendon) or inferior pole (patellar tendon) of the patella. A defect in the patella or quadriceps tendon may be palpable on physical examination. Other physical findings include a low-riding patella (patella baja) with inferior retraction of the patella (quadriceps tendon rupture) or a high-riding patella (patella alta) with superior retraction of the patella (patellar tendon rupture). The integrity of the extensor mechanism should always be evaluated.

AP and lateral radiographs help define the patellar position (alta or baja) and rule out associated fractures. Ultrasound has also been shown to be useful in diagnosing quadriceps tears. MRI is helpful in the diagnosis of partial tears.

Orthopedic consultation in the ED is indicated for suspected injuries and complete ruptures. Initial treatment consists of ice, elevation, and a straight leg immobilizer. Partial tears are treated by immobilization for 4 to 6 weeks, whereas complete tears are treated surgically. Diagnosis is essential given the better outcomes with prompt referral and repair.

Ligamentous and Meniscal Injuries

Knee stability depends on the static stability of the ligaments and the dynamic stability of the muscles. Injuries to the knee involve the following six common mechanisms: (1) valgus stress (laterally directed), (2) varus stress (medially directed), (3) hyperextension, (4) rotational stress, (5) direct anterior stress, and (6) direct posterior stress. These stressors, working in isolation or combination, may result in myriad of ligamentous, meniscal, or chondral injuries.12

Common mechanisms of ligamentous knee injuries are shown in Figure 84.12. Diagnosis is based on clinical examination; plain radiographs are indicated if a fracture is suspected. MRI offers a direct, noninvasive view of the knee ligaments, menisci, and other soft tissue structures; however, it is rarely indicated in the ED setting.

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Fig. 84.12 Common mechanisms of knee injuries.

ACL, Anterior cruciate ligament; LCL, lateral collateral ligament; MCL, medial collateral ligament; PCL, posterior cruciate ligament.

(From Browner BD, Jupiter JB, Levine AM, et al. Skeletal trauma: basic science, management, and reconstruction. 3rd ed. Philadelphia: Saunders; 2003.)

Stability testing for ligamentous and meniscal injuries is outlined in Table 84.1 and Figures 84.13 to 84.16. Ligament function, mechanism of injury, diagnosis, and treatment are outlined in Table 84.3. Most ligamentous and meniscal injuries should be reevaluated by an orthopedist in 3 to 5 days.4

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Fig. 84.13 Lachman test for anterior drawer instability.

The test is done at 20 to 30 degrees of flexion. The examiner stabilizes the femur with one hand and draws the tibia anteriorly with the other hand.

(From Browner BD, Jupiter JB, Levine AM, et al. Skeletal trauma: basic science, management, and reconstruction. 3rd ed. Philadelphia: Saunders; 2003.)

Muscle Strains

Overuse Injuries

Common tendinopathies and bursitis of the knee are described in Chapter 86.

Stress Fractures

A stress fracture is a partial or complete bone fracture that results from repeated stress. Stress fractures are most common in the lower extremities (tibia, fibula, and metatarsal bones). They occur as a result of a repetitive use injury that exceeds the intrinsic ability of the bone to repair itself. Tibial stress fractures are commonly seen in military recruits, as well as in track and long-distance runners.

Patients have localized dull pain in the lower extremity that is not related to trauma. The pain typically worsens during exercise or weight bearing. Stress fractures can be manifested in a similar fashion as shin splints but are associated with more focal bony tenderness.

Evidence of a stress fracture may not appear on plain radiographs for up to 2 to 10 weeks after the onset of symptoms. The presence of a transverse fracture line across the entire anterior shaft of the tibia on a plain radiograph is considered a poor prognostic sign and is associated with a greater likelihood of nonunion.8 Radionuclide scintigraphy can confirm the diagnosis as early as 2 to 8 days after the onset of symptoms.

Initial treatment consists of conservative therapy: ice, antiinflammatory pain medication, and rest for several weeks or until the extremity is free of pain. Tibial fractures that do not improve with conservative management may require splinting in a walking boot or air splint. A midshaft tibial fracture is splinted until the extremity is pain free with radiographic evidence of healing.15

Other Disorders

Popliteal Cyst

A popliteal cyst, or Baker cyst, is an inflammation of the semimembranosus or medial gastrocnemius bursa. A Baker cyst is produced by herniation of the synovial membrane through the posterior knee capsule. It is usually the result of synovitis, arthritis, or an internal derangement of the knee that results in excess synovial fluid in the bursa.

Intermittent swelling can develop behind the knee. If the bursa ruptures, the patient may complain of calf pain, and the findings may be similar to those in patients with thrombophlebitis.

Ultrasonography is helpful to distinguish Baker cysts from other disorders, such as popliteal artery aneurysms, neoplasms, and thrombophlebitis. Treatment is based on the underlying cause. Asymptomatic cysts found incidentally need no further treatment.19

References

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