Intermittent Positive-Pressure Breathing

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14 Intermittent Positive-Pressure Breathing

Note 1: This book is written to cover every item listed as testable on the Entry Level Examination (ELE), Written Registry Examination (WRE), and Clinical Simulation Examination (CSE)

The listed code for each item is taken from the National Board for Respiratory Care’s (NBRC) Summary Content Outline for CRT (Certified Respiratory Therapist) and Written RRT (Registered Respiratory Therapist) Examinations (http://evolve.elsevier.com/Sills/resptherapist/). For example, if an item is testable on both the ELE and the WRE, it will simply be shown as: (Code: …). If an item is only testable on the ELE, it will be shown as: (ELE code: …). If an item is only testable on the WRE, it will be shown as: (WRE code: …).

Following each item’s code will be the difficulty level of the questions on that item on the ELE and WRE. (See the Introduction for a full explanation of the three question difficulty levels.) Recall [R] level questions typically expect the exam taker to recall factual information. Application [Ap] level questions are harder because the exam taker may have to apply factual information to a clinical situation. Analysis [An] level questions are the most challenging because the exam taker may have to use critical thinking to evaluate patient data to make a clinical decision.

Note 2: A review of the most recent Entry Level Examinations (ELE) has shown an average of 3 questions (out of 140), or 2% of the exam, that cover intermittent positive-pressure breathing (IPPB). A review of the most recent Written Registry Examinations (WRE) has shown an average of 2 questions (out of 100), or 2% of the exam, that cover IPPB. The Clinical Simulation Examination is comprehensive and may include everything that should be known by an advanced level respiratory therapist

MODULE A

1. Description

The Respiratory Care Committee of the American Thoracic Society published the following definition in its 1980 Guidelines for the Use of Intermittent Positive Pressure Breathing (IPPB): “‘IPPB treatments’ refers to the use of a pressure-limited respirator to deliver a gas with humidity and/or aerosol to a spontaneously breathing patient for periods of time that are generally no greater than 15 to 20 minutes each.”

A pressure-limited respirator may be powered by compressed gas or electricity. The patient’s tidal volume (VT) should be greater than normal when enhanced by IPPB. This greater-than-normal VT is caused by the use of positive pressure against the lungs. Pressure is also directed against the airways and, through contact with the airways and lungs, the entire chest. The patient’s exhalation is usually passive but can be slowed through modification of the exhalation valve.

Shapiro and associates (1991) list the following as the physiologic effects of IPPB:

d. Alteration of the inspiratory/expiratory ratio

Patients with high airway resistance or low lung compliance often change their breathing patterns to reduce the WOB (see Chapter 1). These new breathing patterns may lead to worsening of the patient’s condition. Alteration of normal ventilation and perfusion ratios in the lungs may worsen hypoxemia. Properly administered and coached IPPB can be used to adjust the inspiratory/expiratory (I:E) ratio to the benefit of the patient. The patient can be taught how to breathe in a more physiologically normal pattern.

2. Indications

The following indications and guidelines are listed in the American Association for Respiratory Care (AARC) Clinical Practice Guidelines (1991, 2003) on IPPB:

5. Initiation of therapy

c. Giving an active treatment

Several authors advocate having the patient take an active treatment in which he or she interacts with the IPPB machine to obtain as deep a breath as possible.

Welch and colleagues (1980) have found that the patient’s posttreatment IC is greatest when the practitioner (1) uses as high a peak pressure as the patient can tolerate and (2) coaches the patient to inhale as deeply as possible with the IPPB machine. They and others believe that this is the best way to treat or prevent atelectasis. Monitor the patient for signs of barotrauma/volutrauma.

7. Initial settings on the Bennett PR-2

The PR-2 is used as the model respirator of the Bennett series. (Although the PR-2 is no longer being manufactured, many are still in clinical use.) Other Bennett units have slightly different controls and features. Refer to Figures 14-2 and 14-3 for the following:

Details on the design and control specifications for the various Bird and Bennett models can be found in the manufacturers’ literature and books on respiratory therapy equipment.

MODULE B

1. Change the patient-machine interface

c. Face mask

The face mask can be used if the mouth seal does not provide an airtight seal. This might be because of the patient’s facial structure or lack of teeth. Mouth trauma, surgery, or lip sores are other reasons to use a face mask.

The mask should be clear and properly sized to fit comfortably over the patient’s nose and mouth. The practitioner should be able to get a seal with a minimum amount of hand pressure (Figure 14-6). The equipment connection opening in the mask has a 22-mm inner diameter (ID) so that it connects directly to the IPPB circuit. A 22-mm-OD male adapter and short length of aerosol tubing can be added for flexibility and patient comfort. The clear mask is important so that the practitioner can see whether the patient has vomited or has a large amount of secretions or saliva in his or her mouth. The mask should never be strapped to the patient’s face so that the practitioner can attend to another patient.

This is the least effective patient attachment device if the therapeutic goal is to deliver an aerosolized medication. Much of the medication drains out on the patient’s face or in the nasal passages if he or she is a nose breather.

2. Improve patient synchrony (Code: IIIG3a) [Difficulty: ELE: R, Ap; WRE: An]

Synchrony refers to the patient breathing in a coordinated pattern with the IPPB unit. Because the patient has to interact with the unit, some practice is required. The respiratory therapist must coach the patient to alter his or her breathing pattern as well as adjust the controls on the IPPB unit. Usually this involves adjusting sensitivity and flow.

b. Adjust the flow

The patient should initially feel comfortable with the flow rate and the inspiratory time. Ideally, the flow should result in a smooth, steady rise in the pressure up to the preset maximum pressure. Reduce the flow if the pressure rises too quickly. Increase the flow if the pressure wavers higher and lower; this indicates the patient is breathing in faster than the gas is being delivered. Ask the patient a simple question such as, “Is the breath coming too fast or too slow?” He or she can give you a short answer or even a hand gesture in response. As the treatment progresses, the practitioner may be able to adjust the flow to modify the patient’s breathing pattern to better achieve the therapeutic goal, for example:

Turning the inspiratory time/flow rate control counterclockwise increases the flow rate on the Bird series. Pulling the air-mix knob out from the center body increases the total flow by allowing room air to be entrained along with the source gas. Flow rate in the PR-II is determined by the patient’s inspiratory effort and the degree to which the Bennett valve is open. Flow can be decreased somewhat by turning the peak flow control clockwise. Flow is not affected by the position of the air dilution knob.

3. Adjust the fractional concentration of inspired oxygen

Compressed air or oxygen can power both the Bird and Bennett units. The patient’s condition determines the oxygen percentage administered. The physician may include the oxygen percentage in the treatment order. Some departments have oxygen protocols in their treatment procedure. Generally, compressed air (21% oxygen) should be used whenever the patient does not need supplemental oxygen.

A patient with COPD who is retaining carbon dioxide and breathing on hypoxic drive should also be given room air via the IPPB unit. The patient may be allowed to keep wearing a nasal cannula with oxygen during the treatment so that the blood oxygen level is kept normal. The unavailability of piped-in compressed air should not be an excuse to give a patient a high oxygen percentage when it may be harmful. IPPB can be given through a gas-powered unit driven by a compressed air cylinder or through an electrically powered unit.

Supplemental oxygen is given if either unit is powered by oxygen and the air-mix/air dilution knobs are set to dilute the source gas with room air. This is appropriate for patients who require supplemental oxygen and are not at risk of stopping their spontaneous ventilation. Most practitioners use this method of giving IPPB because piped oxygen is usually available in all patient rooms.

Pure oxygen should be given to the patient who is severely hypoxemic. Examples include acute pulmonary edema, respiratory failure, and carbon monoxide poisoning. The air-mix/air dilution knobs must be set so that only the source gas (oxygen) is delivered to the patient.

For varying oxygen percentages on the Mark 7, note the following guidelines:

For varying oxygen percentages on the PR-II, note the following guidelines:

4. Adjust the volume, pressure, or both

A review of the current respiratory care textbooks reveals that all authors agree that the basic goal of IPPB is to increase how deeply the patient inspires. Unfortunately, there is considerable difference about what inspiratory volume is being measured or by how much that breath should be increased for therapeutic goals to be achieved. The AARC has released the following guidelines on the subject:

The AARC Clinical Practice Guideline (1991) on incentive spirometry: IPPB, rather than incentive spirometry, is indicated to treat atelectasis if (a) the patient’s IC is less than 33% of the preoperative value or (b) the patient’s VC is less than 10 mL/kg of ideal body weight.
The AARC Clinical Practice Guideline (1993) on IPPB: The tidal volume delivered during an IPPB-assisted breath should be at least 25% greater than the patient’s spontaneous breaths.

Therefore the patient should receive an IPPB-assisted tidal volume of at least 1122 mL.

If the therapeutic goal is to prevent or treat atelectasis, having the patient inspire a deeper than spontaneous tidal volume breath should help. It seems reasonable to follow the AARC guidelines as clinical goals. Following the 2003 updated guidelines would provide the patient with a larger IPPB-assisted tidal volume. However, depending on the patient’s tolerance, it may be necessary to begin with a smaller initial tidal volume goal (1993 guidelines) and then increase the goal as tolerated.

Because all of the current IPPB units are pressure cycled, the only way to increase the inspired volume during a passive treatment is to increase the peak pressure. Coaching the patient during an active treatment results in a larger volume without the need for as great a peak pressure. Decrease the peak pressure if the patient complains of discomfort or cannot hold that much pressure without losing the lip seal.

5. Reduce auto-PEEP (Code: IIIG3l) [Difficulty: ELE: R, Ap; WRE: An]

Positive end-expiratory pressure (PEEP) is pressure added through a mechanical ventilator at the end of an exhalation to prevent the patient from exhaling fully. PEEP keeps the patient’s airway pressure greater than atmospheric (commonly given as the baseline pressure of zero). The clinical effect of PEEP is to increase a patient’s residual volume (RV) and functional residual capacity (FRC) to improve oxygenation. It is used when the patient has a clinical condition, such as atelectasis or acute respiratory distress syndrome (ARDS), that results in small lung volumes. Auto-PEEP is end-expiratory pressure in the lungs that cannot be seen on the IPPB unit’s (or ventilator’s) pressure manometer. Auto-PEEP is caused by air trapping because the patient does not have enough time to exhale completely. In other words, the patient starts another inspiration before the previous breath was completely exhaled. This problem is most commonly seen in patients with small airways disease such as asthma and COPD.

Over the course of an IPPB treatment, patients who have small airways disease can develop air trapping. This can lead to an increased RV and FRC, which is seen as auto-PEEP. This unwelcome lung overinflation increases the risk of pulmonary barotrauma. If auto-PEEP is known or suspected during an IPPB treatment, expiratory retard can be added to help ensure a complete exhalation. Adding expiratory retard to the treatment increases back-pressure on the airways and has the same effect as pursed-lip breathing. The clinical effect of adding some back-pressure on the smallest airways is to keep them open longer so that the more distal air can be exhaled.

The following two procedures can be used to help identify the presence of auto-PEEP:

If auto-PEEP is identified, expiratory retard can be added to reduce it. Too little retard results in some air trapping and an incompletely exhaled tidal volume. Too much retard results in an uncomfortably long expiratory time and additional air trapping. This would cause an increased mean intrathoracic pressure. The proper amount of expiratory retard should result in the patient feeling comfortable with the breathing cycle and being able to completely exhale the delivered tidal volume. Listen to the patient’s breath sounds for a silent pause at the end of exhalation. If wheezing is present, it should be minimized when the proper amount of retard is added. This is because the back-pressure is properly adjusted to minimize small airway collapse and distal gas is fully exhaled.

Bird makes a retard cap that fits over the exhalation valve port on their permanent circuit. The cap has a series of different size holes through which the exhaled gas can pass (Figure 14-8). By rotating the cap progressively from the largest to the smallest opening and evaluating the patient at each setting, the proper size opening and amount of retard can be determined. The largest hole results in the least expiratory retard, while the smallest hole results in the greatest retard.

image

Figure 14-8 Bird retard cap for providing adjustable expiratory resistance.

(From McPherson SP: Respiratory therapy equipment, ed 5, St Louis, 1995, Mosby.)

Bennett makes a retard exhalation valve that can be substituted for the regular exhalation valve on their permanent circuit (Figure 14-9). The valve consists of a spring attached to a nut and a diaphragm. As the nut is turned counterclockwise the spring pushes the diaphragm closer to the exhalation valve opening. This causes resistance to the exhalation of the tidal volume and a back-pressure is created against the airways. Start with the least amount of expiratory retard and evaluate the patient before increasing the amount of expiratory retard. Be aware that if too much pressure is placed against the exhalation valve opening, the patient will not be able to exhale back to atmospheric pressure. This would create PEEP and should not be done without an order from the physician.

image

Figure 14-9 Bennett retard exhalation valve for providing adjustable expiratory resistance.

(From McPherson SP: Respiratory therapy equipment, ed 5, St Louis, 1995, Mosby.)

Be sure to ask the patient’s opinion about the use of expiratory retard. The conscious, cooperative patient can tell you if he or she feels like more air is getting out by the use of the retard or if the lungs feel more full because too much retard is being used. Too much retard may also make the expiratory time uncomfortably long.

MODULE C

2. Put the IPPB circuit together and make sure that it works properly

See Figure 14-10 for the Bird setup and Figure 14-11 for the Bennett setup.

MODULE D

2. Determine the appropriateness of the prescribed respiratory care plan and recommend modifications when indicated

c. Recommend changes in the therapeutic plan when indicated (Code: IIIH4) [Difficulty: ELE: R, Ap; WRE: An]

In order for the patient to receive the best care possible, the respiratory therapist must know the indications, contraindications, complications, and hazards of the respiratory care procedures the patient will receive. The patient must be assessed before, during, and after the treatment or procedure to determine if it was effective. The key goal of an appropriate care plan is that the patient’s condition be treated in the best way possible. Modifications to the care plan must be made, as needed, as the patient’s condition changes.

The respiratory therapist should be a member of the patient care team of the physician, nurse, and others in deciding how best to care for the patient. The following steps are necessary in developing the respiratory care plan for any patient:

The patient should be fully cooperative to make the best use of IPPB. It may be counterproductive to try to force an IPPB treatment on a combative or uncooperative patient. A patient who has a neuromuscular deficit may need assistance in holding the IPPB circuit or keeping a good mouth seal. A mouth seal or face mask treatment may have to be given. Be prepared to make a recommendation to have a patient use either incentive spirometry, short-term continuous positive airway pressure (CPAP), or IPPB for hyperinflation therapy. Also be prepared to make a recommendation for a patient to use either a metered dose inhaler, small volume nebulizer, or IPPB for the delivery of an aerosolized medication.

d. Terminate the IPPB treatment if the patient has an adverse reaction to it (Code: IIIF1) [Difficulty: ELE: R, Ap; WRE: An]

Patient safety should always be an important consideration during the treatment. The respiratory therapist should know the complications and hazards of any patient care activity that is performed. Be prepared to stop the treatment if the patient has a sudden adverse reaction to it (for example, hemoptysis or signs and symptoms of a pneumothorax). Additionally, be prepared to recommend to the physician that the treatment be discontinued if it is likely to result in additional serious adverse reactions.

However, some adverse reaction may only require a pause or adjustment in the IPPB treatment. For example:

3. Respiratory care protocols

BIBLIOGRAPHY

AARC Clinical Practice Guideline. Incentive spirometry. Respir Care. 1991;30:1402.

AARC Clinical Practice Guideline. Intermittent positive pressure ventilation. Respir Care. 1993;38:1189.

AARC Clinical Practice Guideline. Intermittent positive pressure ventilation—2003 revision & update. Respir Care. 2003;48:540.

Branson RD, Hess DR, Chatburn RL, editors. Respiratory care equipment, ed 2, Philadelphia: Lippincott Williams & Wilkins, 1999.

Cairo JM. Lung expansion devices. In Cairo JM, Pilbeam SP, editors: Mosby’s respiratory care equipment, ed 9, St. Louis: Mosby, 2010.

Eubanks DH, Bone RC. Comprehensive respiratory care, ed 2. St Louis: Mosby, 1990.

Fink JB. Volume expansion therapy. In Burton GG, Hodgkin JE, Ward JJ, editors: Respiratory care, ed 4, Philadelphia: Lippincott, 1997.

Fink JB. Bronchial hygiene and lung expansion. In: Fink JB, Hunt GE, editors. Clinical practice in respiratory care. Philadelphia: Lippincott Williams & Wilkins, 1999.

Fink JB. Volume expansion therapy. In Burton GG, Hodgkin JE, Ward JJ, editors: Respiratory care, ed 4, Philadelphia: Lippincott, 1997.

Fink JB. Hess DR: Secretion clearance techniques. In: Hess DR, MacIntyre NR, Mishoe SC, editors. Respiratory care principles & practices. Philadelphia: WB Saunders, 2002.

Fluck RJJr. Intermittent positive-pressure breathing devices and transport ventilators. In Barnes TA, editor: Respiratory care practice, ed 2, St Louis: Mosby, 1994.

McPherson SP. Respiratory care equipment, ed 5. St Louis: Mosby, 1995.

Miller WF. Intermittent positive pressure breathing (IPPB). In: Kacmarek RM, Stoller JK, editors. Current respiratory care. Philadelphia: BC Decker, 1988.

Respiratory Care Committee of the American Thoracic Society. Guidelines for the use of intermittent positive pressure breathing (IPPB). Respir Care. 1980;25:365.

Shapiro BA, Kacmarek RM, Cne RD, et al. Clinical application of respiratory care, ed 4. St Louis: Mosby, 1991.

Weizalis CP. Intermittent positive-pressure breathing. In Barnes TA, editor: Respiratory care practice, ed 2, St. Louis: Mosby, 1994.

Welch MA, et al. Methods of intermittent positive pressure breathing. Chest. 1980;78:463.

White GC. Equipment theory for respiratory care, ed 4. Albany, NY: Delmar, 2005.

Wilkins RL. Lung expansion therapy. In Wilkins RL, Stoller CL, Kacmarek RM, editors: Egan’s fundamentals of respiratory care, ed 9, St. Louis: Mosby, 2009.

SELF-STUDY QUESTIONS FOR THE ENTRY LEVEL EXAM See page 597 for answers

SELF-STUDY QUESTIONS FOR THE WRITTEN REGISTRY EXAM See page 622 for answers