Ear, nose and throat emergencies

Published on 10/02/2015 by admin

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Last modified 10/02/2015

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Ear, nose and throat emergencies

Introduction

Ear, nose or throat (ENT) conditions presenting at the Emergency Department (ED) are often trivialized, even though some can subsequently become life-threatening. For those patients who attend the ED with an ENT disorder, the onset of symptoms is likely to be acute or that the current episode may also be a feature of a chronic condition. It is important to be alert to the danger of viewing the patient only in terms of the presenting symptoms.

It is often the case that such conditions are accompanied by systemic illness precipitated by local infection. Equally important is the fact that the individual may have psychological, social and emotional needs as well as the presenting pathophysiological needs. This may be obvious in the case of an individual who has hearing loss as a direct result of being in close proximity to the seat of an explosion, but may be less apparent in the individual whose hearing loss results from wax impaction, but who is concerned that she may be becoming permanently deaf.

This chapter broadly examines ENT conditions in terms of infection, trauma and foreign bodies. The nursing care of patients is discussed in relation to presenting conditions.

The ear

The attendance at the ED of a patient with an ear-related problem is usually precipitated by one or more of the following symptoms:

Anatomy of the ear

The ear is divided into three sections: external, middle and inner ear (Fig. 32.1). The outer ear funnels sound into the middle ear, which serves to transmit the sound to the auditory apparatus of the inner ear. The external ear consists of the auricle (or pinna), ear canal and tympanic membrane. The S-shaped ear canal is approximately 2.5–3 cm long and terminates at the tympanic membrane. The canal is lined with glands that secrete cerumen, a yellow waxy material that lubricates and protects the ear. Ear wax, sloughed off skin cells and dust may impair sound transmission through the outer ear, especially if a plug of wax attaches to the eardrum. The bone behind and below the ear canal is the mastoid part of the temporal bone. The lowest portion of this, the mastoid process, is palpable behind the lobule (Bickley & Szilagyi 2003).

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Figure 32.1 Anatomy of the ear.

The tympanic membrane (or eardrum) is a thin, translucent, pearly grey oval disc separating the external ear from the middle ear. It can easily be observed with an otoscope. The tympanic membrane vibrates and moves in and out in response to sound. The middle ear is an air-filled cavity containing three tiny bones, the ossicles, which are individually called the malleus (hammer), the incus (anvil) and the stapes (stirrup), so named because of their appearance. The malleus is attached to the tympanic membrane by a set of ligaments. The incus is attached to the malleus and they move as one. The stapes attaches to the oval window, the membrane separating the middle and inner ear. When the tympanic membrane vibrates in response to sound, the malleus and incus are displaced, and the stapes vibrates against the oval window continuing the transmission of sound. The pharyngotympanic tube, formerly known as the Eustachian tube, which connects the middle ear with the nasopharynx, allows the passage of air to equalize pressure on either side of the tympanic membrane. The inner ear is composed of several fluid-filled chambers encased in a bony labyrinth in the temporal bone. The semicircular canals are also important for balance (Zemlin 2011).

Presentation to the ED may be prompted by a single symptom, such as hearing loss resulting from wax impaction. The patient may alternatively have multiple symptoms, resulting from, for example, an ear infection where pain, discharge and hearing loss may be present in combination with systemic illness.

Infections of the ear

Acute otitis externa

The external auditory meatus is a canal-shaped structure which extends from the external opening of the ear to the tympanic membrane. The integrity of the canal is protected from pathogens by its lining. The lateral one-third is composed of skin that is a continuation from the concha, which is the depression in the centre of the shell-shaped external structure of the ear – the pinna. The lining continues as an epithelial layer, protecting not only the medial two-thirds of the external auditory meatus but also the tympanic membrane.

The protective lining of the external auditory meatus may be easily breached by direct trauma, although pre-existing dermatological conditions, typically eczema and psoriasis, as well as external mediators such as maceration by water, may influence the resilience of the lining.

Clinical evidence and management: Acute otitis externa is essentially a localized or diffuse infection of the lining of the external auditory meatus commonly associated with organisms such as Pseudomonas aeruginosa, Staphylococcus aureus and occasionally fungi like Candida and Aspergillus (Sander 2001). Acute otitis externa frequently occurs following bathing or swimming because excessive moisture removes the protective cerumen from the ear canal allowing keratin debris to absorb water to create a nourishing environment for bacteria. For this reason it is often referred to as ‘swimmer’s ear’. Infection may be diffuse within the external auditory meatus or it may be focal in the form of a local swelling known as a furuncle, which may be extremely painful. Taking swabs for microbiological studies may not be well tolerated by the patient. It is essential that careful preparation of the patient takes place before any attempt is made to take a swab, especially if the individual is a child. Attempts to take a swab from an uncooperative child should be avoided as there is a risk that the tympanic membrane may be perforated by the swab if the child moves her/his head.

As the external auditory meatus contains no mucus-secreting cells, discharge from the ear is minimal; however, any discharge that does occur is usually thick and foul-smelling infected wax. The canal may also contain cell debris, which is unlikely to cause hearing loss, but may contribute to the intense irritation the individual may experience.

Treatment is based upon cleaning and drying the external auditory meatus. This should only be done after examination of the ear canal to determine the integrity of the tympanic membrane. Following cleansing of the external auditory meatus, topical medication containing steroids and antibiotics is necessary (Abelardo et al. 2007). Acute otitis externa largely results from identifiable causes and therefore lends itself to prevention strategies. The focus of much of the nursing care may revolve around educating the patient on keeping ears dry and on how to instill their prescribed medication.

Acute otitis media

An acute infection of the middle ear, that is, medial to the tympanic membrane, may cause pain, a feeling of pressure or fullness in the ear and hearing loss, the symptoms being caused by infective material splinting the tympanic membrane. Discharge from the external ear may be present, but in order for this to occur, the tympanic membrane must have been damaged, usually as a result of the increased pressure causing perforation.

Clinical evidence and management: Acute otitis media is often associated with systemic illness and fever, which may be attributed to the otitis media alone or occur in conjunction with coincidental upper respiratory tract infection (Ludman 2007). Acute otitis media is characterized by rapid onset of ear pain, headache, tinnitus, hearing loss, and nausea or vomiting. Infants and young children may present with irritability, crying, rubbing or pulling the ear, restless sleep and lethargy (Olson 2003). Children are often prone to acute otitis, with up to 30 % of those presenting with otitis media being children under three years of age, as the infection frequently results from upper respiratory tract infection of bacterial or viral origin.

Antibiotics are not often necessary in the treatment of uncomplicated otitis media with the mainstay of treatment being analgesia with antipyretic properties. Antibiotics in otitis media provide a modest benefit that must be balanced against the risk of adverse effects (Coker et al. 2010). In most cases involving children, antibiotics only provide symptomatic benefits after the first 24 hours, at which time symptoms are generally resolving. Serious complications, such as meningitis, mastoiditis, intracranial abscess, permanent hearing loss and neck abscess can develop as a result of otitis media (Olson 2003).

If the tympanic membrane has perforated, it is often the painful result of otitis media, trauma or foreign body insertion and is associated with loss of hearing. The individual should be advised to keep the ear dry and prevent water entering the ear. However, the ear should not be packed, and the patient should be advised not to do this at home, as it may prevent the discharge draining from the ear. More than 90% of tympanic membrane perforations heal spontaneously and management includes antibiotics, analgesia and antipyretics (Olson 2003). In some cases, where the tympanic membrane is intact, the infective material may cause the membrane to bulge, which also causes pain and loss of hearing. In such cases, admission to hospital is required in order that the tympanic membrane may be surgically perforated under general anaesthetic and grommets inserted to allow the discharge to drain out freely.

Mechanical obstruction

Impacted wax

The lateral one-third of the external auditory meatus contains cells that secrete a waxy substance called cerumen, the purpose of which is to act as a defense against dust and other foreign material entering the external auditory meatus.

Clinical evidence and management: Cerumen may build up in the external auditory meatus, causing mechanical obstruction, which may be exacerbated by cleaning the ear with cotton-tipped buds. Such activities often cause cerumen to be pushed deep into the canal, causing impaction against the tympanic membrane. Obstruction in either case may cause a reduction in hearing, but rarely causes complete deafness. Impacted cerumen is often hard and resistant to removal by syringing alone; thus, in the ED the most appropriate management is to initiate a regimen to soften the cerumen using commercially available eardrops.

Patient education involves self-administration with advice to contact their GP in 2–3 weeks to arrange for ear syringing. Ear syringing is rarely indicated in the ED. Poor technique and failure to take adequate precautions may cause the patient serious harm; it is therefore imperative that ear syringing is carried out by a nurse who is suitably trained in the technique.

Foreign bodies

Management: Foreign bodies may be removed using a variety of techniques including irrigation, suction and instrumentation, by individuals with the appropriate skills (Davies & Benger 2000). Care should be taken to ensure that this process does not impact the foreign body further in the ear, causing trauma to the external auditory meatus and the tympanic membrane.

If the tympanic membrane is intact then syringing the external auditory meatus with warm water may flush the foreign body out. However, this should only be carried out under direct visualization by those skilled in the technique.

Severe pain and distress are caused to patients when live insects enter the ear and they need to be killed in situ by the instillation of oil or lignocaine prior to removal (Davies & Benger 2000). Analgesic and/or antibiotic treatments should be prescribed as necessary.

Safe removal of a foreign body from the external auditory meatus requires a skilled operator and a cooperative patient, which is not always possible to achieve in the ED. If in any doubt, the patient should be referred to the ENT department. If the object is not retrieved at the first attempt, the patient should be referred to the ENT department.

Direct trauma

This is commonly caused by the insertion of objects either to clean the ear or to relieve itching, although any object inserted into the external auditory meatus has the potential to cause tympanic perforation. Objects frequently used are cotton-tipped buds and hair grips. In most cases, the ruptured tympanic membrane will heal spontaneously in 1–3 months (Bluestone 2007); however, ENT opinion should be sought. Pain relief and prophylactic antibiotics may be required, especially if the mechanism of injury includes contamination by water or a foreign body.

This provides the ED nurse with a health education opportunity in terms of prevention of subsequent episodes particularly in relation to aural hygiene. The importance of keeping the ear dry at all times must be stressed. A protective cotton plug coated with petroleum jelly will enable the patient to shower safely; however, swimming and generally getting the ears wet should be avoided.

Indirect trauma

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