Cardiovascular System

Published on 21/03/2015 by admin

Filed under Pediatrics

Last modified 22/04/2025

Print this page

rate 1 star rate 2 star rate 3 star rate 4 star rate 5 star
Your rating: none, Average: 0 (0 votes)

This article have been viewed 1408 times

Chapter 16. Cardiovascular System
Rationale
Approximately 50% of all children have innocent heart murmurs. Screening and referral of children with murmurs assists in distinguishing between innocent and organic murmurs. Assessment of cardiac and vascular function is an essential component of many hospitalizations, particularly when surgery is performed and when drugs are administered. When cardiac problems have been identified, knowledgeable assessment aids in monitoring the effectiveness of treatment regimens and in early detection of complications.
Anatomy and Physiology
The heart is a muscular four-chambered organ located in the mediastinum. The upper chambers of the heart are the atria; the lower chambers are the ventricles. Septa divide the two ventricles and the two atria. Four valves prevent the backflow of blood into the chambers. The tricuspid valve, located between the right atrium and ventricle, and the bicuspid, or mitral valve, located between the left atrium and ventricle, are the atrioventricular valves. The pulmonic valve, located in the pulmonary artery, and the aortic valve, in the aorta, are the semilunar valves. Closure of the four valves produces vibrations that are thought to be responsible for heart sounds. S1 refers to the “lubb” sound produced by closure of the atrioventricular valves, and S2 to the “dubb” sound produced by closure of the semilunar valves. Table 16-1 summarizes the various sounds and their characteristics.
Table 16-1 Heart Sounds
Sound Cause Location Characteristics
S1 (lubb) Mitral and tricuspid valves are forced closed at the beginning of systole (heart contraction). Apex of heart S1 is longer and lower pitched than S2.
Synchronous with carotid pulse.
Closure of valves usually heard as one sound, but slight asynchrony can produce audible splitting, best heard in the fourth left interspace.
S2 (dubb) Aortic and pulmonic valves are forced closed at the beginning of diastole (heart relaxation). Base of heart Short, high-pitched S2 can be split during inspiration. Splitting is best heard in the aortic area. If the breath is held on inspiration, “physiologic split” is accentuated.
S3 Vibrations are produced by rapid ventricular filling. Apex of heart Heard early in diastole. Dull, low pitched.
Normal in children and young adults.
S4 Resistance to ventricular filling after contraction of atria. Apex of heart Low pitched.
Considered abnormal. Best heard when child is supine.
In its initial stage of development the heart is a straight tube. Between the second and tenth weeks of gestation it undergoes a series of changes to become a four-chambered organ. The heart begins beating during the third week of gestation. During fetal life it primarily distributes the oxygen and nutrients that have been supplied through the placenta. The fetal lungs are largely bypassed by shunts that exist during fetal life. At birth these shunts begin to close as pulmonary vascular resistance drops. Pulmonary vascular resistance approximates adult levels by 6 weeks. Pulmonary vascular resistance is still relatively high in the first month of life, and cardiac defects such as ventricular septal defect might not be detected.
The heart is large in relation to body size in the infant. It lies somewhat horizontally and occupies a large portion of the thoracic cavity. Growth of the lungs causes the heart to assume a lower position, and by 7 years of age the heart has assumed an adult position that is more oblique and lower. Heart size increases in adolescence in conjunction with rapid growth.
At birth ventricular walls are similar in thickness, but with circulatory demands the left ventricle increases in thickness. The thinness of the ventricle produces a low systolic pressure in the newborn. The systolic pressure rises after birth until it approximates adult levels by puberty. Blood vessels lengthen and thicken in response to increased pressures.
Equipment for Assessment of Cardiovascular System
▪ Stethoscope (preferably with a small diaphragm)
Preparation
The child can sit or lie. Allow the child to handle the stethoscope. Listening to the parent’s or nurse’s heart or to their own hearts is often effective in dispelling anxieties in young patients. Ask the parent or child about heart disease in family members. Inquire whether the child has had difficulty in feeding (infant), undue fatigue, intolerance for exercise, poor weight gain, weakness, cyanosis, edema, dizziness, epistaxis, squatting, frequent respiratory tract infections, or delayed development. Ask the parent whether the mother had any infection or took medications during pregnancy, age of mother at infant’s birth, and presence of maternal diabetes or alcohol use. Inquire whether the child had problems at birth, such as low birth weight, prematurity, congenital infection, or respiratory difficulty. Inquire about temperament of the child and family responses to illness.
Assessment of Heart
Assessment Findings
Inspection
Observe the child’s body posture. Clinical Alert
Squatting is seen in tetralogy of
Fallot.
Persistent slight hyperextension of the neck in infants can indicate hypoxia.
Restlessness accompanied by abdominal pain, pallor, vomiting, unconsolable crying, and shock can indicate acute myocardial infarction in susceptible children.
Observe the child for cyanosis, mottling, and edema. Clinical Alert
Cyanosis, pallor, and mottling can indicate heart disease. Edema can indicate congestive heart failure. Edema of sacral and periorbital areas is more common in younger children.
Edema of the extremities is more common in older children but in the younger child can more likely indicate renal failure.
Cyanosis increases with crying in children with an atrioventricular canal defect and with some other cardiac defects.

Assessment Findings
Auscultate for additional sounds, such as S3 and and S4, which are best assessed with the infant or child lying on the left side. Assess for abnormal sounds such as clicks, murmurs, and precordial friction rubs. Murmurs should be evaluated and documented as to: S3 is a normal finding.
Clinical Alert
Innocent or nonpathologic murmurs do not increase over time and do not affect the child’s growth (Table 16-2). Innocent murmurs are usually systolic (Figure 16-2), medium-pitched, and best heard at the second and third left interspaces. Innocent murmurs can disappear with changes in position and can be accentuated during fever, stress, or exercise.
Location, or auscultatory area in which found.
Timing in the S1-S2 cycle
Intensity and whether intensity varies with the child’s position. Intensity is usually graded on a 6-point scale, with grade I being very faint and grade VI being audible even with the stethoscope off the chest. Grade III is considered moderately loud. PitchQuality (whether murmur is musical, blowing, or swishing) Precordial friction rubs are high-pitched grating or scratching sounds that are unaffected by breathing patterns. Pleural friction rubs stop when children hold their breath. Organic murmurs are caused by congenital or acquired heart disease. Murmurs occurring before 3 years of age are usually related to congenital defects, and after 3 years to rheumatic heart disease. (Table 16-3) provides descriptions of murmurs associated with cardiac defects. Murmurs associated with rheumatic heart disease include those of aortic and mitral stenosis and of aortic and mitral regurgitation. Additional sounds and murmurs must always be described and reported for further evaluation.

Assessment of vascular integrity is necessitated by cast application and by other conditions that can impair blood flow. Femoral and dorsal pedal areas should be palpated if cardiac defects are suspected.
Table 16-2 Innocent Murmurs
Adapted from Zator Estes ME: Health assessment & physical examination, ed 2, Clifton Park, 2002, Delmar Thomson Learning.
Type of Murmur Age Intensity Quality Other Characteristics
Pulmonary flow murmur (of the newborn) Low–birth-weight newborn; disappears by 3 to 6 months of age Grade I to IV (very faint to loud) Rough
Stills >2 years <Grade IV (loud) Squeaking, twanging, musical Best heard with child in supine position and with bell
Venous hum 3 to 6 years <Grade IV (loud) Continuous hum Heard best with child in upright position; disappears when supine and if head is turned
Pulmonary ejection 8 to 14 years <Grade IV (loud) Slightly grating
Table 16-3 Murmurs Associated with Childhood Cardiac Defects
Defect Location Timing Intensity Pitch Quality
Aortic stenosis Right second interspace (aortic area) Crescendo effect, occurring between S1 and S2 Variable Medium Harsh
Pulmonic stenosis Pulmonic area, third left interspace Crescendo effect, occurring between S1 and S2 Variable Medium Harsh
Mitral stenosis Mitral area Occurs between S2 and S1 Variable (Grade I to IV); can be accentuated by exercise Low Rumbling
Aortic regurgitation Aortic area Heard between S2 and S1; usually a short period follows S2 before sound begins Variable (Grade I to IV); most audible when child leans forward and exhales High; best heard with diaphragm of stethoscope Blowing Can be confused with breath sounds
Mitral regurgitation Mitral area Occurs between S1 and S2 Variable; unaffected by respiratory cycle High Blowing
Ventricular septal defect Left sternal border; third, fourth, and fifth interspaces Heard between S1 and S2 Very loud High Blowing
Patent ductus arteriosus Second left interspace Continuous; louder in late systole (just before S2); obscures S2; softer in diastole Loud Medium Harsh, machinery like
Tetralogy of Fallot Second and third left interspaces Heard between S1 and S2 Not well transmitted
Assessment Findings
Palpate the peripheral arteries for rhythm and for pulse rate, using the same finger. Use the same fingerpads to palpate the pulses simultaneously for equality. Normally pulses are palpable, equal in intensity, and even in rhythm.
Palpate the radial pulse.
The radial pulse is best felt in children older than 2 years of age and who do not evidence spasticity.
Palpate the femoral pulse by applying deep palpation midway between the iliac crest and the symphysis pubis. Clinical Alert
Diminution or absence of the femoral, popliteal, dorsalis pedis, or posterior tibial pulses can indicate coarctation of the aorta.
The child must be in the supine position (Figure 16-3). A brachial-femoral lag is abnormal, when the two pulses are palpated simultaneously.
Palpate the popliteal pulse by having the child flex the knee (Figure 16-4). Palpate the dorsalis pedis pulse along the upper medial aspect of the foot (Figure 16-5). Palpate the posterior tibial pulse posterior to the ankle on the medial aspect of the foot.
Anxiety: related to change in health status.
B0323044123500185/gr1.jpg is missing
Figure 16-1Cardiac auscultatory areas.(From Hockenberry MJ et al: Wong’s nursing care of infants and children, ed 7, St Louis, 2003, Mosby.)Elsevier Inc.
B0323044123500185/gr2.jpg is missing
Figure 16-2Timing of murmurs in S1-S2 cycle. A, Systolic murmur. B, Diastolic murmur.
B0323044123500185/gr3.jpg is missing
Figure 16-3Femoral pulse.(From Potter PA: Pocket guide to health assessment, ed 3, St Louis, 1994, Mosby.)Elsevier Inc.
B0323044123500185/gr4.jpg is missing
Figure 16-4Popliteal pulse.
B0323044123500185/gr5.jpg is missing
Figure 16-5Dorsalis pedis pulse.
Altered family processes: related to shift in health status of family member.
Fluid volume excess: related to compromised regulatory system.
Altered growth and development: related to chronic illness.
Knowledge deficit: related to diet, drug therapy, signs and symptoms of complications.
Risk for impaired skin integrity: related to fluid excess.
Impaired social interaction: related to fatigue, limited mobility.
Activity intolerance: related to imbalance between oxygen supply/demand.
Sleep pattern disturbance: related to shortness of breath.
Altered tissue perfusion, cardiopulmonary: related to edema, weak or absent pulses, blood pressure changes in extremities.
Decreased cardiac output: related to physical illnesses/anomalies.
Fatigue: related to poor physical condition.
Impaired physical mobility: related to limited cardiovascular endurance.
Altered parenting: related to physical illness, presence of stress.
Social isolation: related to altered state of illness.
Risk for altered development: related to complexity of therapeutic regimen.