Acute Care of the Victim of Multiple Trauma

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Chapter 66 Acute Care of the Victim of Multiple Trauma

Epidemiology

Injury is a leading cause of death and disability in children throughout the world (Chapter 5.1). According to the World Health Organization report on child injury prevention, unintentional injuries are one of the leading causes of death in children younger than 20 yr and the leading cause of death in children between 10 and 20 yr in the world. Road traffic accidents, drowning, fire-related events, and falls rank among the top causes of death and disability in children. In Asia, injury accounts for more than 50% of deaths in children <18 yr, with drowning accounting for approximately half. In the USA, more than 12,000 children die each year secondary to unintentional injury, with motor vehicle related injuries being the leading cause.

Deaths represent only a small fraction of the total trauma burden. Approximately 9.2 million children are treated in U.S. emergency departments (EDs) each year for injury, most commonly for falls. Many survivors of trauma have permanent or temporary functional limitations. Motor vehicle–related injuries and falls rank among the top 15 causes of disability-adjusted life years in children worldwide.

Trauma is frequently classified according to the number of significantly injured body parts (1 or more), the severity of injury (mild, moderate, or severe), and the mechanism of injury (blunt or penetrating). In childhood, blunt trauma predominates, accounting for the majority of injuries. In adolescence, penetrating trauma increases in frequency, accounting for approximately 15% of injuries, and has a higher case fatality rate.

Regionalization and Trauma Teams

Mortality and morbidity rates have decreased in geographic regions with comprehensive, coordinated trauma systems. Treatment at designated trauma centers is associated with decreased mortality. At the scene of injury, paramedics should administer necessary advanced life support and perform triage (Fig. 66-1; Tables 66-1 and 66-2). It is usually preferable to bypass local hospitals and rapidly transport a seriously injured child directly to a pediatric trauma center (or a trauma center with pediatric commitment). Children have lower mortality rates after severe blunt trauma when they are treated in designated pediatric trauma centers or in hospitals with pediatric intensive care units.

image

Figure 66-1 Field triage decision scheme—United States, 2006.

(Adapted from American College of Surgeons: Resources for the optimal care of the injured patient, Chicago, 2006, American College of Surgeons.) Footnotes have been added to enhance understanding of field triage by persons outside the acute injury care field:

*The upper limit of respiratory rate in infants is >29 breaths/min to maintain a higher level of overtriage for infants.

Trauma centers are designated Level I-IV, with Level I representing the highest level of trauma care available.

§Any injury noted in Steps 2 and 3 triggers a “yes” response.

Age <15 yr.

**Intrusion refers to interior compartment intrusion, as opposed to deformation, which refers to exterior damage.

††Includes pedestrians or bicyclists thrown or run over by a motor vehicle or those with estimated impact >20 mph with a motor vehicle.

§§Local or regional protocols should be used to determine the most appropriate level of trauma center; appropriate center need not be Level I.

¶¶Age >55 yr.

***Patients with both burns and concomitant trauma for whom the burn injury poses the greatest risk for morbidity and mortality should be transferred to a burn center. If the nonburn trauma presents a greater immediate risk, the patient may be stabilized in a trauma center and then transferred to a burn center.

†††Injuries such as an open fracture or fracture with neurovascular compromise.

§§§Emergency medical services.

¶¶¶Patients who do not meet any of the triage criteria in Steps 1-4 should be transported to the most appropriate medical facility as outlined in local EMS protocols.

Table 66-1 CHANGES IN FIELD TRIAGE DECISION SCHEME CRITERIA FROM 1999 TO 2006 VERSION*

STEP ONE: PHYSIOLOGIC CRITERIA

STEP TWO: ANATOMIC CRITERIA

STEP THREE: MECHANISM-OF-INJURY CRITERIA

STEP FOUR: SPECIAL CONSIDERATIONS

* Scheme is shown in Fig. 66-1.

Modified from Sasser SM, Hunt RC, Sullivent EE, et al: Guidelines for field triage of injured patients: recommendations of the National Expert Panel on Field Triage, MMWR Recomm Rep 58(RR-1):1–35, 2009. http://www.cdc.gov/mmwr/PDF/rr/rr5801.pdf.

When the receiving ED is notified before the child’s arrival, the trauma team should also be mobilized in advance. Each member has defined tasks. A senior surgeon (surgical coordinator) or, sometimes initially, an emergency physician leads the team. Team compositions vary somewhat from hospital to hospital; the model used at Children’s National Medical Center (Washington, DC) is shown in Figure 66-2. Consultants, especially neurosurgeons and orthopedic surgeons, must be promptly available; the operating room staff should be alerted.

Physiologic status, anatomic locations, and/or mechanism of injury are used for field triage as well as to determine whether to activate the trauma team (see Table 66-2). More importance should be placed on physiologic compromise and less on mechanism of injury. Scoring scales such as the Abbreviated Injury Scale (AIS), Injury Severity Score (ISS), Pediatric Trauma Score, and Revised Trauma Score (Table 66-3) use these parameters to predict patient outcome. The AIS and ISS are used together. First, the AIS is used to numerically score injuries—as 1 minor, 2 moderate, 3 serious, 4 severe, 5 critical, or 6 probably lethal—in each of 6 ISS body regions: head/neck, face, thorax, abdomen, extremity, and external. The ISS is the sum of the squares of the highest 3 AIS region scores.

Primary Survey

During the primary survey, the physician quickly assesses and treats any life-threatening injuries. The principal causes of death shortly after trauma are airway obstruction, respiratory insufficiency, shock from hemorrhage, and central nervous system injury. The primary survey addresses the ABCDEs: Airway, Breathing, Circulation, neurologic Deficit, and Exposure of the patient and control of the Environment.

Airway/Cervical Spine

Optimizing oxygenation and ventilation while protecting the cervical spine from potential further injury is of paramount importance. Initially, cervical spine injury should be suspected in any child sustaining multiple, blunt trauma. Children are at risk for such injuries because of their relatively large heads, which augment flexion-extension forces, and weak neck muscles, which predispose them to ligament injuries. To prevent additional spinal injury, the current standard is to immobilize the cervical (and thoracic and lumbar) spine in neutral position with a stiff collar, head blocks, tape or cloth placed across the forehead, torso, and thighs to restrain the child, and a rigid backboard.

Airway obstruction manifests as snoring, gurgling, hoarseness, stridor, and/or diminished breath sounds (even with apparently good respiratory effort). Children are more likely than adults to have airway obstruction because of their smaller oral and nasal cavities, proportionately larger tongues and greater amounts of tonsillar and adenoidal tissue, higher and more anterior glottic openings, and narrower larynxes and tracheas. Obstruction is common in patients with severe head injuries, owing in part to decreased muscle tone, which allows the tongue to fall posteriorly and occlude the airway. With trauma, obstruction can also result from fractures of the mandible or facial bones, secretions such as blood or vomitus, crush injuries of the larynx or trachea, or foreign body aspiration.

If it is necessary to open the airway, a jaw thrust without head tilt is recommended. This procedure minimizes cervical spine motion. In an unconscious child, an oropharyngeal airway can be inserted to prevent posterior displacement of the mandibular tissues. A semiconscious child will gag with an oropharyngeal airway but may tolerate a nasopharyngeal airway. A nasopharyngeal airway is contraindicated when there is a possibility of a cribriform plate fracture. If these maneuvers plus suctioning do not clear the airway, oral endotracheal intubation is indicated. When endotracheal intubation proves difficult, a laryngeal mask airway can be used as a temporary alternative. A laryngeal mask airway consists of a tube with an inflatable cuff that rests above the larynx and thus does not require placement of the tube into the trachea. Emergency cricothyrotomy is needed in <1% of trauma victims.

Breathing

The physician assesses breathing by counting the respiratory rate; visualizing chest wall motion for symmetry, depth, and accessory muscle use; and auscultating breath sounds in both axillae. In addition to looking for cyanosis, pulse oximetry is standard. If ventilation is inadequate, bag-valve-mask ventilation with 100% oxygen must be initiated immediately, followed by endotracheal intubation. End-expiratory carbon dioxide (CO2) detectors help verify accurate tube placement.

Head trauma is the most common cause of respiratory insufficiency. An unconscious child with a severe head injury may have a variety of breathing abnormalities, including Cheyne-Stokes respirations, slow irregular breaths, and apnea.

Although less common than a pulmonary contusion, tension pneumothorax and massive hemothorax are immediately life-threatening (Tables 66-4 and 66-5). Tension pneumothorax occurs when air accumulates under pressure in the pleural space. The adjacent lung is compacted, the mediastinum is pushed toward the opposite hemithorax, and the heart, great vessels, and contralateral lung are compressed or kinked (Chapter 405). Both ventilation and cardiac output are impaired. Characteristic findings include cyanosis, tachypnea, retractions, asymmetric chest rise, contralateral tracheal deviation, diminished breath sounds on the ipsilateral (more than contralateral) side, and signs of shock. Needle thoracentesis, followed by thoracostomy tube insertion, is diagnostic and lifesaving. Hemothorax results from injury to the intercostal vessels, lungs, heart, or great vessels. When ventilation is adequate, fluid resuscitation should begin before evacuation, because a large amount of blood may drain through the chest tube, resulting in shock.

Circulation

The most common type of shock in trauma is hypovolemic shock due to hemorrhage. Signs of shock include tachycardia; weak pulse; delayed capillary refill; cool, mottled, pale skin; and altered mental status (Chapter 64). Early in shock, blood pressure remains normal because of compensatory increases in heart rate and peripheral vascular resistance (Table 66-6). Some individuals can lose up to 30% of blood volume before blood pressure declines. It is important to note that 25% of blood volume equals 20 mL/kg, which is only 200 mL in a 10-kg child. Losses >40% of blood volume cause severe hypotension that, if prolonged, may become irreversible. Direct pressure should be applied to control external hemorrhage. Blind clamping of bleeding vessels, which risks damaging adjacent structures, is not advisable.

Cannulating a larger vein, such as an antecubital vein, is usually the quickest way to achieve intravenous access. A short, large-bore catheter offers less resistance to flow, allowing for more rapid fluid administration. Ideally, a second catheter should be placed within the first few minutes of resuscitation in a severely injured child. If intravenous access is crucial and not rapidly obtainable, an intraosseous catheter should be inserted; all medications and fluids can be administered intraosseously. Other alternatives are central venous access using the Seldinger technique (e.g., in the femoral vein) and, rarely, surgical cutdown (e.g., in the saphenous vein). Ultrasonography can facilitate central venous catheter placement.

Aggressive, intravenous fluid resuscitation is essential early in shock to prevent further deterioration. Isotonic crystalloid solution, such as lactated Ringer’s injection or normal saline (20 mL/kg), should be infused rapidly. No consensus exists to support the routine use of colloid or hypertonic (3%) saline solution. When necessary, repeated crystalloid boluses should be given. Most children are stabilized with administration of crystalloid solution alone. However, if the patient remains in shock after boluses totaling 40-60 mL/kg of crystalloid, then 10-15 mL/kg of cross-matched, packed red blood cells should be transfused. Although less desirable, type-specific or O-negative cells can be substituted pending availability of cross-matched blood. When shock persists despite these measures, surgery to stop internal hemorrhage is usually indicated.

Neurologic Deficit

Neurologic status is briefly assessed by determining the level of consciousness and evaluating pupil size and reactivity. The level of consciousness can be classified using the mnemonic AVPU: Alert, responsive to Verbal commands, responsive to Painful stimuli, or Unresponsive.

Head injuries account for approximately 70% of pediatric blunt trauma deaths. Primary direct cerebral injury occurs within seconds of the event and is irreversible. Secondary injury is caused by subsequent anoxia or ischemia. The goal is to minimize secondary injury by ensuring adequate oxygenation, ventilation, and perfusion, and maintaining normal intracranial pressure (ICP). A child with severe neurologic impairment—i.e., with a Glasgow Coma Scale (GCS; see Table 62-3) score of 8 or less—should be intubated.

Signs of increased ICP, including progressive neurologic deterioration and evidence of transtentorial herniation, must be treated immediately. Hyperventilation lowers PaCO2, resulting in cerebral vasoconstriction, reduced cerebral blood flow, and decreased ICP. Brief hyperventilation remains an immediate option for patients with acute increases in ICP. Prophylactic hyperventilation or vigorous or prolonged hyperventilation is not recommended, because the consequent vasoconstriction may excessively decrease cerebral perfusion and oxygenation. Mannitol lowers ICP and may improve survival. Because mannitol acts via osmotic diuresis, it can exacerbate hypovolemia and must be used cautiously. Hypertonic saline may be a useful agent for control of increased ICP in patients with severe head injury and may possibly decrease mortality when compared with mannitol. Neurosurgical consultation is mandatory. If signs of increased ICP persist, the neurosurgeon must decide whether to operate emergently.

Secondary Survey

During the secondary survey, the physician completes a detailed, head-to-toe physical examination.

Head Trauma

A GCS or Pediatric GCS score (see Table 62-3) should be assigned to every child with significant head trauma. This scale assesses eye opening and motor and verbal responses. In the Pediatric GCS, the verbal score is modified for age. The GCS further categorizes neurologic disability, and serial measurements identify improvement or deterioration over time. Patients with low scores 6-24 hr after injuries have poorer prognoses.

In the ED, CT scanning of the head without a contrast agent has become standard to determine the type of injury. Diffuse cerebral injury with edema is a common and serious finding on CT scan in severely brain-injured children. Focal evacuable hemorrhagic lesions (e.g., epidural hematoma) occur less commonly but may require immediate neurosurgical intervention (Fig. 66-3).

Monitoring of ICP should be strongly considered for children with severe brain injury, particularly for those with a GCS score of 8 or less and abnormal head CT findings (Chapter 63). An advantage of an intraventricular catheter over an intraparenchymal device is that cerebrospinal fluid can be drained to treat acute increases in ICP. Hypoxia, hypercarbia, hypotension, and hyperthermia must be aggressively managed to prevent secondary brain injury. Cerebral perfusion pressure should be maintained >40 mm Hg at least (although some experts recommend an even higher minimum).

A child with a severe brain injury must be treated aggressively in the ED because it is very difficult to accurately predict long-term neurologic outcome. Compared with adults with similar injuries, children are thought to have better functional outcomes.

Cervical Spine Trauma

Cervical spine injuries occur in <3% of children with blunt trauma—with the risk being substantially higher in those with GCS scores ≤8—but they are associated with significant mortality and morbidity. Bony injuries occur mainly from C1 to C4 in children younger than 8 yr. In older children, they occur equally in the upper and lower cervical spine. The mortality rate is significantly higher in patients with upper cervical spine injuries. Spinal cord injury without radiographic (vertebral body) abnormalities (SCIWORA) may be present. Patients with SCIWORA have neurologic symptoms, and spinal cord abnormalities are nearly always noted on MRI. Approximately 30% of all patients with cervical spine injuries have permanent neurologic deficits.

Evaluation begins with a detailed history and neurologic examination. Identifying the mechanism of injury helps in estimating the likelihood of a cervical spine injury. Both the patient and the paramedic should be asked whether any neurologic symptoms or signs, such as weakness or abnormal sensation, were present before arrival in the ED. In a child with neurologic symptoms and normal findings on cervical spine plain radiographs and CT scan, SCIWORA must be considered.

Whenever the history, physical examination, or mechanism of injury suggests a cervical spine injury, radiographs should be obtained after initial resuscitation. In adults, the Canadian C-spine rule helps identify low-risk patients who may not require radiographs (Fig. 66-4). The standard series of plain radiographs includes lateral, anteroposterior, and odontoid views. Some centers use cervical spine CT as the primary diagnostic tool, particularly in patients with abnormal GCS scores and/or significant injury mechanisms, recognizing that CT is more sensitive in detecting bony injury than plain radiographs. CT is also helpful if an odontoid fracture is suspected, because young children typically do not cooperate enough to obtain an “open-mouth” (odontoid) radiographic view. Use of cervical spine CT scan must be balanced with the knowledge that CT exposes thyroid tissue to 90-200 times the amount of radiation from plain films. MRI is indicated in a child with suspected SCIWORA.

Rapid diagnosis of spinal cord injury is essential. Initiating high-dose intravenous methylprednisolone within 8 hr of spinal cord injury has been shown to improve motor outcome and remains standard therapy.

Abdominal Trauma

Liver and spleen contusions, hematomas, and lacerations account for the majority of intra-abdominal injuries from blunt trauma. The kidneys, pancreas, and duodenum are relatively spared because of their retroperitoneal location. Pancreatic and duodenal injuries are more common after a bicycle handlebar impact or a direct blow to the abdomen (Table 66-8).

Although a thorough examination for intra-abdominal injuries is essential, achieving it often proves difficult. Misleading findings can result from gastric distention after crying or in an uncooperative toddler. Calm reassurance, distraction, and gentle, persistent palpation help with the examination. Important findings include distention, bruises, and tenderness. Specific symptoms and signs give insight into the mechanism of injury and the potential for particular injuries. Pain in the left shoulder may signify splenic trauma. A lap belt mark across the abdomen suggests a bowel or mesentery injury. The presence of certain other injuries, such as lumbar spinal fractures and femur fractures, increases the likelihood of intra-abdominal injury. Other risks are listed in Table 66-9.

Table 66-9 PREDICTION RULE FOR IDENTIFICATION OF CHILDREN WITH INTRA-ABDOMINAL INJURIES AFTER BLUNT TORSO TRAUMA

If any one of the following is present, the patient likely has intra-abdominal injury:

* Serum aspartate aminotransferase level >200 U/L or serum alanine aminotransferase level >125 U/L.

>5 red blood cells/high powered field.

Modified from Holmes JF, Mao A, Awasthi S, et al: Validation of a prediction rule for the identification of children with intra-abdominal injuries after blunt torso trauma, Ann Emerg Med 54:528–533, 2009.

An abdominal CT scan with intravenous contrast medium enhancement rapidly identifies structural and functional abnormalities and is the preferred study in a stable child. It has excellent sensitivity and specificity for splenic (Fig. 66-5), hepatic (Fig. 66-6), and renal injuries but is not as sensitive for diaphragmatic, pancreatic, or intestinal injuries. Small amounts of free fluid or air or a mesenteric hematoma may be the only sign of an intestinal injury. Administration of an oral contrast agent is not routinely recommended for all abdominal CT scans, but it sometimes aids in identifying an intestinal, especially a duodenal, injury.

Although focused assessment with sonography in trauma (FAST) examination helps detect hemoperitoneum, the variably low sensitivity of this test in children suggests that it should not be used to exclude intra-abdominal injury in patients with a high pretest probability for injury. Serial FAST exams over time may be used by skilled ultrasonographers to rule out injury in need of intervention. FAST is most useful in patients who have blunt trauma and are hemodynamically unstable or patients who require operative intervention for nonabdominal injuries, because in these cases the performance of a CT scan may not be feasible.

Nonoperative treatment has become standard for hemodynamically stable children with splenic, hepatic, and renal injuries from blunt trauma. The majority of such children can be treated nonsurgically. In addition to avoiding perioperative complications, nonoperative treatment decreases the need for blood transfusions and shortens hospital stay. When laparotomy is indicated, splenic repair is preferable to splenectomy.

Radiologic and Laboratory Evaluation

Some authorities recommend ordering multiple studies in the ED that include lateral cervical spine, anteroposterior chest, and anteroposterior pelvis radiographs; arterial blood gas analysis; serum lactate determinations; complete blood cell count; electrolyte measurements; blood glucose and blood urea nitrogen measurements; serum creatinine, amylase, and lipase determinations; liver function tests; prothrombin and partial thromboplastin time determinations; blood typing and cross-matching; and urinalysis. One benefit of standardizing the evaluation of patients with major trauma is that fewer decisions need to be made on an individual basis, possibly expediting ED management.

Some of these studies have prognostic importance. A large base deficit is associated with a higher mortality rate, and elevated lactate values correlate with poor prognosis.

There are limitations of standard tests. The lateral cervical spine radiograph can miss significant injuries. Hemoglobin and hematocrit values provide baseline values in the ED, but they may not have yet equilibrated after a hemorrhage. Abnormal liver function test results or elevated serum amylase and lipase values may be noted in patients with significant abdominal trauma, but most patients with significant trauma to the abdomen already have clinical indications for CT scanning or surgery. The majority of previously healthy children have normal coagulation profiles; these may become abnormal after major head trauma. Although routine urinalysis or dipstick urine testing for blood has been recommended for children, other data suggest that this evaluation may be unnecessary in patients without gross hematuria, hypotension, or other associated abdominal injuries.

Clinical prediction rules that combine patient history with physical exam findings have been developed to identify those at low risk of injury for whom specific radiographic and laboratory studies may not be necessary. The Canadian C-spine rule for adults is one such rule (see Fig. 66-4). In children, a clinical prediction rule for the identification of children with intra-abdominal injuries following blunt trauma has been validated. The presence of any of the risk factors in this prediction rule had a sensitivity of 95% for identifying children with intra-abdominal injury (see Table 66-9). Several clinical prediction rules have also been developed to predict traumatic brain injury following blunt head trauma. These rules, if successfully validated, may allow for more appropriate use of CT in pediatric trauma patients and may potentially reduce unnecessary radiation exposure.

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66.1 Care of Abrasions and Minor Lacerations

Lacerations and Cuts

Lacerations are tears of the skin caused by blunt or shearing forces. A cut (or a stab), in contrast, is an injury inflicted by a sharp object. Although distinguishing between the two can be important for forensic purposes, their evaluation and management are similar. In this chapter, lacerations include cuts and stabs.

Evaluation

The history should include the mechanism of injury, the amount of force, and the time the injury occurred. The mechanism helps determine whether there may be foreign material in the wound, which would increase the risk for infection. Particularly in children, it is essential to determine whether the injury was inflicted intentionally. If nonaccidental trauma is suspected, the state or local child protective services agency should be notified. The type of force causing the laceration also influences the risk of infection, as a significant crush injury is more likely to become infected than a shearing one. Blunt injury, such as bumping the head, is a common cause of lacerations in children and is less likely to become infected. Theoretically, the amount of bacteria in the wound should increase exponentially with the time from injury to repair; however, the length of time that results in a clinically significant increase in wound infection is unclear. The patient or parent should be asked about any special host factors that may predispose to infection or impede healing, such as diabetes, malnutrition, obesity, and steroid therapy.

The laceration’s location also is important with regard to both the risk for infection and the cosmetic outcome. Compared with lacerations in adults, those in children occur more commonly on the face and scalp and less commonly on the upper extremities. Because the face and scalp are more vascular, wounds located there are less likely to become infected. Lacerations overlying joints are more likely to develop wider scars as a result of tension during healing.

Treatment

The goals of treatment are to minimize the risk of infection, to restore skin and underlying tissue integrity, and to produce the most functionally and cosmetically acceptable result possible. In adults, the infection rate for uncomplicated lacerations is approximately 3-7%.

Any significant bleeding must be controlled (with external pressure usually) before a thorough evaluation of the wound can occur. If there is a skin flap, it should be returned to its original position before application of pressure. Clothing over the injury should be removed to minimize wound contamination. Jewelry encircling an injured extremity should be removed to prevent the jewelry from forming a constricting band when the extremity swells.

It is best to administer a local anesthetic early, before exploration and more meticulous cleansing of the wound. This anesthetic can be applied topically (e.g., lidocaine, epinephrine, and tetracaine gel) or infiltrated locally or as a regional nerve block (e.g., lidocaine or bupivacaine), depending on the location of the laceration and the complexity of the repair. Sometimes procedural sedation and analgesia are required for a young uncooperative child. The wound should be examined under proper light to allow identification of foreign bodies or damage to nerves or tendons.

Many lacerations, especially heavily contaminated ones, benefit from irrigation to reduce the risk of infection. It is important to recognize that many traumatic lacerations treated in the ED or office are only minimally contaminated, containing less than 102 bacterial colonies. In fact, in one of the few human studies on irrigation, irrigation did not decrease the infection rate of minimally contaminated scalp or facial lacerations in patients who presented to an ED within 6 hours of injury. Another concern is that higher-pressure irrigation may actually increase tissue damage, making the wound and adjacent tissue more susceptible to infection and delaying healing. These caveats notwithstanding, irrigation has benefits, although which technique to use—i.e., which device, what size syringe, what size needle, which solution, how much volume, how much pressure—remains to be determined. These features may vary for different types of lacerations. In heavily contaminated wounds, the benefit of higher-pressure irrigation likely outweighs the harm of tissue damage. For heavily contaminated lacerations, a typical recommendation is to use a 35- to 65-mL syringe attached to a plastic splatter shield, or a 19-gauge needle if a splatter shield is unavailable, and to irrigate with 250-1,000 mL of normal saline. Conversely, for relatively clean wounds, lower-pressure irrigation minimizes tissue damage, which may be more important for outcome than any decrease in bacterial clearance that may ensue. Debridement of devitalized tissue with higher-pressure irrigation, scrubbing, or surgical excision can also be necessary in certain cases, such as crush injuries.

Most lacerations seen in the pediatric ED or office should be closed primarily. Contraindications to primary closure (e.g., certain bite wounds) do exist (Chapter 705). Although it is commonly accepted that the time from injury to repair should be as brief as possible to minimize the risk of infection, there is no universally accepted guideline as to what length of time is too long for primary wound closure. Also, this length of time varies for different types of lacerations. A prudent recommendation is that higher-risk wounds should be closed within 6 hr at most after the injury but that some low-risk wounds (e.g., clean facial lacerations) may be closed as late as 12-24 hr.

Many lacerations can be closed with simple, interrupted, 4-0, 5-0, or 6-0, nonabsorbable sutures. For lacerations under tension, horizontal or vertical mattress sutures—which provide added strength and may evert the wound edges better—can be used instead. For lacerations in cosmetically significant areas, a running intradermal stitch may produce a less conspicuous, more aesthetic scar than simple or mattress skin sutures, which leave unattractive track marks. Deeper lacerations may need repair with an absorbable dermal and/or fascial layer. Other complex lacerations—e.g., those involving the ear, eyelid, nose, lip, tongue, genitalia, or fingertip—sometimes require more advanced techniques as well as subspecialty consultation.

Staples, topical skin adhesives, and surgical tape are acceptable alternatives to sutures, depending on the laceration’s location and the health care provider’s preference. Staples are particularly useful for lacerations of the scalp, where the appearance of the scar tends to be less important. Topical skin adhesives (e.g., octyl cyanoacrylates or butyl cyanoacrylates) are ideal for linear, relatively superficial lacerations, with easily approximated edges that are not under tension, particularly when these lacerations are located in areas where suture track marks are especially undesirable.

Maintaining a warm, moist, wound environment following repair accelerates wound healing without increasing the risk of infection. A topical antimicrobial ointment (e.g., bacitracin or a bacitracin, neomycin, and polymyxin B combination) and conventional gauze dressing provide such an environment and reduce the infection rate as well. Compared with conventional dressings, occlusive dressings (e.g., hydrocolloids, hydrogels, polyurethane films) may be better at accelerating healing, reducing infection, and decreasing pain but are more expensive; occlusive dressings that adhere (e.g., hydrocolloids or polyurethane films) are impractical for lacerations with protruding sutures. If the laceration overlies or is near a joint, splinting helps limit mobility and can speed healing and minimize dehiscence.

For most routine lacerations evaluated in the ED or office that are repaired early and carefully, prophylactic systemic antibiotics are unnecessary because they do not decrease the rate of infection. Antibiotic prophylaxis is or may be indicated for human and many animal bites, for open fractures and joints, and for grossly contaminated wounds, as well as for wounds in patients who are immunosuppressed or have prosthetic devices. Tetanus prophylaxis should be administered, if indicated, according to Centers for Disease Control and Prevention guidelines (Chapter 203).