75: Pulmonary Artery Catheter and Pressure Lines, Troubleshooting

Published on 06/03/2015 by admin

Filed under Critical Care Medicine

Last modified 06/03/2015

Print this page

rate 1 star rate 2 star rate 3 star rate 4 star rate 5 star
Your rating: none, Average: 2 (1 votes)

This article have been viewed 1413 times

PROCEDURE 75

Pulmonary Artery Catheter and Pressure Lines, Troubleshooting

PREREQUISITE NURSING KNOWLEDGE

• Knowledge of the cardiovascular anatomy and physiology is needed.

• Knowledge of the pulmonary anatomy and physiology is necessary.

• An understanding of basic dysrhythmia recognition and treatment of life-threatening dysrhythmias is important.

• Advanced cardiac life support knowledge and skills are needed.

• Knowledge of principles of aseptic technique is necessary.

• Understanding of the set-up of the hemodynamic monitoring system (see Procedure 76) is needed.

• Anatomy of the pulmonary artery (PA) catheter (see Fig. 73-1) and the location of the PA catheter in the heart and pulmonary artery (see Fig. 73-2) should be understood.

• Pulmonary artery occlusion pressure may be referred to as pulmonary artery wedge pressure.

• After wedging of the PA catheter, air is passively removed by disconnecting the syringe from the balloon-inflation port. Active withdrawal of air from the balloon is avoided because it can weaken the balloon, pull the balloon structure into the inflation lumen, and possibly cause balloon rupture.

• The pulmonary artery diastolic pressure (PADP) and the pulmonary artery occlusion pressure (PAOP) are indirect measures of left ventricular end-diastolic pressure (LVEDP). Usually, the PAOP is approximately 1 to 4 mm Hg less than the PADP. Because these two pressures are similar, the PADP is commonly followed, which minimizes the frequency of balloon inflation, thus decreasing the potential of balloon rupture.

• Differences between the PADP and the PAOP may exist for patients with pulmonary hypertension, chronic obstructive lung disease, adult respiratory distress syndrome, pulmonary embolus, and tachycardia.

• Pulmonary artery pressures (PAPs) may be elevated because of pulmonary artery hypertension, pulmonary disease, mitral valve disease, left ventricular failure, atrial or ventricular left-to-right shunt, pulmonary emboli, or hypervolemia.

• PAPs may be decreased because of hypovolemia or vasodilation.

• The waveforms that occur during insertion should be recognized, including right atrial (RA), right ventricular (RV), PA, and pulmonary artery occlusion (PAO; see Fig. 73-3).

• a wave reflects atrial contraction. The c wave reflects closure of the atrioventricular valves. The v wave reflects passive filling of the atria during ventricular systole (see Figs. 73-4 and 73-5).

• Knowledge of normal hemodynamic values (see Table 67-1) is needed.

• Elevated a and v waves may be evident in RA or central venous pressure (CVP) and in PAO waveforms. These elevations may occur in patients with cardiac tamponade, constrictive pericardial disease, and hypervolemia.

• Elevated a waves in the RA or CVP waveform may occur in patients with pulmonic or tricuspid stenosis, right ventricular ischemia or infarction, right ventricular failure, pulmonary artery hypertension, and atrioventricular (AV) dissociation.

• Elevated a waves in the PAO waveform may occur in patients with mitral stenosis, acute left ventricular ischemia or infarction, left ventricular failure, and AV dissociation.

• Elevated v waves in the RA or CVP waveform may occur in patients with tricuspid insufficiency.

• Elevated v waves in the PAO waveform may occur in patients with mitral insufficiency or ruptured papillary muscle.