The Elbow

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Chapter 6 The Elbow

The elbow is a strong hinge joint that allows flexion and rotation of the forearm. It also provides the bony origin for most of the extrinsic muscles of the wrist and hand. It is frequently affected by inflammatory and traumatic conditions that seriously alter its function. Osteoarthritis is rare, however.

Anatomy

The elbow joint is formed by the articulation between the humerus and the radius and ulna (Fig. 6-1). The humerus widens distally to form the lateral and medial condyles. The capitellum of the lateral condyle articulates with the radial head, and the trochlea articulates with the ulna. The head of the radius also articulates with the lateral aspect of the ulna and is held in position by the orbicular ligament. Medial and lateral collateral ligaments provide additional stability.

Adjacent to each condyle are the epicondyles, which are the bony attachments for many forearm muscles. The flexor–pronator muscle group takes its origin from a common tendon that attaches to the medial epicondyle, and the extensor–supinator group arises in a similar manner from the lateral epicondyle. Posteriorly, the triceps attaches to the olecranon; anteriorly, the biceps and brachialis attach to the radius and ulna, respectively.

Three major nerves cross the elbow joint on their way into the forearm. The median nerve passes deep in the antecubital fossa medial to the biceps and brachialis, and the radial nerve passes lateral to them. The ulnar nerve reaches the forearm by coursing posteriorly in a groove between the medial epicondyle and the olecranon process, where it is easily palpated. It is also vulnerable to injury in this superficial location.

Epicondylitis

Epicondylitis is one of a large group of musculoskeletal disorders commonly termed “overuse syndromes.” Although it is often called an inflammatory condition, degeneration (tendinosis) is usually present instead, often with the development of local neovascular tissue. The condition is characterized by pain at the origin of the flexor muscles at the medial epicondyle or the extensor muscles at the lateral epicondyle. Some cases may start with a direct blow, but the cause is usually unknown. Minor tears in the tendinous attachments of these muscles are often present. The disorder is common in individuals whose activities require repeated use of the extensor or flexor mechanism of the forearm. The lateral side (“tennis elbow”) is more commonly involved. In tennis players, the backhand swing seems to be the main offender. Involvement of the medial epicondyle is often called “golfer’s elbow.”

TREATMENT

Treatment is similar to that for other “musculotendinous overuse syndromes.” Rest is important, and this can frequently be obtained merely by avoiding the offending activity. Applying ice after exercise can help. A careful exercise program of gentle stretching and strengthening is begun as pain subsides (Fig. 6-6). Nonsteroidal anti-inflammatory drugs (NSAIDs) are given as necessary. Local infiltration of the affected area with 1 to 2 mL of a steroid/lidocaine mixture often provides permanent or at least long-lasting relief (Fig 6-7). The injection is placed in the area of maximum local tenderness, usually about 1 cm distal to the bony epicondyle, and may be repeated two or three times. A tennis elbow counterforce strap may also be tried. It theoretically works by dampening the force transmitted to the elbow from the hand and wrist (see Chapter 15). Extracorporeal shock wave therapy (ESWT) has even been tried in epicondylitis, but the results are inconclusive. The disease is usually self-limited, but symptoms may persist for several months before full recovery. Conservative treatment is effective in most cases. Often, spontaneous rupture of the aponeurosis probably occurs, which cures the pain, usually without any significant residual weakness. Surgery is reserved for cases that do not respond to medical management. Eventually, the recreational tennis player may simply have to decide to withdraw from the sport.

Osteochondritis Dissecans

Osteochondritis dissecans is a condition in which a portion of subchondral bone undergoes avascular necrosis. This segment of bone, with its overlying articular cartilage, may partially or completely separate from the adjacent bone and even extrude into the joint to form a loose body. The disorder is most commonly seen in the knee joint, but a similar condition also occurs in the elbow, ankle, and hip joints. The cause is unknown, but it is probably traumatic in origin. Repetitive compression of the lateral elbow joint may be responsible. The condition is sometimes a cause of “Little League elbow.”

Pronator Syndrome

The median nerve gives off its motor branch, the anterior interosseous nerve, below the elbow as it passes between the two heads of the pronator teres muscle just behind the biceps aponeurosis. An uncommon form of compression neuropathy can occur at this site, which is called pronator syndrome. Entrapment can also cause a very specific clinical presentation, sometimes referred to as the anterior interosseous nerve syndrome if only the motor branch is affected. The etiology is usually some form of localized anatomic compression, but the disorder can follow injury or even a traumatic phlebotomy.

Clinical features include forearm discomfort and fatigue, often resulting from repetitive pronation. The onset is insidious, and nocturnal paresthesias are not typical, in contrast to carpal tunnel syndrome. Vague hand numbness in the median nerve distribution may be present, but sensory changes usually do not develop if only the motor branch is involved.

If only the anterior interosseous branch is affected, sensation to the hand is normal. Forearm pain and weakness may develop, and if sufficient compression has occurred, weakness of the flexor pollicis longus (FPL) and flexor digitorum profundus (FDP) to the index finger may be present. The patient may then be unable to form a circle when trying to pinch the index and thumb because of inability to flex the distal phalanges of those fingers (Fig. 6-9).

Electrodiagnostic studies may be helpful, although results of such studies are often normal. Plain radiographs should be obtained to rule out bony abnormalities that could cause compression.

Treatment is directed toward identifying and eliminating any known causative factors. Rest, stretching exercises, and NSAIDS may be helpful. Surgical referral is indicated in cases of failed medical management or when motor weakness is present.

NOTE: This condition is often difficult to diagnose. Sometimes, the anatomic site of compression can only be determined at the time of surgery when all potential sites are explored and decompressed.

Pulled Elbow

Pulled elbow, or “nursemaid’s elbow,” is a disorder in which the head of the radius becomes subluxed beneath the orbicular ligament (Fig. 6-10). This occurs as a result of longitudinal traction on the hand with the elbow extended and the forearm in a pronated position. A common situation in which this occurs is when a child is lifted up by the wrist or hand.

Olecranon Bursitis

The olecranon bursa overlies the olecranon process and is extremely vulnerable to direct trauma and repeated irritation. It may also be involved in gout and rheumatoid arthritis. After an acute traumatic episode, usually a contusion, a tender, painful swelling may develop over the tip of the olecranon. Elbow motion is usually normal. The bursa sac fills with blood or clear fluid. At this stage, aspiration (with culture if fluid is suspicious for infection) of the fluid with application of a compression dressing and ice may prevent reformation of the fluid and recurrence. Many acute lesions will spontaneously subside, however. Roentgenograms should be obtained to rule out fracture of the olecranon if trauma is involved.

With repetitive trauma, a chronic inflammatory reaction may occur that results in the formation of a thickened, rubbery bursa (Fig. 6-13). This bursa is usually not painful, in contrast to the swelling that occurs after an acute injury. Palpation may reveal multiple, small, hard nodules that feel like loose bodies. These usually are not chips of bone but instead represent villous thickenings or tissue debris in the bursa. Aspiration of the bursa may be attempted, but the fluid often recurs. Resolution may be improved by intrabursal injection of 1 mL of steroid (Fig. 6-14). Incision is not recommended because a chronic draining sinus infection often results. If the bursa is chronically painful, excision is recommended. Usually, the bursa will “dry up” eventually on its own, however. Elbow pads or other forms of protection may be helpful.

Occasionally, the bursa is the site of an acute infection, usually staph, that sometimes follows trauma. The treatment is the same as that for any other infection and consists of antibiotics, moist heat, and splinting, with repeated aspirations or incision and drainage when necessary.

Dislocation of the Elbow

Dislocation of the elbow is a common injury and is usually posterior in direction (Fig. 6-15). It is generally the result of a fall on the outstretched hand with the elbow extended. Examination will reveal obvious deformity that must be differentiated from a supracondylar fracture. Avulsion fractures of the medial epicondyle and fractures of the radial head occasionally occur at the same time that may require surgical intervention. Reduction is performed as soon as possible. It can usually be accomplished by gentle, steady traction on the wrist with countertraction on the shoulder (Fig. 6-16). A general anesthetic is usually unnecessary. Extension of the elbow to unlock the olecranon may be necessary. After reduction, the elbow is tested for stability, and postreduction roentgenograms are always obtained. Lack of full motion following reduction suggests the possibility of an intraarticular fracture fragment. If the elbow is stable after reduction, it is rested in a sling for a few days. Gentle range-of-motion exercises are instituted as early as possible. Temporary stiffness is common, and full recovery of elbow motion may take several weeks. Motion should never be forced. The patient should be allowed to progress as tolerated. Forced passive motion only encourages more swelling, which leads to more stiffness. Some residual restriction of motion is not uncommon, but it is usually of such a minor degree that it does not interfere with function.

Dislocation of the Radial Head in Children

This is a disorder that is usually traumatic but may occasionally be congenital or developmental in nature. Most traumatic cases dislocate anteriorly (Fig. 6-17). The mechanism of injury is usually a fall on the outstretched pronated arm, and the injury is frequently missed. Some cases may be associated with a “bent” ulna, suggesting a Monteggia type of injury. Congenital dislocations are usually posterior and are frequently bilateral. They may be associated with other congenital anomalies such as Ehlers–Danlos syndrome. Developmental dislocations are usually the result of cerebral palsy or neurologic injury and are commonly posterolateral. These two types of radial head dislocations usually have no pain and little functional impairment. No treatment is required unless symptoms are present.

Traumatic dislocations, however, need early reduction to prevent stiffness and pain. Old neglected dislocations may benefit from surgery.

Recognition is important. Remember: a line drawn through the long axis of the radius should always pass through the capitellum in any view, and all articular surfaces should match together perfectly.

Fractures of the Elbow Region

FRACTURES OF THE HEAD AND NECK OF THE RADIUS

Fractures of the radial head and neck result from a fall on the outstretched hand with the elbow extended (Fig. 6-18). All are characterized by tenderness over the radial head, local swelling, and pain on rotation or flexion of the forearm.

Undisplaced or minimally displaced fractures in adults and children are treated conservatively. If the swelling is extremely painful, the joint may be aspirated through the posterolateral triangle. A sling alone may be used, or a light posterior splint may be added with the elbow flexed 90 degrees. Protection beyond 7 to 10 days is usually not required, and early motion is encouraged. (Undisplaced radial head fractures are sometimes not easily visualized on initial roentgenograms. If there is a clinical suspicion of fracture, such as radial head tenderness and pain with rotation, treatment is indicated.) Full return of motion often takes several months. Large fragments may require open reduction with internal fixation.

Significantly comminuted fractures in adults are usually treated by early excision of the entire radial head. Otherwise, permanent restriction of joint motion and traumatic arthritis may result. Early removal is especially indicated in grossly comminuted and displaced fractures because the fracture fragments may act as a nidus for soft tissue calcification in the anterior elbow region, and myositis ossificans may result (Fig. 6-19). Open reduction may be considered in fractures with a single large displaced fragment.

Children’s fractures with less than 15 to 30 degrees of angulation are treated as undisplaced fractures with a long arm splint for 3 weeks. Displaced fractures, or fractures that are angulated greater than 15 to 30 degrees, are treated by closed or open reduction. However, the radial head is never removed in the growing child, because removal of the epiphysis will result in unequal growth of the forearm bones.

Regardless of the method of treatment, some mild loss of extension of the elbow is not uncommon. However, little functional impairment usually results.

BIBLIOGRAPHY

Ackerman G, Jupiter JB. Nonunion of fractures of the distal end of the humerus. J Bone Joint Surg Am. 1988;70:75-83.

Adams JE. Bone injuries in very young athletes. Clin Orthop. 1968;58:129-140.

Ashe MC, McCauley T, Khan KM. Tendinopathies in the upper extremity: a paradigm shift. J Hand Ther. 2004;17:329-334.

Baker BE, Bierwagen D. Rupture of the distal tendon of the biceps brachii. Operative versus non-operative treatment. J Bone Joint Surg Am. 1985;67:414-417.

Blount WP. Fractures in children. Baltimore: Williams & Wilkins, 1955.

Boyd HB, McLeod ACJr. Tennis elbow. J Bone Joint Surg Am. 1973;55:1183-1187.

Broberg MA, Morrey BF. Results of delayed excision of the radial head after fracture. J Bone Joint Surg Am. 1986;68:669-674.

Cain ELJr, Dugas JR, Wolf RS, et al. Elbow injuries in throwing athletes: a current concepts review. Am J Sports Med. 2003;31:621-635.

Chen FS, Rokito AS, Jobe FW. Medial elbow problems in the overhead-throwing athlete. J Am Acad Orthop Surg. 2001;9:99-113.

Chung B, Wiley JP. Effectiveness of extracorporeal shock wave therapy in the treatment of previously untreated lateral epicondylitis: a randomized controlled trial. Am J Sports Med. 2004;32:1660-1667.

Ciccotti MG, Charlton WP. Epicondylitis in the athlete. Clin Sports Med. 2001;20:77-93.

Cohen MS, Hastings H. Acute elbow dislocation: evaluation and treatment. J Am Acad Orthop Surg. 1998;6:15-23.

Dobbie RP. Avulsion of the lower biceps brachii tendon: analysis of 51 previously unreported cases. Am J Surg. 1941;51:662.

Galloway M, DeMais M, Mangine R. Rehabilitation techniques in the treatment of medial and lateral epicondilitis. Orthopedics. 1992;15:1089-1099.

Garden RS. Tennis elbow. J Bone Joint Surg Br. 1961;43:100.

Haake M, Konig IR, Decker T, et al. Extracorporeal shock wave therapy in the treatment of lateral epicondylitis: a randomized multicenter trial. J Bone Joint Surg Am. 2002;84-A:1982-1991.

Hak DJ, Golladay GJ. Olecranon fractures: treatment options. J Am Acad Orthop Surg. 2000;8:266-275.

Hotchkiss RN. Displaced fractures of the radial head: internal fixation or excision? J Am Acad Orthop Surg. 1997;5:1-10.

Hudson DA, De Beer JD. Isolated traumatic dislocation of the radial head in children. J Bone Joint Surg Br. 1986;68:378-381.

Ikeda M, Sugiyama K, Kang C, et al. Comminuted fractures of the radial head. Comparison of resection and internal fixation. J Bone Joint Surg Am. 2005;87:76-84.

Jobe FW, Giccotti MG. Lateral and medial epicondylitis of the elbow. J Am Acad Orthop Surg. 1994;2:1-8.

Kobayashi K, Burton KJ, Rodner C, et al. Lateral compression injuries in the pediatric elbow: Panner’s disease and osteochondritis dissecans of the capitellum. J Am Acad Orthop Surg. 2004;12:246-254.

Kragh JFJr, Basamania CJ. Surgical repair of acute traumatic closed transection of the biceps brachii. J Bone Joint Surg Am. 2002;84-A:992-998.

Letts M, Locht R, Wiens J. Monteggia fracture-dislocations in children. J Bone Joint Surg Br. 1985;67:724-727.

Lindholm TS, Osterman K, Vankka E. Osteochondritis dissecans of elbow, ankle, and hip: a comparison survey. Clin Orthop Relat Res. 1980;148:245-253.

Lloyd-Roberts 1977 Lloyd-Roberts GC, Bucknill TM. Anterior dislocation of the radial head in children: aetiology, natural history and management. J Bone Joint Surg Br. 1977;59-B:402-407.

March HC. Osteochondritis of the capitellum (Panner’s disease). AJR Am J Roentgenol. 1944;51:682. B

Mehlhoff TL, Noble PC, Bennett JB, et al. Simple dislocation of the elbow in the adult. Results after closed treatment. J Bone Joint Surg Br. 1988;70:244-249.

Morrey B. Current concepts in the treatment of fractures of the radial head, the olecranon, and the coronoid process. J Bone Joint Surg Am. 1995;77:316-327.

Nirschl RP, Ashman ES. Tennis elbow tendinosis (epicondylitis). Instr Course Lect. 2004;53:587-598.

O’Donoghue 1976 O’Donoghue DH. Treatment of injuries to athletes, ed 3. Philadelphia: WB Saunders, 1976.

Otsuka NY, Kasser JR. Supracondylar fractures of the humerus in children. J Am Acad Orthop Surg. 1997;5:19-26.

Park GY, Kim JM, Lee SM. The ultrasonographic and electrodiagnostic findings of ulnar neuropathy at the elbow. Arch Phys Med Rehabil. 2004;85:1000-1005.

Pettrone FA, McCall BR. Extracorporeal shock wave therapy without local anesthesia for chronic lateral epicondylitis. J Bone Joint Surg Am. 2005;87:1297-1304.

Pirone AM, Graham HK, Krajbich JI. Management of displaced extension-type supracondylar fractures of the humerus in children. J Bone Joint Surg Am. 1988;70:641-650.

Ramsey ML. Distal biceps tendon injuries: diagnosis and treatment. J Am Acad Orthop Surg. 1999;7:199-207.

Reckling FW. Unstable fracture-dislocations of the forearm (Monteggia and Galeazzi lesions). J Bone Joint Surg Am. 1982;64:857-863.

Ring DR, Jupiter JB, Waters PW. Monteggia fractures in children and adults. J Am Acad Orthop Surg. 1998;6:215-224.

Roberts N, Hughes R. Osteochondritis dissecans of the elbow joint; a clinical study. J Bone Joint Surg Br. 1950;32-B:348-360.

Salter RB. Disorders and injuries of the musculoskeletal system. Baltimore: Williams & Wilkins, 1970.

Rompe JD, Decking J, Schoellner C, et al. Repetitive low-energy shock wave treatment for chronic lateral epicondylitis in tennis players. Am J Sports Med. 2004;32:734-743.

Skaggs DL. Elbow fractures in children: diagnosis and management. J Am Acad Orthop Surg. 1997;5:303-312.

Smidt N, van der Windt DA, Assendelft WJ, et al. Corticosteroids, physiotherapy, or a wait-and-see policy for lateral epicondylitis: a randomised controlled trial. Lancet. 2002;359:657-662.

Tullos HS, King JW. Lesions of the pitching arm in adolescents. JAMA. 1972;220:264-271.

Wang CJ, Chen HS. Shock wave therapy for patients with lateral epicondylitis of the elbow: a one- to two-year follow-up study. Am J Sports Med. 2002;30:422-425.