Scuba Diving–Related Disorders

Published on 14/03/2015 by admin

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51

Scuba Diving–Related Disorders

The disorders related to scuba diving include those caused by environmental exposure (see Chapters 3 and 50), dysbarism, nitrogen narcosis, contaminated breathing gas, decompression sickness (DCS), and hazardous marine life (see Chapters 52 and 53) (Box 51-1).

Dysbarism

Dysbarism encompasses all the pathologic changes caused by altered environmental pressure. At sea level, atmospheric pressure is 760 mm Hg (14.7 psi). Each 10-m (32.8-foot) descent under water increases the pressure by 1 atm. Gas in enclosed spaces obeys Boyle’s law, which states that the pressure of a given quantity of gas when its temperature remains unchanged varies inversely with its volume.

Mask Squeeze

An air space is present between the face and the glass of a scuba (self-contained underwater breathing apparatus) diving mask. If nasal exhalations do not maintain air pressure within this space during descent, the volume of air contracts, creating negative pressure. This leads to capillary rupture, which is potentially dangerous after keratotomy because of the slow healing rate of corneal incisions.

Ear Canal Squeeze

A tight-fitting wet suit hood, earplugs, exostoses, or cerumen impaction can trap air in the external auditory canal. On descent, this air contracts in the enclosed space between the tympanic membrane and the (occluded) external opening of the ear.

Middle Ear Squeeze (Barotitis Media)

If air cannot enter the middle ear via the (contracted or blocked) eustachian tube during an underwater descent, the existing air in the middle ear space contracts, creating a relative vacuum and pulling the tympanic membrane inward (Fig. 51-1).

Treatment

1. Before tympanic membrane rupture, administer an oral decongestant and a long-acting topical decongestant nasal spray such as oxymetazoline. In a severe case, if the tympanic membrane is intact, a short course of prednisone (50 mg PO, tapered over 7 days) may be helpful. An antihistamine may be administered if there is an allergic component.

2. Repeated gentle autoinflation of the middle ear by use of the Frenzel maneuver may help to displace any collection of middle ear fluid through the eustachian tube.

3. For tympanic membrane rupture, administer an antibiotic such as amoxicillin/clavulanate for 7 days. In addition, administer fluoroquinolone otic drops. Suspend all diving activities until the tympanic membrane is fully healed or has been surgically repaired and eustachian tube function allows easy autoinflation.

Barosinusitis

Barosinusitis, or “sinus squeeze,” results from an inability to inflate a paranasal sinus during descent, at which time contraction of the trapped air creates a relative vacuum. This damages the sinus wall mucosa, which ultimately hemorrhages. Less often, a “reverse sinus squeeze” can occur on ascent in the water because the expanding air cannot be vented from the sinus.

Pulmonary Barotrauma of Ascent (Pulmonary Overpressurization Syndrome)

Pulmonary barotrauma of ascent results from expansion of gas trapped in the lungs, which ruptures alveoli or is forced across the pulmonary capillary membrane.

Arterial Gas Embolism

Arterial gas embolism results from air bubbles entering the pulmonary venous circulation from ruptured alveoli. Gas bubbles are showered into the heart, from which they may be distributed to the coronary and carotid arteries. Arterial gas embolism typically develops immediately after a diver surfaces.

Treatment

1. Transport the patient for recompression treatment in a hyperbaric (oxygen) chamber.

2. Maintain the patient in a supine position.

3. Administer oxygen, 5 to 15 L/min, by nonrebreather mask.

4. Begin an intravenous infusion of isotonic solution to maintain urine output at 1 to 2 mL/kg/hr.

5. Obtain help with the treatment of dive-related incidents 24 hours a day by calling the Divers Alert Network at Duke University (919-684-9111).

6. If it is available, administer lidocaine intravenously per protocol as an adjunct to recompression therapy.

Nitrogen Narcosis

Nitrogen narcosis is the increasing development of anesthesia or intoxication as the partial pressure of nitrogen in inspired compressed air increases at depth.

Decompression Sickness

DCS is caused by the formation of bubbles of inert gas (e.g., nitrogen) within the intravascular and extravascular spaces after a reduction in ambient pressure. Symptoms of DCS are often categorized into type I and type II, with type I referring to the mild forms of DCS (cutaneous, lymphatic, and musculoskeletal) and type II including the neurologic and other serious forms. Some investigators have advocated use of the term type III decompression sickness to refer to combined arterial gas embolism and DCS with neurologic symptoms.

Signs and Symptoms

1. Symptoms developing in the first hour after surfacing from a dive, with some patients noticing symptoms within 6 hours after diving; rarely, symptoms not noted until 24 to 48 hours after diving

2. Musculoskeletal DCS or “limb bends”: periarticular joint pain most common symptom

3. Neurologic DCS: back pain, girdling abdominal pain, extremity heaviness or weakness, paresthesias of extremities, anal sphincter weakness or fecal incontinence, loss of bulbocavernosus reflex, bladder distention and urinary retention, paralysis, hyperesthesia or hypoesthesia, paresis, scotomata, headache, dysphagia, confusion, visual field deficit, spotty motor or sensory deficits, disorientation, mental dullness

4. Fatigue

5. Cutaneous: pruritus, mottling, local or generalized hyperemia, marbled skin (cutis marmorata)

6. “Chokes”: dyspnea, substernal pain made worse on deep inhalation, nonproductive cough, cyanosis, tachypnea, tachycardia

7. Vasomotor DCS: weakness, sweating, unconsciousness, hypotension, tachycardia, pallor, mottling, decreased urine output

Treatment

Although experimental proof of their efficacy is lacking, high-dose parenteral corticosteroids have been widely recommended as an adjunct to recompression treatment. They are used much less often than in the past. If you elect to use these, administer hydrocortisone hemisuccinate, 1 g, or methylprednisolone sodium succinate, 125 mg, followed by dexamethasone, 4 to 6 mg q6h for 72 hours.