Rotator Cuff Repair and Rehabilitation
Lisa Maxey and Mark Ghilarducci
Etiology
Rotator cuff disorders are generally thought to have a multifactorial cause, including trauma, glenohumeral (GH) instability, scapulothoracic dysfunction, congenital abnormalities, and degenerative changes of the rotator cuff. Intrinsic factors of primary tendon degeneration and extrinsic mechanical factors have been described extensively and are felt to be the primary contributors of rotator cuff pathology. Intrinsic tendon degeneration has been described. In 1931, Codman and Akerson1 suggested that degenerative changes in the rotator cuff lead to tears. Microvascular studies of the vascular pattern of the rotator cuff have demonstrated a hypovascular zone in the supraspinatus adjacent to the supraspinatus insertion into the humerus.2–4 Relative ischemia in this hypovascular zone is believed to lead with aging to decreased tendon cellularity and the eventual disruption of the rotator cuff attachment to bone.
Compression of the rotator cuff between the acromion and the humeral head may subject the cuff to wear as the supraspinatus passes under the coracoacromial arch. Neer5,6 postulated that 95% of rotator cuff tears are caused by impingement of the rotator cuff under the acromion. Neer5 classified three stages of impingement as a continuum that eventually led to cuff tears. Stage I is characterized by subacromial edema and hemorrhage of the rotator cuff and usually occurs in patients younger than 25 years old. Stage II includes fibrosis and tendinosis of the rotator cuff and occurs more commonly in patients 25 to 40 years old. Stage III is a continued progression characterized by partial or complete tendon tears and bone changes. Typically, this involves patients older than 40 years old.5 Bigliani, Morrison, and April7 described three types of acromion shapes: (1) type I is flat, (2) type II is curved, and (3) type III is hooked. An increased incidence of rotator cuff tears is associated with a curved (type II) or a hooked (type III) acromion. Other sources of extrinsic impingement postulated include acromioclavicular (AC) osteophytes, the coracoid process, and the posterosuperior aspect of the glenoid.8
A rotator cuff tear may occur spontaneously after a sudden movement or a traumatic event.9 Ruptures of the rotator cuff have been estimated to occur in up to 80% of persons older than 60 years of age with GH dislocations.10 Cuff tears usually occur late in the shoulder deterioration process (after secondary impingement) and in older adults.
In athletes who participate in repetitive overhead activities (e.g., throwers, swimmers, tennis players), small rotator cuff tears may appear late in the deterioration process from secondary impingement. Secondary impingement is caused by instability of the GH joint or by functional scapulothoracic instability.11 The primary underlying GH instability may progress along a continuum from anterior subluxation to impingement to rotator cuff tearing. Treatment must be directed to the primary instability problem.12
The throwing athlete also may have secondary impingement caused by functional scapular instability. Fatigue of the scapular stabilizers from repetitive throwing leads to abnormal positioning of the scapula. As a result, humeral and scapular elevation lose synchronization and the acromion is not elevated enough to allow free rotator cuff movement.11 The rotator cuff abuts the acromion, causing microtrauma and impingement. A tear may gradually or spontaneously occur.
In summary, rotator cuff disease has a multifactorial cause. Vascular factors, impingement, degenerative processes, and developmental factors all contribute to the overall evolution and progression of rotator cuff disorders (Box 5-1).
Clinical Evaluation
History
The majority of patients with rotator cuff dysfunction have pain. They may complain of fatigue, functional catching, stiffness, weakness, and symptoms of instability. An acute or macrotraumatic presentation is important to distinguish from an overuse or microtrauma presentation. Most patients report a gradual onset of pain with no history of trauma. A gradual onset of weakness is usually associated with a chronic tear and an acute onset of weakness after years of shoulder pain is suggestive of an acute on chronic tear.
Pain is typically localized in the upper arm in the region of the deltoid tuberosity and anterior lateral acromion. Pain is usually worse at night. Overhead activities often induce the patient’s symptoms. Typical findings are a loss of endurance during activities, catching, crepitus, weakness, and stiffness.
Physical Examination
A thorough examination of the shoulder should include evaluation of the cervical spine and an upper extremity neurologic examination. The opposite shoulder must also be examined for comparison. Inspection, palpation for tenderness, and range of motion (ROM) tests should be completed. Palpation should include the AC joint, sternoclavicular (SC) joint, subacromial space, biceps tendon, trapezius muscle, and cervical spine. Impingement sign tests as described by Hawkins, Misamore, and Hobeika13 (i.e., forward flexion to 90° and internal rotation) and by Neer and Welch14 (forward elevation and internal rotation) are performed to elicit pain. If these tests produce pain, then they are considered positive signs of impingement and suggest rotator cuff dysfunction. The rotator cuff muscles are tested for strength. Subscapularis muscle strength tests include the lift-off test and the belly-press test. The lift-off test places the arm behind the back and up the spine. The patient is then asked to lift the hand off the back against restriction. The belly-press test places the arms onto the elbows bent to 90°, and the elbows are then lifted anteriorly against resistance. Examination for instability is performed. Apprehension sign, a positive-relocation test, or inferior sulcus sign are all indicative of instability. Stability testing should be performed in different positions (i.e., seated, supine) to eliminate instability as the cause of secondary impingement. Evidence of rotator cuff pathology includes painful impingement signs or weakness and a painful arc of motion.5
Diagnostic Testing
Diagnostic testing includes injections, radiographs, arthrogram, or magnetic resonance imaging (MRI) or ultrasonography. An impingement test (subacromial space injection with at least 10 mL of 1% lidocaine) is invaluable in evaluating the origin of shoulder pain. Physical examination several minutes after the injection, including for ROM, impingement signs, strength, and instability should be completed. Resolution of the shoulder symptoms without instability indicates primary rotator cuff pathology or impingement. Relief of pain with evidence of instability indicates possible primary instability with secondary rotator cuff changes because of altered shoulder mechanics.
Convention radiography is an important tool in the evaluation of rotator cuff tear pathology and is used to rule out arthritis and fractures, assess the morphology of the acromion, and look for calcifications about the shoulder. Arthrography is no longer the gold standard for identification of rotator cuff tears. Although reliable for the diagnosis of complete rotator cuff tears, it is less reliable for the evaluation of partial-thickness rotator cuff tears. MRI has evolved to provide an excellent noninvasive tool in the diagnosis of rotator cuff pathology and the shoulder labrum and is the modality of choice for the evaluation of cuff tear pathology, followed by ultrasonography. MRI provides information not available by other diagnostic testing, including muscle atrophy, amount of rotator cuff retraction in full-thickness tears, bursal swelling, the status of the AC joint, and the shoulder articular cartilage. The combination of intraarticular contrast (gadolinium) and MRI (magnetic resonance arthrography) has been developed to better delineate abnormalities of the rotator cuff and labrum including partial surface rotator cuff tears.15,16 Ultrasonography of the shoulder has increased in popularity, and as technical advances continue to improve, it has become an increasingly useful tool in the diagnosis of cuff pathology. Ultrasonography has several advantages. These include a relatively low cost, the lack of the contraindications as with MRI, and its use in the examination of the patient both statically and dynamically. Unfortunately, the accuracy of ultrasonography is operator dependent and typically requires a long learning curve.
Treatment
Symptoms of rotator cuff dysfunction are usually treated initially in a nonoperative fashion (tendonitis, partial- or full-thickness rotator cuff tears). Nonsteroidal antiinflammatory drugs (NSAIDs), heat, ice, relative rest, cortisone injection, and rehabilitation programs are used in the treatment. The initial goal of treatment is restoration of normal ROM. This is followed by a rotator cuff strengthening program. Stretch cords for resistance are initiated and are followed by free weights as tolerated. To avoid aggravation of the rotator cuff, all strength training initially should be below shoulder level. External rotation strengthening with the arm at the side may minimize subacromial pressure and pain while increasing the cuff’s ability to act as a humeral head depressor.
Nonoperative treatment programs usually continue for 3 to 6 months. Success varies from 50% to 90%.17–21 Approximately 50% of patients with complete symptomatic rotator cuff tears have satisfactory results with nonoperative measures, but these results may deteriorate with time.22 This wide range of outcomes is likely the result of lack of uniformity in classification, indications, and treatment. Some individuals have rotator cuff tears with no pain and normal function, whereas others may have debilitating pain. This demonstrates a need for better understanding of the factors that lead to symptoms.
Indications for Surgery
Indications for rotator cuff surgery include failure of 3 to 6 months of conservative care or an acute full-thickness tear in an active patient younger than 50 years. Failure of treatment can be determined before an entire rehabilitation course is completed. Indications for earlier surgical treatment can include return to full strength with persistent symptoms, failure to tolerate therapy because of pain, or plateau of initial improvement with persistent symptoms. Early surgical intervention is also indicated for patients sustaining acute trauma with full-thickness tears associated with significant rotator cuff weakness and posterior cuff involvement, particularly in young patients with higher functional demands. In addition, patients with acute tears or extension of chronic cuff tears may benefit from early surgery.23
In general, the duration of nonoperative treatment must be individualized based on pathology involved, the patient’s response to treatment, and individual functional demands and expectations.
Surgical Goals
The primary goal of rotator cuff surgery is decreased pain, including rest pain, night pain, and pain with activities of daily living (ADLs). Arrest of the progression of rotator cuff pathology and improved shoulder function are additional surgical goals.
Surgical Procedures
Tendonitis and Partial Rotator Cuff Tear
Surgical management for impingement syndrome generally involves open anterior acromioplasty as described by Neer63 or arthroscopic subacromial decompression (SAD) with release or partial release of the coracoacromial ligament. The coracoacromial ligament is released from the undersurface of the anterior and lateral acromion. An acromioplasty is performed using a burr to achieve a flat acromion. If osteophytes are present on the inferior surface of the AC joint, they are removed from the distal clavicle. Partial thickness cuff tears may be bursal or articular sided. Partial thickness cuff tears may be treated with débridement alone, débridement and acromioplasty, or rotator cuff repair. A subacromial decompression/acromioplasty is generally indicated if the partial tear is bursal sided or there are signs of mechanical impingement. Partial rotator cuff tears greater than 50% of the width of the tendon generally are treated with arthroscopic repair insitu or transtendineus repair, or may be treated with takedown and repair of the rotator cuff either miniopen or arthroscopic.24 Repair of partial-thickness tears improves results compared with débridement alone in this patient group. The overhead throwing athletes with rotator cuff disease have different requirements. Acromioplasty is rarely necessary in the throwing athlete.
Results of surgical treatment of low-grade (<50%) partial-thickness rotator cuff tears after débridement with and without acromioplasty have reported 75% to 88% satisfactory outcome on short-term follow-up.18,25–27 Repair of a high-grade (>50%) partial-thickness tear has had similar outcome to débridement of a low-grade partial-thickness tear or repair of a small full-thickness cuff tear.28
Full-Thickness Tears
The mainstay of treatment for full-thickness tears is surgical repair. The type, pattern, and size of the tear, as well as the surgeon’s preference, dictate whether the repair is a full-arthroscopic, miniopen, or a completely open procedure.
Small- or moderate-sized (3 cm or less) partial- or full-thickness supraspinatus or infraspinatus tears may be repaired fully with an arthroscopic or miniopen technique. Large width (3 to 5 cm) tears may also be repaired miniopen or arthroscopic if the cuff is mobile enough to allow anatomic repair.
Large, immobile cuff tears involving the subscapularis or teres minor, as well as tears of the musculotendinosis junction, may require an open approach. Massive chronic atrophic cuff tears should be considered for arthroscopic débridement for pain control.
Surgical Technique
All operative procedures discussed in recent literature for primary repair of rotator cuff tears include use of an anterioinferior acromioplasty to decompress the subacromial space. The author presently performs GH arthroscopy and subacromial bursoscopy on all patients undergoing surgery for cuff pathology.
Shoulder arthroscopy and decompression is performed as previously described. After acromioplasty, the bursal surface of the rotator cuff is evaluated. With shoulder rotation, the cuff can be completely visualized. Mobile tears may be treated with a miniopen technique or with arthroscopic repair.
Arthroscopic Rotator Cuff Repair
In the past decade as arthroscopic repair of rotator cuff tears has become more widespread, there have been significant advances in arthroscopic surgical techniques and instrumentation. Arthroscopic repair involves the same general steps as miniopen or open rotator cuff repair. Bony landmarks are outlined and marked with a sterile pen. Multiple arthroscopic portals are made. Posterior, anterior, and lateral portals are made for all arthroscopic repairs. Additional portals are made depending on the rotator cuff tear configuration and include posterior lateral, anterior lateral, and lateral acromial portals. These allow access to various rotator cuff configurations and for suture anchor placement. The GH joint is completely and systematically evaluated followed by subacromial bursoscopy. The subacromial bursa is excised, and the anterior acromion is flattened with a burr. If symptomatic, then the AC joint is excised arthroscopically. The rotator cuff is visualized. The quality and the integrity of the rotator cuff are evaluated. The mobility of the cuff is evaluated. Adhesions on the bursal and articular sides of the tear are released. The goal is a mobile cuff which can be repaired to the normal footprint on the greater tuberosity with minimal tension. A three dimensional understanding of the cuff tear configuration is developed. Margin convergence repair principles are used for U-shaped tears or L- or reverse L-shaped tears. Sutures are placed in a side-to-side fashion through the anterior and posterior leafs of the tear. This convergences the edges of the tear and minimizes the tension on the tendon to bone repair. The result may be an arthroscopic repair of an otherwise irreparable tear. The greater tuberosity of the humerus is lightly decorticated with a burr. The tendon is then repaired to bone with suture anchors. The number of suture anchors used is predicated on the size of the tear and its configuration. In general, one suture anchor is used for each one centimeter of cuff tear.
Miniopen Rotator Cuff Repair
In the miniopen procedure, the lateral subacromial portal incision is extended either longitudinally or transversely to expose the deltoid fascia. The deltoid fascia is then split in line with its fibers directly over the tear. The anterior deltoid insertion of the anterior acromion is preserved. The deltoid fibers should not be split more than 4 cm lateral to the lateral acromion to avoid axillary nerve injury (Fig. 5-1). Rotation of the arm provides access to the tear. Digital palpation can be used to assess the adequacy of the acromioplasty. A bony trough is prepared in the greater tuberosity of the humerus. The rotator cuff may then be repaired through a bony bridge or with suture anchors. The permanent sutures are tied, pulling the rotator cuff down into its trough on the humerus.
Open Rotator Cuff Repair
Tears that are fixed, retracted, but reparable, may be repaired open using principles developed by Neer.5,6 An oblique incision in Lagers line from the anterior edge of the acromion to a point about 2 cm lateral to the coracoid process is made. The anterior deltoid is released from the anterior aspect of the acromion and splitting the deltoid no more than 4 cm lateral to the acromion (Fig. 5-2). The deltoid origin over the acromion is elevated subperiosteally. The coracoacromial ligament is released.
The anterior acromion is osteotomized. The acromion anterior to the anterior aspect of the clavicle is removed, and the undersurface of the acromion is flattened from anterior to posterior. The AC joint may be removed if arthritic and symptomatic. The distal clavicle is excised parallel to the AC joint so that no contact occurs with adduction of the arm.
The cuff tear is visualized and mobilized. A bony trough 0.5 cm in width parallel to the junction of the humeral articular cartilage and greater tuberosity of the humerus is made. The rotator cuff tear is repaired to bone with suture anchors or through a bony bridge in the greater tuberosity using permanent sutures (Fig. 5-3). The goal is to repair the cuff with minimal tension with the arm at the side.
Watertight repairs are not necessary for good functional outcome. Excellent and good results have been shown in patients with residual cuff holes.29,30 The anterior deltoid is repaired back to the acromion by preserved periosteum or through drill holes with permanent sutures. Routine skin closure is performed.
Postoperative management of cuff repairs must be individualized to incorporate tear size, tissue quality, difficulty of repair, and patient goals. Passive motion is initiated immediately. In general, supine active-assisted motion is started on the first postoperative day. Waist-level use of the hand can usually be started after surgery. Active ROM and isotonic strengthening are started 6 to 8 weeks after surgery. Progress of strengthening is individualized with full rehabilitation taking from 6 to 12 months. Function can continue to improve for 1 year after surgery.
Management of Massive Tendon Defects
The management of massive irreparable tendon defects remains controversial. Options include SAD and débridement of nonviable cuff tissue without attempt at repair, use of autogenous or allograft tendon grafts, and use of active tendon transfers. Operations that require tendon transfer to nonanatomic sites to cover rotator cuff defects are likely to alter mechanics of the shoulder unfavorably.31 Débridement may be pursued (either open or arthroscopic).
Results of Treatment of Full-Thickness Rotator Cuff Tears
Satisfactory results after rotator cuff repair for pain relief occur 85% to 95% of the time5,13,32,33 and appear to correlate with the adequacy of the acromioplasty and SAD. Functional outcomes correlate with integrity of the cuff repair, preoperative size of the cuff tear, and quality of the tendon issue. Poor outcomes are also associated with deltoid detachment or denervation.34
Arthroscopic-assisted miniopen rotator cuff repair provides favorable clinical results. Results comparable with open cuff repair have been reported for small- and moderate-sized rotator cuff repair (less than 3 cm.).35–37 These studies have shown the most important factor affecting outcome was cuff tear size. Tears of small or moderate size had better results. Blevins and associates,38 in a retrospective study, have shown 83% good or excellent results regardless of cuff size. Most studies have shown more rapid return to full activities with miniopen repair.
Fully arthroscopic repair studies have shown outcomes approaching the results of open rotator cuff repair or miniopen rotator cuff repair.39–47
A surgical technique that initially includes arthroscopy has the advantage of providing identification and treatment of intraarticular pathology (articular cartilage, labrum, biceps tendon). Additional advantages of arthroscopic rotator cuff repair include decreased soft tissue dissection, improved cosmesis, preservation of the deltoid attachment, decreased postoperative pain, and earlier return of normal ROM.
Unfortunately, rehabilitation cannot be accelerated for arthroscopic or miniopen cuff repairs because the limiting factor, tendon-to-bone healing, is not changed by the surgical technique.
No ideal surgical technique exists. Each surgeon must individualize treatment based on the type of lesion present, as well as the expertise of the physician. As has been noted over the past decade with the widespread use of shoulder arthroscopy and improved surgical technique and instrumentation, arthroscopic rotator cuff surgery will continue to increase in frequency and open or miniopen cuff repairs will continue to decrease in frequency. All these methods have a role in the treatment of rotator cuff tears.
Therapy Guidelines for Rehabilitation
The general guidelines that follow are for the rehabilitation of a type 2 rotator cuff tear (a medium-to-large rotator cuff tear that is larger than 1 cm and smaller than 5 cm). We have also included a table of guidelines to follow for large tears. The protocol is designed for active patients (i.e., recreational athletes, laborers). Older, more sedentary individuals progress through the stages more slowly. These patients are not appropriate candidates for the more aggressive exercises. Recent studies suggest that longer periods of immobilization and a more conservative approach to restoring ROM early on leads to more successful outcomes in terms of fewer repeat tears following surgery or insufficient healing of the rotator cuff. Even if the tear is not completely healed, the patient can be satisfied with the results. However, they are happier if the cuff is healed. Therefore, the goal is for a healed rotator cuff repair. Too many ROM exercises or too much stress on the repaired tissues early on may create an increase in scar tissue. This tissue has a poorer quality of intracellular tissue. Studies have also shown that after 1 year there is no difference in the ROM of patients in different groups following surgery.48 Groups that received early ROM treatment versus groups that received delayed ROM treatments had the same ROM at 1 year. The group that delayed ROM treatments actually had a higher rate of healing versus the group who received passive range of motion (PROM) early in the rehabilitation process.48
Many factors contribute to the healing rates of these repairs: retraction of the tissue, age, early repair versus late repair, surgical technique, patient selection, and postoperative rehabilitation. Poorer outcomes have been noted with patients over 65 years of age, manual laborers, those with poor bone stock, tears greater than 5 cm, workers’ compensation cases, or active litigation clients. Better outcomes have been noted with younger patients, smaller tears, and early surgical repair. In light of the recent discussion of early versus delayed ROM following a rotator cuff repair, we are presenting a more conservative approach to rehabilitating these patients in this third edition. Patients who have early signs of stiffness should be treated with a more liberal approach to restoring ROM. Benefits to early ROM treatments are minimal; however, the benefits to maintaining a safe environment for optimal healing are far more beneficial. The goal is to avoid overstressing the healing tissues and preventing shoulder stiffness.
These guidelines are designed to help guide therapists and provide treatment ideas. The scope of this chapter does not include instructions on treatment methods or applications. All modalities, mobilizations, and exercises suggested in this chapter are recommended only for therapists who have been trained in these methods and can appropriately apply them. The therapist must choose the treatments that are beneficial and safe for each patient while following the restrictions outlined by the operating surgeon.
Phase I
TIME: 1 to 4 weeks after surgery
GOALS: Comfort, maintain integrity of repair, increasing ROM as tolerated without progressing to full range, decreased pain and inflammation, minimal cervical spine stiffness, protection of the surgical site, maintenance of full elbow and wrist ROM (Table 5-1)
TABLE 5-1
Rotator Cuff Repair for Moderate-Sized Tears
< ?comst?>
Rehabilitation Phase | Criteria to Progress to this Phase | Anticipated Impairments and Functional Limitations | Intervention | Goal | Rationale |
Phase I Postoperative 1-4 wk
|
|
|
< ?comen?>< ?comst1?>
< ?comst1?>
< ?comen1?>
Refer to Box 5-2 for a shoulder evaluation following a rotator cuff repair. The therapist must maintain the protection of the patient and the surgical repair while obtaining an evaluation; therefore, some tests will need to be deferred until later in the treatment process.
To reduce pain and swelling, use cryotherapy. Electrical stimulation may also be used for pain reduction. Instruct the patient in posturing for comfort. Encourage the patient to experiment with different positions. Usually a loose packed GH position (shoulder in some flexion, abduction, and internal rotation) with the arm supported by pillows while supine or sitting is more comfortable. Usually patients cannot sleep much after surgery in the supine position. Therefore suggest sleeping semireclined in a recliner chair with the upper extremity supported in the loose packed position. The patient may also try the supine position in bed, with the arm supported by pillows in a loose packed position.
Gentle mobilizations using grades I and II oscillations, and distractions may help reduce pain, muscle guarding, and spasms. These mobilizations also help maintain nutrient exchange and therefore prevent the painful and degenerating effects that long periods of immobilization produce (i.e., a swollen and painful joint).18 Occasionally, some people produce increased levels of scar tissue and tighten up quickly. For these cases, passive exercises provide nourishment to the articular cartilage and assist in collagen tissue synthesis and organization.49–51 The organization of collagen may then follow stress patterns, and adverse collagen tissue formation may be minimized. Limited periods of PROM and pendulum exercises are initiated during this initial stage. For large to massive tears, consider withholding PROM exercises until 4 weeks postsurgery. Recently, it has been suggested that early ROM or excessive ROM treatment of the GH joint may delay tissues from healing. Therefore, when conducting PROM treatments, be careful to protect healing tissues from too much stress from ROM exercises. PROM exercises are done in protected planes. PROM exercises for shoulder flexion are initiated in the scapular plane with the elbow flexed 90°, and external rotation is done with the palm facing the patient and beginning at 45° of abduction.52 Performing PROM exercises in the scapular plane is beneficial because of decreased tension on the capsuloligament-tendon complex.52 “Rotation exercises should be initiated at 45° of abduction to minimize tension across the repair.”52
Remember to avoid horizontal adduction, extension, and internal rotation during this phase. Also advise patients to avoid leaning on the elbow, sleeping on the affected side, sudden movements, pushing/pulling, lifting, and carrying for 12 weeks.53
A general guideline to use in judging the force being applied is slight discomfort with a slight increase in motion after several repetitions. Remain sensitive and aware of the feedback the patient’s body is exhibiting during ROM or mobilization techniques. The patient’s response will dictate the amount of force applied or the plane of movement chosen. If muscle guarding continues to increase after several repetitions, the force being applied should be reduced or the plane of movement chosen needs to be slightly altered or decreased (or both need to be done) to avoid pinching sensations or increased pain. The therapist usually can find a groove (i.e., line of movement that can be progressed more easily) or line of motion with less muscle guarding. Therefore, constantly assess treatment application while treating the patient with manual PROM. Vary the treatment application as the patient’s feedback dictates (i.e., exact plane of movement, force, and repetitions). An increase in ROM will often accompany a decrease in pain if executed with a sensitive hand. However, general treatment soreness may be expected. Treatment soreness is usually more pronounced when progressing the patient from PROM to active range of motion (AROM) and then again when progressing to resisted ROM exercises. Remember during this stage we do not want full ROM. The superseding goal is to provide an environment where the tissues can heal while preventing stiffness.
Patients usually exhibit protective muscle guarding from the necessary insult of the surgery and the preceding shoulder pathology. Muscle guarding is present in the cervical region and the shoulder musculature. Therefore patients perform cervical AROM exercises and stretches. Appropriate cervical spine mobilization techniques may be valuable for decreasing cervical joint stiffness and muscle guarding, allowing more unrestricted movement of the shoulder complex.
Phase II
TIME: 5 to 8 weeks after surgery
GOALS: Protection of surgical site, improvement of ROM, increase in active strength, decrease in pain and inflammation, maintenance of elbow and wrist ROM, and minimizing of cervical stiffness (Table 5-2)
TABLE 5-2
Rotator Cuff Repair for Moderate-Sized Tears
< ?comst?>
Rehabilitation Phase | Criteria to Progress to this Phase | Anticipated Impairments and Functional Limitations | Intervention | Goal | Rationale |
Phase II Postoperative 5-8 wk
|
|
Buy Membership for Orthopaedics Category to continue reading. Learn more here
|