Primary Mandibular Deficiency Growth Patterns with or without Maxillary Arch Constriction

Published on 13/06/2015 by admin

Filed under Surgery

Last modified 22/04/2025

Print this page

rate 1 star rate 2 star rate 3 star rate 4 star rate 5 star
Your rating: none, Average: 0 (0 votes)

This article have been viewed 2666 times

19

Primary Mandibular Deficiency Growth Patterns with or without Maxillary Arch Constriction

The surgical correction of the underdeveloped mandible was first reported in the American literature by Vilray P. Blair in the Journal of the American Medical Association in July 1909.7 Dr. Blair presented two cases of retrognathia in which a horizontal ramus osteotomy was performed on each side of the mandible. The operative technique was carried out through a preauricular stab incision; an aneurysm needle was introduced posteriorly to the neck of the condyle and directed medially around to the ascending ramus, where it then exited through the cheek in front of the anterior border of the ramus. A Gigli saw was then put in place and blindly used to complete an osteotomy. The same procedure was completed on the opposite side of the jaw, and then the mandible was advanced into the desired occlusion. Intermaxillary fixation was applied until a callus formed. As a result of the limited bone contact across the osteotomy sites, a strong relapse tendency required persistent counterforces with continued intermaxillary elastics. Extrusion of the incisors with periodontal sequelae and recurrent malocclusion was a frequent observation.

Mandibular deficiency is the most prevalent form of dentofacial deformity.2,30,32,39,43 It is more common among Caucasians than Africans or Asians, which suggests that a hereditary component is a predominant part of the etiology. During the mid 1970s, the public health service of the U.S. Department of Health, Education, and Welfare conducted an assessment of the occlusions of children who were 6 to 11 years old and 12 to 17 years old. These collected data indicated that approximately 20% of the sample populations had an Angle Class II malocclusion. Unfortunately, no other means of evaluation were performed to separate those children with a Class II jaw disharmony from those with a more straightforward dental Class II malocclusion. According to a National Health and Nutrition Examination Survey, severe mandibular deficiency (defined as >7 mm of positive overjet) in the U.S. Caucasian population has a prevalence of approximately 10%.43 This pattern of Class II excess overjet is less frequent among African-American and Mexican-American groups. It can be assumed that more than 7 mm of overjet indicates a jaw discrepancy of such magnitude that orthognathic surgery would be the preferred way to manage the problem. The majority of these patients will have dentofacial deformities that fall into the categories of a long face growth pattern, maxillomandibular deficiency, or primary mandibular deficiency with or without arch-width discrepancy.

This chapter will focus on the patient with a mandibular deficiency who presents without a significant component of either vertical or horizontal upper jaw deformity. These individuals frequently will have a degree of maxillary arch-width constriction that also requires correction. Although this categorization of primary mandibular deficiency may appear arbitrary, it focuses on a distinct subgroup of patients who routinely present for clinical assessment and treatment.

Functional Aspects

• Articulation errors during speech and swallowing difficulties that result from the Class II malocclusion are expected (see Chapter 8).

• Difficulties with chewing and with lateral and protrusive mandibular movements as a result of the Class II malocclusion are also expected.

• Upper airway obstruction during sleep often exists as a result of the retropositioned tongue. Obstructive sleep apnea should be ruled out (see Chapter 26).

• Detrimental long-term effects on the dentition and the periodontium as a result of the crowding of the mandibular teeth in limited alveolar housing or from previous camouflage orthodontic treatments are often seen (see Chapters 5 and 6).

• A higher incidence of temporomandibular disorders that result from mandibular deficiency Class II malocclusion have been reported (see Chapters 9 and 36).

Dental Findings

• There are varied degrees of increased overjet with a Class II molar and canine relationship.

• The crowding of the teeth within the lower arch (i.e., the smaller the mandible, the greater the crowding) needs to be evaluated, and so does lower incisor flaring. Mandibular dental compensations often mask the true skeletal discrepancy.

• There are varied degrees of dental compensation in the upper jaw with either procumbency (i.e., “buck teeth”) or maxillary incisors that are retroclined.

• The anterior deep bite (i.e., excessive overbite) may involve impingement of the anterior maxillary palatal tissues by the mandibular incisors.

• There is an excessive curve of Spee in the mandible (i.e., the deeper the anterior bite, the greater the excess curve).

• A reduced or negative curve of Spee is found in the maxillary arch.

• Dental crowding may also be seen in the maxilla (i.e., the more constricted the maxilla, the greater the crowding).

• Excessive wear facets, cusp fractures, and significant restorations in the posterior dentition are frequent findings in adults.

• Accelerated loss of the periodontium associated with the maxillary incisors (due to traumatic occlusion or previous orthodontics); the maxillary molars (due to arch constriction or previous orthodontics); and the mandibular incisors (due to supraeruption and anterior flaring from previous orthodontics) may be seen in adults.

Treatment Indications and Objectives

The individual’s immediate aesthetic concerns often center on 1) unfavorable lower-lip contours (e.g., a curled and everted lower lip with an accentuated labiomental fold) 2) a lack of prominence of the chin (e.g., a weak profile) and 3) an unattractive neck (e.g., a soft-tissue double-chin tendency).27,38,39,42,48,47,50,59 Functional issues are also to be expected. For example, when the anterior bite is “deep,” it will cause irritation of the gingival tissues of the palate just behind the maxillary incisors. Partial obstruction of the upper airway as a result of a retropositioned tongue is also common (i.e., heavy snoring, restless sleep, daytime fatigue, and documented or undiagnosed sleep apnea; see Chapter 26).11,62 Complaints of an uncomfortable “dual” bite, chewing difficulties, masticatory muscle discomfort, and audible speech articulation errors are frequent. The individual with a Class II deep bite may have a greater tendency toward clicking (i.e., anterior disc displacement) in each temporomandibular joint.25,51 Interestingly, there is little scientific evidence to support the notion that the signs of clicking will into more significant temporomandibular joint pathology (see Chapter 9).

When the mandible is deficient, with significant overjet, and when the anterior facial height is short, the lower lip tends to “get caught” behind the maxillary incisors. During growth, if the lower lip stays behind the upper teeth, then the upper teeth will adapt by tipping forward, thus creating a Class II, Division 1 malocclusion with varied degrees of maxillary incisor spacing. When the mandibular deficiency and the protrusive lower incisors are combined with procumbent maxillary incisors, the individual tends to be concerned with both the lack of chin prominence and the protruding maxillary anterior teeth (i.e., a toothy smile). Alternatively, if the lower lip remains in front of the maxillary incisors, there is an adaptive tendency for the maxillary incisors to tip lingually (i.e. Class II, Division 2).

The Role of Growth Modification in the Preadolescent Child

When the preadolescent child with mandibular deficiency is identified, a growth modification approach to treatment is frequently considered by the evaluating orthodontist.13,33,32 Cozza and colleagues completed a systematic review of the literature to assess the efficacy of functional appliance use for enhancing mandibular growth in subjects with Class II malocclusions.14 With the use of the Medline database, the survey covered the period from January 1966 to January 2005. A review of 704 articles confirmed only 22 that qualified for the final analysis. Of these 22 articles, only four randomized clinical trials were retrieved and found to be worthy of inclusion. Interestingly, none of these four trials reported a clinically significant change in mandibular length to be induced by functional appliances in the patients with Class II deformities. According to Proffit, the only concrete indications for the treatment of the preadolescent mixed dentition child with a mandibular deficiency who presents without an anterior open bite are the following44:

Despite the review by Cozza and colleagues and Proffit’s stated limited indications for early mixed dentition treatment in the child with a mandibular deficiency, a growth modification approach is often carried out by the evaluating orthodontist. The goals are frequently stated to be the restraining of forward maxillary growth while encouraging the mandible to grow downward and forward. In accordance with this rationale, mixed dentition growth modification treatment frequently incorporates the following steps:

Orthodontic Camouflage Approach

When the orthodontist proceeds with growth modification maneuvers and a less than favorable biologic response is realized, it is apparently tempting to proceed with a camouflage (dental compensation) approach (Figs. 19-1 through 19-5).34,35,37,36,42 This would be carried out to “neutralize” the occlusion rather than to position the teeth solidly into the alveolar process in combination with orthognathic surgery. Current evidence-based medical standards recommend that the treating orthodontist have a “timeout” (i.e., a sit-down discussion and reassessment with the parents and the patient) that includes a review of the records (e.g., photos, models, radiographs) and a discussion of the biologic effects on the airway, the facial aesthetics, the occlusal stability, and the periodontium that may result from the various treatment options. A referral to an experienced orthognathic surgeon for the discussion of possible future surgical aspects is highly recommended.

An orthodontic camouflage approach for the individual with mandibular deficiency may take the form of maxillary premolar extractions so that the upper incisors can be retracted. This is generally combined with the flaring of the mandibular incisors. When this is carried out in the individual with a mandibular deficiency, the short-term objective of a neutralized occlusion may be achieved, but the following are potential sequelae: 1) unfavorable long-term facial aging 2) the risk for obstructive sleep apnea later in life 3) long-term periodontal deterioration 4) lower anterior crowding with recurrent malocclusion and 5) an inconsistent occlusion (i.e., a dual bite) (see Figs. 19-1 through 19-5). The orthodontist may feel favorable toward a camouflage approach on the basis of the belief that the patient can always have a “chin implant” to improve his or her facial aesthetics. Unfortunately, this approach will rarely achieve a pleasing aesthetic outcome. A chin implant tends to result in 1) an accentuated labiomental fold 2) an overly prominent pogonion and 3) a residual obtuse neck–jaw angle (Fig. 19-4). Furthermore, it does nothing for any baseline masticatory and airway dysfunction (see Chapter 37).

Definitive Reconstruction

Timing of Orthognathic Surgery

Stahl and colleagues carried out a longitudinal study to compare the biologic jaw growth changes of untreated subjects with Class II, Division 1 malocclusions as compared with those subjects with a normal Class I occlusion.55 Each subject was followed from the prepubertal stage through the postpubertal stage of development. The study provides evidence of a similar growth pattern in both Class II and Class I subjects. The absolute amount of mandibular lengthening at the pubertal peak was significantly smaller in the Class II subjects than in those with normal occlusion. A significant deficiency was measured in overall mandibular length (i.e., Condyle-Gonion [Co-Gn]) in the Class II malocclusion subjects at the completion of growth. Interestingly, the authors’ findings show that a Class II dentofacial disharmony does not have a tendency to self-correct with growth.

There are numerous reports in the literature of surgeons carrying out successful orthognathic corrections in patients with mandibular deficiencies who are between the ages of 13 and 15 years. This is before the age at which growth maturity is typically considered to be complete. Interestingly, after the patient with a mandibular deficiency has undergone successful surgical correction, there are no reports late mandibular catch up growth.5,20,23,29,45,52,61

A review of available research data and clinical reports indicated that, when orthognathic surgery was undertaken in the patient with a mandibular deficiency as early as the completion of the adolescent growth spurt, further significant forward growth of the mandible almost never occurred.31,55 Further horizontal growth of the maxilla at this age is also not expected. In theory, limited vertical growth of both the mandible and maxilla may continue; however, if it does, it tends to be proportionate. Proceeding with definitive orthognathic surgery in the patient with a mandibular deficiency as early as the age of 13 or 14 years—but only after the eruption and orthodontic alignment of the teeth—is generally safe from a growth perspective.

Periodontal Considerations before Orthodontic Treatment

The position of the mandibular incisors in the alveolar housing has important periodontal consequences (see Chapter 6). In the retrognathic patient, the more the mandibular incisors are orthodontically tipped forward, the greater the chance of labial bone loss and gingival recession. There should be a healthy band of attached gingiva at the beginning of treatment and flaring of the incisors is to be avoided. This is a frequent concern in borderline crowding cases, when the final decision is made to not extract and then further labial tipping of the incisors occurs.

Even after successful gingival grafting, if further incisor flaring is allowed to occur in the presence of deficient labial bone, negative periodontal sequelae are to be expected. For patients with borderline crowding, if the incisor crowns are morphologically large, the judicious use of interproximal reduction may create the needed space to uncrowd the arch and to simultaneously upright the teeth (see Chapter 17).

Orthodontic Preparation for Orthognathic Surgery

The clinicians (i.e., the orthodontist and surgeon), the patient, and the patient’s family should agree about the treatment approach from the outset. It makes no sense for the orthodontist to tell the patient that he or she will first try to avoid surgery. The orthodontic objectives are diametrically opposite, depending on plans for either full correction (i.e., surgery and orthodontics) or camouflage (i.e., orthodontics alone). When a definitive correction is planned for the individual with mandibular deficiency, lower extractions to uncrowd the arch should always be considered. The need for maxillary extractions is not typical. The labial region of the mandibular incisors should be assessed for the need for gingival augmentation before the initiation of treatment.

For the patient with mandibular deficiency who has agreed to a combined treatment approach, an important orthodontic objective is to place the incisors more completely within the alveolus. Incisor placement will determine the eventual horizontal and vertical positioning of the mandible in relation to the maxilla as well as the long-term periodontal health.

The orthodontic leveling of an excessive curve of Spee in the lower arch is accomplished through a combination of the intrusion of the incisors and the extrusion of the mid-arch posterior teeth. With an excessive curve of Spee, premolar extrusion gives the major effect with only a small amount of incisor intrusion to be expected. This is initiated before surgery, but it is most efficiently fully accomplished after the mandible has been surgically repositioned (i.e., forward and down). Alternatively, if extensive intrusion of the anterior teeth is desired to correct the deep curve, this should be done before the mandibular advancement surgery. The correction of a deep curve of Spee via incisor intrusion will result in less clockwise rotation of the mandible in association with surgical advancement. This can be leveraged for enhanced profile aesthetics.

The mechanics of the orthodontic leveling of the curve of Spee will generally result in the lengthening of the arch. The arch expansion primarily occurs at the incisors (i.e., labial flaring) and, to a lesser degree, at the molars. Space is required for both leveling and for aligning. For this reason, pre-alignment extractions to create space will be required if the severity of the curve of Spee is significant and if the available alveolar bone is limited. With a combination of incisor crowding and a deep curve of Spee, extraction therapy is always preferred. If the upper incisors are extremely flared, maxillary premolar extraction to gain space for retraction is also preferred; however, this is rarely the case.

When the deficient mandible is surgically advanced and vertically lengthened to achieve a more ideal overbite and overjet, the relative widening of the mandibular arch in the molar region occurs. Maxillary arch expansion may then be required to correct posterior crossbites. If more than several millimeters is required, then simultaneous surgical arch expansion of the maxilla through segmental Le Fort I osteotomy is the preferred approach (see Chapter 17).

Key aspects of presurgical orthodontics in the individual with mandibular deficiency include the following:

Additional orthodontic considerations for the individual with mandibular deficiency include the following:

• To manage an excessive curve of Spee in the lower arch, a majority of the leveling will be done after jaw surgery via the extrusion of the premolars.

• If the mandibular arch is fully leveled before surgery, then the bicuspid extraction spaces should also be closed before surgery.

• If a degree of lower arch leveling is to be accomplished after jaw surgery, then it is desirable to leave some of the bicuspid extraction space to be closed postoperatively.

• Passive surgical arch wires to be used for stabilization during surgery should be full-dimensional and rectangular.

• With effective plate and screw fixation at each osteotomy site, continuous intermaxillary fixation after surgery is not warranted. Nevertheless, the orthodontic placement of stable hooks throughout the arch wires is useful both at the time of surgery and to accomplish supportive vectored elastics postoperatively.

Immediate Presurgical Planning

The immediate presurgical planning takes into account collaborative clinical efforts between the orthodontist, the surgeon, the patient, and the patient’s family that will have been ongoing since the initial consultation visits. Depending on the condition of the dentition and its supporting structures, the restorative dentist and the periodontist may also have been intricately involved.

Patient-specific medical and dental records are reviewed, which include (but are not limited to) the following: radiographs (e.g. computed tomography scans; panoramic, lateral, and posterior anterior cephalometric and periapical scans); photographs (facial and occlusal); and dental models.

Immediate presurgical records (i.e., within 2 to 6 weeks of surgery) will most importantly include facial measurements taken during the direct visual examination; alginate impressions of the maxilla and mandible; an accurate centric relation bite; and an accurate face-bow registration.

First, broad-stroke decisions are made regarding preferred vector changes for each jaw; only then are the precise millimeter distances and angular changes that are required in each jaw to achieve the desired result determined (see Chapter 12).3,4,17,42 Analytic model planning is carried out on the articulated dental casts with use of the Erickson Model Table. Splints are constructed to assist with the achievement of the precise occlusion and the preferred facial aesthetics in the operating room (see Chapter 13).

During surgical planning for the individual with a primary mandibular deficiency growth pattern, it is known that bilateral sagittal split ramus osteotomies with advancement into a corrected overbite and overjet position will be required (Figs. 19-6 through 19-9).6,21 An important consideration is whether or not surgical repositioning of the upper jaw (i.e., Le Fort I osteotomy) will also be useful to correct arch-width discrepancies; to further adjust the horizontal and vertical position of the maxilla; and to alter the maxillary plane (i.e., pitch orientation) and cant (i.e., roll orientation) (Figs. 19-10 through 19-15).3,4,15,18,41,40,46,49,60 An assessment of the baseline chin morphology is also important. Anticipating the location of the pogonion after jaw repositioning and considering the advantage of an osseous genioplasty to achieve an enhanced horizontal and vertical position of the chin are important (see Chapter 37).

image

image

image

image

Figure 19-12 A 30-year-old woman was referred for surgical assessment only after orthodontic treatment was well under way. When the patient was between 9 and 15 years old, orthodontic growth modification and then a camouflage approach were carried out, but these were not successful. The current orthodontic treatment was flaring the maxillary anteriors even further forward. The patient was referred to this surgeon and agreed to a combined surgical and orthodontic approach. Orthodontic retraction of the maxillary anteriors was then initiated. With sufficient orthodontic (dental) decompensation, surgery was carried out, including bilateral sagittal split osteotomies of the mandible (horizontal advancement and counterclockwise rotation); maxillary Le Fort I osteotomy in segments (arch expansion, horizontal advancement, and minimal counterclockwise rotation); osseous genioplasty (horizontal advancement); and an anterior approach to the soft tissues of the neck (cervical flap elevation, neck defatting, and vertical platysma muscle plication). A, Frontal views in repose before and after treatment. B, Frontal views with smile before and after treatment. C, Oblique facial views before and after treatment. D, Profile views before and after treatment. E, Occlusal views before retreatment, with orthodontics in progress, and after the completion of treatment. Note the early gingival recession of the maxillary canine and bicuspid teeth as a result of orthodontic mechanics. F, Articulated dental casts that indicate analytic model planning. G, Lateral cephalometric radiographs before and after treatment.

image

image

image

image

image

Figure 19-15 A 15-year-old girl was referred for surgical assessment only after orthodontic treatment was well under way. She had severe mandibular deficiency and microgenia, and she had already undergone four bicuspid extractions and a full course of orthodontic treatment. An anterior open bite with procumbent maxillary incisors remained. Evaluation was carried out, including a computed tomography scan, a sleep study, and speech and otolaryngology consultations. The computed tomography scan confirmed congenital dysplasia of the condyle–glenoid fossa complex without condylar resorption. Orthodontics were reinitiated, and surgical reconstruction was chosen. The surgical procedures included a maxillary Le Fort I osteotomy (horizontal advancement, vertical shortening, and counterclockwise rotation); bilateral sagittal split ramus osteotomies (horizontal advancement and counterclockwise rotation); and osseous genioplasty (horizontal advancement). A mild anterior open bite has recurred, but this had been stable for several years. A review of the immediate and late postoperative cephalometric radiographs confirmed a consistent sella–nasion–B-point angle without radiographic evidence of condylar resorption (i.e., no skeletal relapse could be documented). Dental relapse in the anterior dentition was felt to be the cause of recurrent malocclusion. A, Frontal views in repose before and after treatment. B, Frontal views with smile before and after treatment. C, Oblique facial views before and after treatment. D, Profile views before and after treatment. E, Occlusal views before retreatment (after four bicuspid extractions), before surgery, and then after treatment. F, Articulated dental casts that indicate analytic model planning. G and H, Computed tomography scans of the maxillomandibular complex that indicate congenital condyle glenoid fossa dysplasia. I, Lateral cephalometric radiographs before and after treatment.

Basic Surgical Approach

In addition to lower jaw advancement (i.e., sagittal ramus osteotomies), maxillary surgery to correct arch-width discrepancies may be required for the individual with primary mandibular deficiency. This is generally accomplished by segmental maxillary osteotomies. Through the Le Fort I down-fracture, any needed improvement in the upper-lip-to-upper-tooth relationship can also be simultaneously carried out via the alteration of the vertical height. Any deficiencies in the horizontal position (i.e., projection) of the maxilla are also managed. The correction of a maxillary midline discrepancy (i.e., yaw), the aesthetic advantages of maxillary plane alteration (i.e., pitch), cant correction (i.e., roll), and intranasal procedures (i.e., septoplasty and inferior turbinate reduction) can also be simultaneously accomplished through the Le Fort I osteotomy (see Chapter 12).

In cases of only borderline maxillary deformity, limiting the surgery to the lower jaw is generally possible to manage the malocclusion and to address aesthetic aspects.

• When the decision is made to limit surgery to the mandible and with only a minimum arch-width discrepancy, the option of narrowing the mandible (i.e., a midline symphyseal split) may be considered.

• When only borderline maxillary vertical deficiency coexists (i.e., not enough to warrant a Le Fort I osteotomy), additional surgical vertical lengthening of the chin may be incorporated to achieve an aesthetic advantage.

• When mandibular deficiency is combined with a less than ideal clockwise rotated and vertically deficient maxilla, the surgically advanced mandible (without simultaneous Le Fort I osteotomy) may not achieve the preferred profile aesthetics (i.e., there will be a suboptimal A-point to B-point relationship in profile). In borderline cases, a vertical lengthening and a limited horizontal advancement osseous genioplasty may provide the necessary degree of aesthetic compensation.

imageCOMMENT: When there are only borderline indications for maxillary surgery, the ability to fine-tune facial aesthetics with a compensating osseous genioplasty may exist (see Chapter 37).48 Nevertheless, it is important to understand that a percentage of patients who present with primary mandibular deficiency will also have degrees of maxillary dysmorphology that are best served by double jaw surgery.

Postsurgical Orthodontic Maintenance and Detailing

If the patient has a presurgical habit of posturing the mandible forward (i.e., a dual bite), there may also be a tendency to do so during the immediate postoperative healing phase. Intermittent elastics are used during this time 1) to retrain the masticatory muscles, 2) to provide movement in the temporomandibular joints, 3) to increase patient comfort, and 4) to confirm the stability of the occlusion.

If segmental maxillary osteotomies were also carried out, a prefabricated splint will have been secured to the maxillary occlusal surfaces during surgery. The orthodontist sees the patient within 24 hours of splint removal (approximately 5 weeks after surgery) and replaces the maxillary sectional arch wires with a rigid continuous arch wire. The maxillary teeth are ligated together to maintain the arch form. The use of a transpalatal appliance or a palatal plate may also be used to stabilize and maintain the arch form if significant transverse expansion was carried out.12 Close orthodontic monitoring for skeletal and dental shifts during the first 6 months after surgery is essential (see Chapter 17).1,8,9,10,16,22,26,33,54,56,63

The postsurgical orthodontic objectives should be to bring the teeth into ideal occlusion and to maintain the surgical result. In the patient in whom the curve of Spee is to be fully leveled postoperatively, the use of vertical elastics to close the mid-arch open bites and for the full closure of the bicuspid extraction sites will also be priorities. This process can be initiated immediately after surgery.

Complications, Informed Consent, and Patient Education

The orthodontist is frequently asked to evaluate a preadolescent child who is presenting with primary mandibular deficiency. The clinician may attempt to alter nature’s events through growth modification maneuvers.53 Clinical studies now confirm limited success with a growth modification approach if the goal is to “normalize” the deficient lower jaw. The camouflage orthodontic approach that often follows may neutralize the occlusion, but it will not improve baseline upper airway dysfunction, and it will not enhance the appearance of a retrusive profile. Furthermore, it may have detrimental effects on the periodontium (see Figs. 19-1 through 19-5). The onus is on the clinician to communicate the shortcomings of these isolated orthodontic maneuvers to the patient and the family before beginning treatment.

After a combined surgical and orthodontic approach is agreed to, the clinician’s ability to communicate the potential risks, complications, and benefits of the recommended coordinated plan to the patient and the family is essential (see Chapter 17).19,24 The patient’s and the family’s informed decision to proceed provides the basis for shared responsibility of the outcome (see Chapter 7).

Temporomandibular Disorders: The Effects of Orthodontics and Orthognathic Surgery

Temporomandibular disorders (TMDs) include various symptoms and signs of the temporomandibular joint, the masticatory muscles, and related structures. The symptoms and signs may include a spectrum of referred head and neck pain; joint noise (e.g., popping, clicking, crepitus); reduced or altered mandibular movements (e.g., muscle spasm or disc displacement with or without reduction); condylar head erosion; and pain on direct palpation of either temporomandibular joint or of the masticatory muscles.

Occlusal features more likely to be associated with symptomatic TMDs include a centric relation to centric occlusion slide of more than 2 mm; overjets of more than 4 mm; and the presence of a skeletal anterior open bite. It is likely for these reasons that White and colleagues found that TMDs were significantly greater among patients with Class II skeletal deformities as compared with those patients with Class III deformities.58 Westermark and colleagues studied a consecutive series of Class II patients to understand the effects of orthognathic surgery on the signs and symptoms of TMD.57 Preoperatively, 43% of the patients reported subjective symptoms of TMD; postoperatively, 28% reported such symptoms. This difference suggests an overall beneficial effect of orthognathic surgery for many individuals with Class II mandibular deficiencies.28 Interestingly, clinical studies indicate that patients with mandibular retrusion and TMD do not improve as much from an orthognathic correction as patients with a Class III skeletal pattern do (see Chapter 9).

Conclusions

In the American and Northern European populations, mandibular deficiency is the most frequently seen developmental jaw deformity. This results in an Angle Class II malocclusion with an excessive overjet and often with a deep overbite. The mandible and the chin region will be deficient, and the maxilla may also be deformed. The basic facial dysmorphology in an individual with primary mandibular deficiency is that of diminished vertical and horizontal strength in the lower third of the face. There will be lack of support for the overlying soft tissues that results in an overly obtuse neck–chin angle, with double-chin formation and early jowling. The tongue will be retropositioned within a small oral cavity and have negative effects on the airway. The laxity of the soft-tissue envelope only worsens throughout the normal aging process.

The correction of this frequently seen facial imbalance requires the orthodontic removal of dental compensations, often with extractions. Orthognathic surgery is performed for the achievement of a stable occlusion, long-term periodontal health, improvements in the upper airway, and the enhancement of facial aesthetics.

References

1. Abeloos, J, De Clercq, C, Neyt, L. Skeletal stability following miniplate fixation after bilateral sagittal split osteotomy for mandibular advancement. J Oral Maxillofac Surg. 1993; 91:624.

2. Angle, EH. Treatment of malocclusion of the teeth: Angle’s system, ed 7. Philadelphia: S. S. White; 1907.

3. Arnett, GW, Bergman, RT. Facial keys to orthodontic diagnosis and treatment planning—part I. Am J Orthod Dentofacial Orthop. 1993; 103:299–312.

4. Arnett, GW, Bergman, RT. Facial keys to orthodontic diagnosis and treatment planning—part II. Am J Orthod Dentofacial Orthop. 1993; 103:395–411.

5. Bell, WH, Proffit, WR, White, RP. Surgical correction of dentofacial deformities. Philadelphia: W. B. Saunders Company; 1980.

6. Bell, WH, Schendel, SA. Biologic basis for modification of the sagittal split ramus osteotomy. J Oral Surg. 1977; 35:362.

7. Blair, VP. Surgery of the mouth and jaws. St. Louis: Mosby; 1914.

8. Blomqvist, JE, Ahlborg, G, Isaksson, S, et al. A comparison of skeletal stability after mandibular advancement and use of two rigid fixation techniques. J Oral Maxillofac Surg. 1997; 55:568.

9. Bouwman, JP, Kerstens, HC, Tuinzing, DB. Condylar resorption in orthognathic surgery. The role of intermaxillary fixation. Oral Surg Oral Med Oral Pathol. 1994; 78:138.

10. Broadbend, TR, Woolf, RM. Our experience with sagittal split osteotomy for retrognathia. Plast Reconstr Surg. 1977; 60:860.

11. Burstein, FD, Cohen, SR, Scott, PH, et al. Surgical therapy for severe refractory sleep apnea in infants and children: application of the airway zone concept. Plast Reconstr Surg. 1995; 96:34.

12. Chamberland, S, Proffit, WL. Short-term and long-term stability of surgically assisted rapid palatal expansion revisited. Am J Orthod Dentofacial Orthop. 2011; 139:815–822.

13. Coben, SE. Growth and Class II treatment. Am J Orthod. 1966; 52:5–26.

14. Cozza, P, Baccetti, T, et al. Mandibular changes produced by functional appliances in Class II malocclusion: A systematic review. Am J Orthod Dentofacial Orthop. 2006; 129:599e1–599e12.

15. Epker, BN. Occlusal plane alteration in orthognathic surgery [discussion]. J Oral Maxillofac Surg. 1993; 51:740–741.

16. Epker, BN, Wessberg, GA. Mechanisms of early skeletal relapse following surgical advancement of the mandible. Br J Oral Surg. 1982; 20:175–182.

17. Ewing, M, Ross, RB. Soft tissue response to mandibular advancement and genioplasty. Am J Orthod Dentofacial Orthop. 1992; 101:550–555.

18. Finn, RA. Biomechanical considerations in the surgical correction of mandibular deficiency. J Oral Surg. 1980; 38:257–264.

19. Friehofer, H, Petresevic, D. Late results after advancing the mandible by sagittal splitting of the rami. J Maxillofac Surg. 1975; 3:250–257.

20. Friehofer, HP. Results of osteotomies of the facial skeleton in adolescence. J Maxillofac Surg. 1977; 5:267–297.

21. Gallo, W, Moss, M, Gaul, J. Modification of the sagittal split ramus osteotomy for retrognathia. J Oral Surg. 1976; 34:178–179.

22. Guernse, YL. Stability of treatment results in Class II malocclusion corrected by full mandibular advancement surgery. Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 1974; 37:668.

23. Huang, CS, Bruce-Ross, R. Surgical advancement of the retrognathic mandible in growing children. Am J Orthod. 1982; 82:89–103.

24. Jackson, L. Outpatient orthognathic surgery: review of 205 cases [discussion]. J Oral Maxillofac Surg. 1997; 55:563.

25. Jacobs, JS. Mandibular retrusion, temporomandibular joint derangement, and orthognathic surgery planning [discussion]. Plast Reconstr Surg. 1992; 90:230.

26. Jäger, A, Kubein-Meesenburg, D, Luhr, HG. Longitudinal study of combined orthodontic and surgical treatment of Class II malocclusion with deep overbite. Int J Adult Orthod Orthognath Surg. 1991; 6:29.

27. Johnston, LEJr. Level of agreement in clinicians’ perceptions of Class II malocclusions [discussion]. J Oral Maxillofac Surg. 1994; 52:572.

28. Magnusson, T, Ahlborg, G, Svartz, K. Function of the masticatory system in 20 patients with mandibular hypo- or hyperplasia after correction by a sagittal split osteotomy. Int J Oral Maxillofac Surg. 1990; 19:289.

29. McNeil, RW, West, RA. Severe mandibular retrognathism: orthodontic versus surgical orthodontic treatment. Am J Orthod. 1977; 72:176–182.

30. Newman, GV. Prevalence of malocclusion in children six to fourteen years of age and treatment in preventable cases. J Am Dent Assoc. 1965; 52:566–575.

31. Ngan, PW, Byczek, E, Scheick, J. Longitudinal evaluation of growth changes in Class II Division 1 subjects. Semin Orthod. 1997; 3:222–231.

32. O’Brian, K. Is early treatment for Class II malocclusion effective? Results from a randomized clinical trial. Am J Orthod Dentofacial Orthop. 2006; 120(Suppl 1):564–565.

33. O’Brian, K, Wright, J, Conboy, F, et al. Effectiveness of early orthodontic treatment with the twin-block appliance: a multi-center, randomized, controlled trial. Part 2: Psychosocial effects. Am J Orthod Dentofacial Orthop. 2003; 124:488–495.

34. Obwegeser, HL. The surgical correction of mandibular prognathism and retrognathia with consideration of genioplasty: part 1. Oral Surg. 1957; 10:677.

35. Obwegeser, HL. The indications for surgical correction of mandibular deformity by the sagittal splitting technique. Br J Oral Surg. 1964; 1:157–171.

36. Obwegeser, HL. Variations of a standard approach for correction of the bird-face deformity. J Craniomaxillofac Surg. 1988; 16:247–265.

37. Obwegeser, HL, Hadjianghelou, O. Two ways to correct bird-face deformity. Oral Surg Oral Med Oral Pathol. 1987; 64:507–518.

38. Otuyemi, OD, Jones, SP. Long-term evaluation of treated Class II Division 1 malocclusions utilizing the PAR index. Br J Orthod. 1995; 22:171–178.

39. Phillips, C, Bailey, LJ, Sieber, RP. Level of agreement in clinicians’ perceptions of Class II malocclusions. J Oral Maxillofac Surg. 1994; 52:565.

40. Posnick, J, Fantuzzo, J, Orchin, J. Deliberate operative rotation of the maxillo-mandibular complex to alter the A-point to B-point relationship for enhanced facial esthetics. J Oral Maxillofac Surg. 2006; 64:1687–1695.

41. Posnick, JC. Occlusal plane rotation: aesthetic enhancement in mandibular micrognathia [discussion]. Plast Reconstr Surg. 1993; 91:1241.

42. Posnick JC, ed. Mandibular deficiency with or without maxillary deformity. Craniofacial and maxillofacial surgery in children and young adults. W. B. Saunders Company: Philadelphia, 2000:981–1003.

43. Proffit, WR, Fields, HW, Jr., Moray, LJ. Prevalence of malocclusion and orthodontic treatment need in the United States: estimates from the NHANES III survey. Int J Adult Orthodon Orthognath Surg. 1998; 13:97.

44. Proffit, WR, Phillips, C, Douvartzidis, N. A comparison of outcomes of orthodontic and surgical-orthodontic treatment of Class II malocclusion in adults. Am J Orthod Dentofacial Orthop. 1992; 101:556–565.

45. Reichenback, E. Uber den zeitpunk kieferchirurgisch-orthod-padischer, Eingriffe, Stsch. Zahn-Mund-Kierferheilkd. 1966; 47:302.

46. Reyneke, JP, Evans, WG. Surgical manipulation of the occlusal plane. Int J Adult Orthodon Orthognath Surg. 1990; 5:99–110.

47. Rosen, H. Aesthetics in facial skeletal surgery. Perspect Plast Surg. 1993; 6:1.

48. Rosen, HM. Surgical correction of the vertically deficient chin. Plast Reconstr Surg. 1988; 82:247.

49. Rosen, HM. Occlusal plane rotation: aesthetic enhancement in mandibular micrognathia. Plast Reconstr Surg. 1993; 91:1231.

50. Rowe, NL. The etiology, clinical features and treatment of mandibular deformity. Br Dent J. 1960; 108:45–112.

51. Schellhas, KP, Piper, MA, Bessette, RW, et al. Mandibular retrusion, temporomandibular joint derangement, and orthognathic surgery planning. Plast Reconstr Surg. 1992; 90:218.

52. Schendel, SA, Wolford, LM, Epker, BN. Mandibular deficiency syndrome. III. Surgical advancement of the deficient mandible in growing children: treatment results in twelve patients. Oral Surg Oral Med Oral Pathol. 1978; 45:364–377.

53. Simmons, KE, Turvey, TA, Phillips, C, Proffit, WR. Surgical orthodontic correction of mandibular deficiency: five-year follow-up. Int J Adult Orthodon Orthognath Surg. 1992; 7:67–80.

54. Snow, MD, Turvey, TA, Walker, D, et al. Surgical mandibular advancement in adolescents: postsurgical growth related to stability. Int J Adult Orthodon Orthognath Surg. 1991; 6:143.

55. Stahl, F, Baccetti, T, Franchi, L, McNamara, JA. Longitudinal growth changes in untreated subjects with Class II Division 1 malocclusion. Am J Orthod Dentofacial Orthop. 2008; 134:125–137.

56. Van Sickels, JE. A comparative study of bicortical screw and suspension wires versus bicortical screws in large mandibular advancements. J Oral Maxillofac Surg. 1991; 49:1293.

57. Westermark, A, Shayeghi, F, Thor, A. Temporomandibular dysfunction in 1516 patients before and after orthognathic surgery. Int J Adult Orthodon Orthognath Surg. 2001; 16:145–151.

58. White, CS, Dolwick, MF. Prevalence and variance of temporomandibular dysfunction in orthognathic surgery patients. Int J Adult Orthodon Orthognath Surg. 1992; 7:7–14.

59. Wolfe, SA, Berkowitz, S. The mandible. In: Wolfe SA, Berkowitz S, eds. Plastic surgery of the facial skeleton. Boston: Little, Brown; 1989:149.

60. Wolford, LM, Chemello, PD, Hilliard, FW. Occlusal plane alteration in orthognathic surgery. J Oral Maxillofac Surg. 1993; 51:730.

61. Woldford, LM, Schendel, SA, Epker, BN. Surgical orthodontic correction of mandibular deficiency in growing children (long term treatment result). J Maxillofac Surg. 1979; 7:61–72.

62. Yu, LF, Pogrel, MA, Ajayi, M. Pharyngeal airway changes associated with mandibular advancement. J Oral Maxillofac Surg. 1994; 52:40.

63. Zarrinkelk, HM, Throckmorton, GS, Ellis, E, III., et al. A longitudinal study of changes in masticatory performance of patients undergoing orthognathic surgery. J Oral Maxillofac Surg. 1995; 53:777.