Microdermabrasion

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23 Microdermabrasion

Microdermabrasion (MDA) is a superficial skin resurfacing procedure that utilizes gentle mechanical abrasion to remove the outermost layers of the epidermis.1 Removal of outer skin layers, also called exfoliation, has been used for skin rejuvenation since 1500 BC, when the ancient Egyptians used sandpaper and sour milk baths containing lactic acid. Microdermabrasion uses refined abrasive elements, such as diamond-tipped pads or a constant flow of crystals swept across the skin, to remove the stratum corneum.2 Due to popular marketing, patients are aware of MDA and it is often considered as an initial treatment for aesthetic rejuvenation. It is one of the most commonly performed aesthetic procedures in the United States today, with more than half a million MDA treatments performed annually, according to data from the American Society for Aesthetic Plastic Surgery,3 and it is one of the most common aesthetic procedures incorporated into office practice.4,5

Mechanical exfoliation of the skin ranges in depth from very superficial microbead scrubs found over the counter, which partially remove the stratum corneum, to deep operative procedures, such as laser resurfacing and dermabrasion, which can ablate the reticular dermis. The target depth for most microdermabrasion procedures is removal of the stratum corneum; however, the depth of resurfacing achieved with MDA can vary from the stratum corneum to the upper papillary dermis.

Skin rejuvenation with microdermabrasion is based on the principles of wound healing. By wounding and removing superficial skin layers in a controlled manner, cell renewal is stimulated with regeneration of a healthier epidermis and dermis.1 After a series of MDA treatments, histologic changes in the skin are evident. These changes include a compacted stratum corneum and smoother epidermis, increased dermal thickness with fibroblast production of new collagen and elastin,6 and increased skin hydration with improved epidermal barrier function.7,8 Clinical improvements can be seen in hyperpigmentation9 and rough skin texture.10 Some studies also show improvements in fine lines, pore size, superficial acne scars, and acne vulgaris.7,9,11 MDA can be readily combined with other minimally invasive aesthetic procedures, such as chemical peels and nonablative laser treatments, many of which can be performed in the same visit, to enhance skin rejuvenation results.12

Patient Selection

While almost any patient will benefit from exfoliation with MDA, patients with mild to moderate photoaging changes of solar lentigines, rough texture, and fine lines (e.g., Glogau types I and II) typically derive the most noticeable benefits (see Chapter 19, Aesthetics Principles and Consultation, for a description of Glogau types). Results with MDA are slow and progressive, requiring a series of treatments for visible improvements. Assessment of patients’ expectations at the time of consultation and commitment to a series of treatments is essential to the success of these treatments.

Patients of all Fitzpatrick skin types may be treated with MDA (see Chapter 19 for a description of Fitzpatrick skin types). It is advisable to treat darker skin types (IV through VI) conservatively because they have increased risks of pigmentary changes such as postinflammatory hyperpigmentation. Elderly patients with thin and friable skin and patients with erythematous conditions such as rosacea, telangiectasias, and poikiloderma of Civatte are also treated conservatively, because they have greater risks of abrasion and worsening of erythema respectively.

Alternative Therapies

The depth of skin resurfacing with MDA is comparable to a light chemical peel, which is a reasonable alternative treatment for skin rejuvenation. MDA offers certain advantages over chemical peels such as greater control over the depth of exfoliation, comparatively less discomfort, and no “downtime” for skin flaking and peeling. Superficial dermaplaning, performed with a specialized scalpel blade gently scraped across the skin, is a good alternative to MDA for patients with erythematous conditions such as rosacea, because there is no vacuum suction with this procedure to potentially exacerbate erythema. Dermaplaning requires greater treatment times and takes longer to acquire proficiency than MDA.

For more aggressive skin resurfacing, procedures such as medium-depth chemical peels, dermabrasion, or laser resurfacing are required. Dermabrasion involves the use of a high-speed rotating brush to abrade the skin and can penetrate to the reticular dermis. Laser resurfacing can also penetrate to the reticular dermis, and may be performed as an ablative procedure, where the epidermis is removed (e.g., with 2940 nm wavelength), or as a nonablative procedure, where the epidermis remains intact (e.g., with 1550 nm wavelength). Relative to MDA, these more aggressive resurfacing procedures offer significantly greater reduction of wrinkles and improvements in photodamaged skin. However, deeper resurfacing procedures such as these have longer recovery times and can be associated with complications such as scarring, dyspigmentation, and infection.

Microdermabrasion will not improve deep wrinkles and folds related to volume loss or hyperdynamic musculature, which respond to treatment with dermal fillers and botulinum toxin, respectively.

Microdermabrasion Devices Currently Available

Most MDA devices utilize a closed-loop vacuum system, which draws the skin up to an abrasive element on the handpiece tip. The handpiece is passed across the skin and cellular debris is removed and suctioned up into a collection container and disposed of after treatment. The process of MDA exfoliation using a diamond-tipped device is shown in Figure 23-1. Abrasive elements used by MDA devices vary and the most commonly used elements are:

image

FIGURE 23-1 Microdermabrasion exfoliation process.

(SilkPeel™ Envy Medical, Los Angeles, CA.)

 

Aluminum oxide (typically 100 µm in diameter) is the crystal most often used for MDA. It is inert, very hard, water insoluble, and its crystalline structure has multiple sharp edges, making it good for abrasion. Other types of crystals less commonly used include sodium chloride, sodium bicarbonate, and magnesium oxide. Most crystal devices blow crystals across the skin and aspirate them along with skin debris into a closed container. Some devices use crystal covered pads rather than aerosolized crystals. Aerosolized crystal devices have the disadvantage of leaving a dust residue, and have risks of ocular injury and dust inhalation. Crystals are not reusable but their expense is modest.

Crystal-free MDA devices have become popular because they lack these ocular and inhalation risks. Common crystal-free abrasive elements include diamond-tipped pads and bristles. Figure 23-2 shows a MDA handpiece with diamond-tipped treatment heads of various coarseness. Most of these devices have reusable treatment heads that can be sterilized following treatment. Some crystal-free devices combine simultaneous application of topical solutions during exfoliation, called infusion. The intent is to deliver topical products more effectively to the skin by taking advantage of the transient disruption of the epidermal barrier. Selection of topical solutions is based on the presenting condition. For example, hydroquinone or cosmeceuticals such as kojic acid and decapeptide-12 may be used for patients with hyperpigmentation; erythromycin and salicylic acid for acne and rosacea; and hyaluronic acid and glycerin for dehydrated skin.

MDA devices are classified by the FDA as type I devices, which do not require the manufacturer to establish performance standards or perform clinical trials to demonstrate efficacy. With more than 30 MDA devices available, this presents a challenge to providers when selecting a device. Most MDA machines are either manufactured for estheticians (“esthetician grade”) or for clinical providers (“medical grade”), with the latter capable of deeper exfoliation with higher vacuum pressures and more abrasive treatment heads. Commonly used MDA devices are listed in the Resources section at the end of the chapter.

Anatomy

Histologic evaluation of skin immediately after treatment with MDA demonstrates smoothing of the stratum corneum.13 Each pass of an aluminum oxide crystal MDA handpiece removes approximately 10 to 15 µm of skin, two passes fully remove the stratum corneum, and four passes penetrate to the stratum granulosum layer of the epidermis.14 The epidermal barrier function is transiently disrupted for 2 to 3 days after treatment with increased transepidermal water loss. These effects, however, are reversed 1 week after treatment, and a more compacted stratum corneum is regenerated with improved barrier function and increased skin hydration compared to pretreatment skin.15,16

In addition to short-term improvements in epidermal barrier function, longer term improvements have also been demonstrated in the epidermis and dermis following repeated MDA treatments. Epidermal thickness increases by up to 40% due to increased cellularity,7 and dermal collagen and elastin deposition increase, which can be seen clinically as a reduction of coarse pores and fine lines.6

Microdermabrasion: Steps and Principles

The following recommendations are guidelines for microdermabrasion treatments on the face using the SilkPeel (manufactured by Envy), which is a crystal-free device that utilizes diamond-tipped pads as the abrasive element with simultaneous infusion of topical solutions. Comparisons and recommendations for crystal and bristle MDA devices are also included where possible. Recommended treatment parameters vary according to the device used, and specific manufacturer guidelines should be followed at the time of treatment.

General Treatment Technique

MDA may be performed as a very superficial or superficial skin resurfacing procedure (see Chapter 19 for definitions of resurfacing terminology). Greater depths of penetration have greater potential for improvements. Although MDA has few risks, greater depths of penetration with any exfoliation procedure can be associated with a greater risk of complications.

Safety Zone

The area within which MDA treatments can be safely performed on the face, called the Microdermabrasion Safety Zone, is shown in Figure 23-3. All areas of the face may be treated, apart from the area within the bony orbital rim and, for most devices, the lips. Some MDA devices have nonabrasive smooth treatment tips that may be used on the lips (Figure 23-2). MDA can be used on the face, neck, chest, hands, back, and almost any area of the body requiring exfoliation.

Performing Microdermabrasion

 

TABLE 23-1 Treatment Parameters for SilkPeel Microdermabrasion

Treatment Intensity

Tip Coarseness

Vacuum Pressure (psi)

Mild 140 grit 3.0–4.0
Moderate 120 grit 3.5–4.3
Aggressive 100 grit 4.4–5.0
6. Select the vacuum setting by inverting the handpiece and occluding the tip with a gloved finger (Figure 23-4). The strength of the vacuum affects the depth of resurfacing, and small adjustments in this parameter can fine-tune the intensity of a treatment. Vacuum pressures are device dependent and the manufacturer’s recommended settings should be used. In general, conservative vacuum settings should be selected for initial treatments.
8. The direction of handpiece strokes on the face are from the medial face toward the periphery (Figure 23-5). Begin at the forehead, then proceed down the nose, cheeks, chin, and lastly, around the mouth.

Results

Microdermabrasion is a progressive treatment. A slight improvement in skin texture or skin brightness after a single treatment may be evident. However, for more marked results and improvements in pigmentation, acne, fine lines, and other conditions, a series of six treatments is usually necessary.

Figure 23-8 shows a patient demonstrating signs of moderate photodamage with solar lentigines, dullness, and fine lines (A) before and (B) after a series of six treatments with a bristle MDA using an infusion of topical solutions containing glycolic and lactic acid, and cosmeceutical lightening agents of kojic and azelaic acids, bearberry, and licorice.

Figure 23-9 shows a patient with papulopustular acne (A) before and (B) after a series of six MDA treatments with a diamond-tipped MDA using an infusion of topical solutions containing salicylic, glycolic and kojic acids and arbutin.

Figure 23-10 shows a patient with darker Fitzpatrick skin type with solar lentigines and dullness (A) before and (B) after a series of six MDA treatments with a diamond-tipped MDA using an infusion of topical solutions containing a lightening peptide, decapeptide-12 (Lumixyl™).

Complications

 

MDA has minimal risks of side effects and complications.1 One study of more than 100 patients receiving MDA during a 2-year period reported no instances of infection, long-term hyperpigmentation, or scarring.18 However, complications are possible with any procedure, and knowledge of these is important to help ensure the best possible outcomes.

Postprocedure erythema is common and the duration depends on the aggressiveness of the procedure and inherent reactivity of the patients’ skin. Most erythema resolves spontaneously within a few hours to 1 day. Prolonged erythema of more than 5 days, particularly in patients with darker Fitzpatrick skin types (IV through VI), can result in postinflammatory hyperpigmentation (PIH). If erythema is marked immediately after treatment, ice can be applied to the skin for 15 minutes every hour followed by a topical steroid cream. A high-potency topical steroid cream may be used in the office (e.g., triamcinolone 0.5%) and the patient sent home with a low-potency steroid (e.g., hydrocortisone 2.5% or 1%) to be used twice daily for 3 to 5 days or until erythema resolves. Sun avoidance is very important to reduce the risk of PIH when erythema is present.

Excessive dryness or pruritis can be managed with application of moisturizers. Use of highly occlusive moisturizers, such as Aquaphor, will reduce these symptoms but can be associated with development of acne and milia. Therefore, thinner, less occlusive moisturizer formulations (e.g., Epidermal Repair by SkinCeuticals) with frequent application is preferable to manage dryness and pruritis.

Superficial abrasions can occur with deeper MDA penetration. These are usually erythematous immediately after treatment and can either hyperpigment or crust slightly. Superficial abrasions are also referred to as striping, due to the appearance of lines on the skin. Abrasions may also be circular if the tip has dwelled too long or excessive downward pressure was applied in one spot. Crusting is managed with moist wound care using a topical antibiotic ointment (e.g., Bacitracin) as needed daily until healed.

Hyperpigmentation can be treated with lightening agents such as hydroquinone or cosmeceutical agents such as kojic acid, arbutin, and licorice (see Chapter 24, Skin Care Products). In darker Fitzpatrick skin types, resolution of hyperpigmentation typically takes several months. Hyperpigmentation usually resolves, but in rare instances can be permanent.

Urticaria has been reported as a rare complication after MDA,19 and is presumed to be dermatographic or pressure-induced. Products applied during treatment may also trigger an allergic response. Ice may be applied, an oral antihistamine (e.g., cetirizine 10 mg) given to the patient, and a high-potency topical steroid applied to the treatment area. Although extremely unlikely, it is also advisable to assess patients for signs and symptoms of more severe allergic responses such as bronchospasm or anaphylaxis if an allergic reaction occurs.

Petechiae and purpura can occur with overly aggressive vacuum settings, particularly in older patients with thin skin or if using anticoagulants. If petechiae occur, vacuum settings should be decreased and the affected area avoided for the remainder of treatment. Petechiae resolve more rapidly than purpura, which can take up to 2 weeks to clear.

Scarring is a remote possibility and can occur if treatment parameters are aggressive, and is more likely if infection occurs or crusts are excoriated. Hypopigmentation is also a rare possibility, with greater risks in darker Fitzpatrick skin types. Hypopigmentation can be a temporary or permanent complication.

Ocular injury due to crystal adherence to the conjunctiva or punctuate keratopathy can occur with crystal MDA devices. Symptoms include ocular pain, photophobia, and conjunctival erythema.11 Protective eye shields or moist gauze is advised with crystal MDA to cover patients’ eyes, appropriate eyewear and masks for providers, and removal of all crystals from the face following treatment.

Treating Specific Lesions

Erythematous conditions such as rosacea, telangiectasia, and poikiloderma of Civatte. Poikiloderma of Civatte is a pattern of erythema and mottled pigmentation commonly seen on the sides of the neck, face, and chest due to photodamage (Figure 23-11). Treatment of erythematous conditions is controversial with MDA. Clearly, excessive vacuum pressures can accentuate superficial vasculature, making erythema worse. However, in rosacea patients, for example, who have impaired barrier function, mild MDA treatments performed at low vacuum settings, may ultimately strengthen the epidermal barrier, reducing erythema.20 Some providers, therefore, do perform MDA on patients with erythematous conditions using reduced vacuum settings in areas of high vascularity such as the midface and chin, and higher settings around the periphery of the face (Figure 23-12). Patients intolerant of MDA may alternatively receive superficial dermaplaning.
image

FIGURE 23-11 Poikiloderma of Civatte.

(Copyright Rebecca Small, MD.)

References

1. Grimes PE. Microdermabrasion. Derm Surg. 2005;31(9 Pt 2):1160-1165.

2. Small R. Microdermabrasion. In: Mayeaux E, editor. The Essential Guide to Primary Care Procedures. Philadelphia: Lippincott Williams & Wilkins; 2009:265-277.

3. Cosmetic Surgery National Data Bank 2010 Statistics. American Society for Aesthetic Plastic Surgery; 2010.

4. Small R. Aesthetic procedures in office practice. Am Fam Physician. 2009;80(11):1231-1237.

5. Small R. Aesthetic procedures introduction. In: Mayeaux E, editor. The Essential Guide to Primary Care Procedures. Philadelphia: Lippincott Williams & Wilkins; 2009:195-199.

6. Rubin MG, Greenbaum S. Histologic effects of aluminum oxide microabrasion on facial skin. J Aesthet Dermatol Cosm Surg. 2000;1(4):237-239.

7. Coimbra M, Rohrich RJ, Chao J, et al. A prospective controlled assessment of microdermabrasion for damaged skin and fine rhytides. Plast Reconstr Surg. 2004;113(5):1438-1443.

8. Karimipour DJ, Kang S, Johnson TM, et al. Microdermabrasion with and without aluminum oxide crystal abrasion: a comparative molecular analysis of dermal remodeling. J Am Acad Dermatol. 2006;54(3):405-410.

9. Shim EK, Barnette D, Hughes K, et al. Microdermabrasion: a clinical and histopathologic study. Derm Surg. 2001;27(6):524-530.

10. Hernandez-Perez E, Ibiett EV. Gross and microscopic findings in patients undergoing microdermabrasion for facial rejuvenation. Derm Surg. 2001;27(7):637-640.

11. Tsai RY, Wang CN, Chan HL. Aluminum oxide crystal microdermabrasion. A new technique for treating facial scarring. Derm Surg. 1995;21(6):539-542.

12. Briden ME, Jacobsen E, Johnson C. Combining superficial glycolic acid (alpha-hydroxy acid) peels with microdermabrasion to maximize treatment results and patient satisfaction. Cutis. 2007;79(1 Suppl):13-16.

13. Koch RJ, Hanasono MM. Microdermabrasion. Facial Plast Surg Clin North Am. 2001;9(3):377-382.

14. Blome D. Microdermabrasion. In Pfenninger JL, Fowler GC, editors: Pfenninger and Fowler’s Procedures for Primary Care, 3rd ed, Philadelphia: Mosby/Elsevier, 2010.

15. Freedman BM, Rueda-Pedraza E, Waddell SP. The epidermal and dermal changes associated with microdermabrasion. Derm Surg. 2001;27(12):1031-1033.

16. Rajan P, Grimes PE. Skin barrier changes induced by aluminum oxide and sodium chloride microdermabrasion. Derm Surg. 2002;28(5):390-393.

17. Nootheti PK, Gold MH, Goldman MP. Photodynamic therapy for photorejuvenation. In: Goldman MP, editor. Photodynamic Therapy. Philadelphia: Saunders/Elsevier; 2008:125-135.

18. Freeman MS. Microdermabrasion. Facial Plast Surg Clin North Am. 2001;9(2):257-266.

19. Farris P, Rietschel R. An unusual response to microdermabrasion. Dermatol Surg. 2002;28:606-608.

20. Desai T, Moy RL. Evaluation of the SilkPeel system in treating erythematotelangectatic and papulopustular rosacea. Cosm Dermatol. 2006;19:51-57.

21. Taub A. Photodynamic therapy: other uses. Dermatol Clin. 2007;25(1):101-109.