KNEE

Published on 16/03/2015 by admin

Filed under Orthopaedics

Last modified 16/03/2015

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CHAPTER 11

KNEE

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Précis of the Knee Assessment*

History

Observation

Examination

Active movements

Passive movements (as in active movements)

Resisted isometric movements

Tests for ligament stability

Test for one-plane medial instability

Test for one-plane lateral instability

Tests for one-plane anterior and posterior instabilities

Tests for anteromedial and anterolateral rotary instabilities

Tests for posteromedial and posterolateral rotary instabilities

Special tests

Reflexes and cutaneous distribution

Joint play movements

Palpation

Diagnostic imaging


*Although an examination of the knee may be performed with the patient in the supine position, some of the tests may require the patient to move to other positions (e.g., standing, lying, prone, sitting). When these tests are used, the examination should be planned so that movements (and therefore the patient’s discomfort) are kept to a minimum. The sequence should proceed from standing, to sitting, to supine lying, to side lying, and finally to prone lying. After any examination, the patient should be warned that the assessment may result in an exacerbation of symptoms.

SELECTED MOVEMENTS

ACTIVE MOVEMENTS1,2 image

Extension

INDICATIONS OF A POSITIVE TEST

Active knee extension is approximately 0º but may be −15º, especially in women, who are more likely to have hyperextended knees (genu recurvatum). If the range of movement is less than this or is less than in the unaffected leg, the test result is positive. The knee extensor muscles develop the greatest force near 60º, and the knee flexor muscles develop their greatest force between 45º and 10º. To complete the last 15º of knee extension, a 60% increase in the force of the quadriceps muscles is required. Therefore, the examiner should watch for evidence of quadriceps lag, which means the quadriceps muscles are not strong enough to fully extend the knee. The lag results from loss of mechanical advantage, muscle atrophy, decreasing power of the muscle as it shortens, adhesion formation, effusion, or reflex inhibition that results in instability of the knee.

PATELLAR MOBILITY3,4 image

SPECIAL TESTS FOR ONE-PLANE MEDIAL INSTABILITY

Relevant Special Tests

Mechanism of Injury

The MCL/TCL functions to restrain valgus stress and lateral rotation of the tibia. A blow to the outside of the knee most commonly injures the ligament. Contact injuries involving direct valgus loading to the knee are the usual mechanism in a complete tear. Noncontact, or indirect, injuries occur with deceleration, cutting, and pivoting motions. Anatomically, the MCL/TCL is composed of two layers, the superficial layer and the deep layer; the deep layers attach to the medial meniscus.

Avulsion of ligaments generally occurs between the unmineralized and mineralized fibrocartilage layers. MCL/TCL injury occurs most often at the femoral attachment (65% of cases).611

ABDUCTION (VALGUS STRESS) TEST12,13 image

HUGHSTON’S VALGUS STRESS TEST12 image

SPECIAL TESTS FOR ONE-PLANE LATERAL INSTABILITY

Relevant Special Tests

Mechanism of Injury

The LCL/FCL functions to control varus loading and lateral rotation of the tibia running from the femoral condyle to the head of the fibula. Contact injuries involve a direct varus load to the knee; this is the usual mechanism in a complete tear. The most common method of injury is a direct varus force with the foot plantar flexed and the knee in extension. Related injuries include injuries to the peroneal nerve, posterolateral capsule damage, or posterior cruciate ligament damage. The mechanism of knee adduction, flexion, and lateral rotation of the femur on the tibia is a much less common mechanism.

With excessive force, the LCL/FCL usually is disrupted initially, followed by the capsular ligaments, the arcuate ligament complex, the popliteus, the iliotibial band, the biceps femoris, and the common peroneal nerve; one or both cruciate ligaments may be disrupted.

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