Clinical Assessment of the Cardiopulmonary System

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Clinical Assessment of the Cardiopulmonary System

Clinical Assessment of the Cardiopulmonary System Requires a Number of Specific Steps.

II Chart Review

III The Patient Interview: Medical History

The interview provides unique information because it is the patients’ prospective on their illness.

The interview is designed to accomplish three related goals:

Effective patient interviewing requires that the clinician pay attention to a number of professional issues as listed in Box 18-1.

BOX 18-1   Guidelines for Effective Patient Interviewing

1. Project a sense of undivided interest in the patient:

2. Establish your professional role during the introduction.

3. Show your respect for the patient’s beliefs, attitudes, and rights:

• Be sure the patient is appropriately covered.

• Position yourself so that eye contact is comfortable for the patient. (Ideally, patients should be sitting up, with their eye level at or slightly above yours).

• Avoid standing at the foot of the bed or with your hand on the door because this may send the nonverbal message that you do not have time for the patient.

• Ask the patient’s permission before moving any personal items or making adjustments in the room.

• Remember that the patient’s dialog with you and his or her medical records are confidential.

• Be honest; never guess at an answer or provide information that you do not know. Do not provide information beyond your scope of practice; providing new information to the patient is the privilege and responsibility of the attending physician.

• Make no moral judgments about the patient; set your values for patient care according to the patient’s values, beliefs, and priorities.

• Expect the patient to have an emotional response to illness and the health care environment.

• Listen, then clarify and teach, but never argue.

• Adjust the time, length, and content of the interview to your patient’s needs.

4. Use a relaxed, conversational style:

From Wilkins RL et al: Egan’s Fundamentals of Respiratory Care, ed 8. St. Louis, Mosby, 2003.

Questions during the interview can be structured in a number of different ways depending on the type of response the clinician desires. Table 18-1 lists a number of different types of questions.

TABLE 18-1

Types of Questions in Taking a Medical History

Question Type Description Comments Examples
Open-ended Broad, general question about patient’s symptom or illness Allows patients to give history spontaneously without bias or influence from interviewer; patients direct discussion to whatever they want to cover first; can provide greatest amount of information; should generally be used first in interview “Tell me about your shortness of breath.”
“What brings you to the clinic today?”
Focused Interviewer defines area of inquiry more than in open-ended question or statement Directs discussion into more specific area but still gives patients latitude in answering “What treatment have you had for this condition in the past?”
“What are the physical requirements of your job?”
Closed-ended More specific question, which can generally be answered yes or no or by giving objective data such as dates, names, or numbers Best way to obtain specific data but limits scope of information by restricting patients to individual items requested “Have you ever had tuberculosis?”
“How may puffs from your inhaler do you use in a given day?”
Compound Two or more separate questions asked at once, without giving patients chance to respond to them individually May confuse patients; prevents patients from giving answers to all components; induces patients to focus on last question in series; should not be used “Tell me about yourself—how old are you, where do you live, and what do you do for a living?”
“Have you ever smoked cigarettes, used drugs, worked with asbestos, or been exposed to tuberculosis?”
Leading Interviewer phrases questions so as to lead patient in a particular direction in answering Reflects interviewer’s bias; tends to produce inaccurate, unreliable answer; should not be used “You’re feeling better today, aren’t you?”
“You’ve never used drugs, have you?”

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From Pierson DJ, Kacmarek RM: Foundations of Respiratory Care. New York, Churchill Livingstone, 1992. Churchill Livingstone

Occupational or environmental exposures are a key aspect of a patient interview. Table 18-2 lists a number of common exposures associated with pulmonary disease.

TABLE 18-2

Common Occupational or Environmental Exposures Associated with Pulmonary Disease

Occupation or Activity Exposure Disease
Asbestos mining/milling/manufacture; pipe fitting; shipbuilding/ship fitting; insulation, construction, demolition, living with someone employed in any of the above Asbestos Lung cancer, asbestosis, malignant mesothelioma, nonmalignant inflammatory pleural effusion
Hard-rock mining, quarrying, stone cutting, abrasive industries, foundry work, sandblasting Crystalline quartz (silica) Silicosis
Coal mining Coal dust Coal workers’ pneumoconiosis
Farming, grain handling Grain dust Chronic bronchitis, chronic obstructive pulmonary disease
Farming, animal attendants Moldy hay (spores of thermophilic actinomycetes [fungus]) Hypersensitivity pneumonitis (farmer’s lung)
Cotton/flax/hemp workers, textile industry Cotton dust Byssinosis
Pigeon breeding, bird handling Proteins derived from parakeets, budgerigars, pigeons, chickens, turkeys (avian droppings or feathers) Hypersensitivity pneumonitis (e.g., pigeon-breeders’ lung), bird-fanciers’ lung
Woodworking, lumber industry Wood dust, Alternaria (fungus), Western red cedar, oak, others Hypersensitivity pneumonitis, woodworker’s lung, occupational asthma

From Pierson DJ, Kacmarek RM: Foundations of Respiratory Care. New York, Churchill Livingstone, 1992. Churchill Livingstone

Symptoms expressed as a concern by patients should be explored in detail. Box 18-2 lists specific questions that should be asked regarding a specific symptom.

BOX 18-2   What, Where, When, and How: Ten Questions to Ask a Patient About a Pain or Other Symptom

1. What does it feel like? Have the patient use own words in describing the character of the symptoms (e.g., dyspnea or pain).

2. Where is it? If the symptom is a pain, ask the patient to localize it as precisely as possible.

3. Where else does it go? Inquire about radiation of the pain to other parts of the body.

4. How bad is it? If possible, have the patient quantitate the symptom (e.g., by using a hypothetical scale of 1 of 10, with 10 being the worst discomfort ever experienced).

5. How long does it last? Again quantitate as much as possible. Does the pain come and go or is it constant? Is it always the same?

6. When does it occur? Ask about associations with time of day, physical activity, body position, emotion or stress, and any relationship to eating or drinking.

7. What brings it on or makes it worse? What would the patient do who wanted to bring on the pain or make it worse?

8. What relieves it or makes it better? Ask about any medication or activity that the patient has noted improves the symptom.

9. How does it affect you? What activities are prevented or limited by the symptom? Quantitate if possible. Ask the patient to compare present limitations with past performance or present capabilities with those of peers.

10. What else is associated with it? Inquire about other phenomena (e.g., fever, diaphoresis, dyspnea) that occur with the symptom in question.

From Pierson DJ, Kacmarek RM: Foundations of Respiratory Care. New York, Churchill Livingstone, 1992. Churchill Livingstone

All interviews of patients receiving respiratory care should focus on the following symptoms and signs.

1. Cough

2. Sputum production

3. Hemoptysis

4. Pedal edema

5. Jugular vein distention

6. Dyspnea

7. Chest pain

8. Cyanosis

9. Clubbing

10. Stridor

11. Wheezing

12. Level of consciousness

a. Is the patient oriented to person, place, and time?

b. Box 18-3 lists the terms with their definition of various levels of consciousness.

c. If the patient is falling asleep during the interview, his or her CO2 level may be elevated.

IV Vital Signs

Temperature: An indicator of metabolic rate.

1. Normal temperature

2. Hyperthermia (fever): In the hospitalized patient it is normally indicative of infection, which is usually bacterial but may be viral.

3. Hypothermia is rare in hospitalized patients but may be a result of:

4. Hypothermia decreases O2 consumption and CO2 production.

Pulse: A general reflection of pump capabilities of the heart.

1. Heart rate in adults is normally 60 to 100 beats/min.

2. Rhythm: A regular rhythm should be noted. Irregular rhythm may indicate:

3. Strength: The force of the beat should be easily noted.

Blood pressure: The force exerted by the arterial pressure against the wall of the artery.

1. Systolic pressure is normally 95 to 139 mm Hg (between 130 and 139 mm Hg currently referred to as prehypertension) in the adult.

2. Diastolic pressure is normally 60 to 89 mm Hg in the adult.

3. Pulse pressure is the difference between the systolic and diastolic pressure, normally 35 to 40 mm Hg. If it is <25 to 30 mm Hg, the peripheral pulse is difficult to palpate. This pressure provides the gradient for peripheral perfusion.

4. Hypertension is a pressure ≥140/90 mm Hg and may be reflective of:

5. Prehypertension, a systolic pressure of 130 to 140 mm Hg, is now considered a precursor to hypertension and requires a change in lifestyle and in some cases treatment.

6. Hypotension is a pressure <95/60 mm Hg and may be reflective of:

Respiratory rate

Physical Assessment of the Chest

Chest assessment includes the following (sequentially performed as listed):

Inspection is the observation of the patient’s chest configuration and pattern of breathing. During inspection, the following should be evaluated:

1. Position

2. Chest configuration

3. Ventilatory pattern

a. Sequence of normal lung expansion

b. Uniform bilateral chest expansion

c. Use of accessory muscles of ventilation

d. Acute cardiopulmonary stress normally results in an increased ventilatory rate.

e. Patients with chronic obstructive lung disease may have a decreased ventilatory rate, whereas patients with chronic restrictive lung disease may have an increased ventilatory rate.

f. Pursed lipped breathing is indicative of chronic airway obstruction.

g. Inspiratory/expiratory ratios should be about 1:2.

h. The presence of audible wheezes, crackles, or rhonchi is indicative of secretions or bronchospasm.

Palpation is the touching of the chest to evaluate movement and underlying lung function.

1. Symmetric movement of the thoracic cage.

2. Tone of ventilatory muscles.

3. Presence of consolidation, pneumothorax, atelectasis, or pleural effusion. These may cause a shift in the mediastinum. Palpation of the trachea at the suprasternal notch identifies shifting.

4. Fremitus: The vibration produced over the thoracic cage by the conduction of sound waves.

5. Subcutaneous emphysema: If it is present, an air leak has allowed gas to enter the tissue.

Percussion is the production of audible and tactile vibrations over the chest by tapping the chest wall (Figure 18-1).

Auscultation is the evaluation of breath sounds with a stethoscope (see Figure 18-2).

1. Normal breath sounds (Figure 18-3)

2. Adventitious or abnormal breath sounds (Tables 18-3 and 18-4)

TABLE 18-3

Recommended Terminology for Lung Sounds Versus Terminology in Other Publications

Recommended Term Classification Terms Used in Other Publications
Crackles Discontinuous Rales
    Crepitations
Wheezes High-pitched, continuous Sibilant rales
    Musical rales
    Sibilant rhonchi
  Low-pitched, continuous Rhonchi
    Sonorous rales

From Wilkins RL, Krier SJ, Sheldon RL: Clinical Assessment in Respiratory Care, ed 4. St. Louis, Mosby, 2000.

TABLE 18-4

Application of Adventitious Lung Sounds

Lung Sounds Possible Mechanism Characteristics Causes
Wheezes Rapid airflow through obstructed airways caused by bronchospasm, mucosal edema High-pitched most often occur during exhalation Asthma, congestive heart failure, bronchitis
Stridor Rapid airflow through obstructed airway caused by inflammation High pitched, often occurs during inhalation Croup, epiglottitis, bronchitis
Crackles
 Inspiratory and expiratory Excess airway secretions moving with airflow Coarse and often clear with cough Bronchitis, respiratory infections
 Early inspiratory Sudden opening of proximal bronchi Scanty, transmitted to mouth, not affected by cough Bronchitis, emphysema, asthma
 Late inspiratory Sudden opening of peripheral airways Diffuse, fine, occur initially in the dependent regions Atelectasis, pneumonia, pulmonary edema, fibrosis

image

From Wilkins RL, Krider SJ, Sheldon RL: Clinical Assessment in Respiratory Care, ed 4. St. Louis, Mosby, 2000.

a. Crackles (rales): A discontinuous sound (<20 msec) that is perceived as a wet, crackling, bubbling sound associated with gas moving through liquid. Normally they are heard during:

b. Rhonchi: A continuous sound (>25 msec) that is low in pitch and normally indicative of secretions in large airways. In patients who can successfully mobilize their own secretions, rhonchi clear with coughing.

c. Wheezes: A continuous sound (>25 msec) that is high pitched and normally indicative of bronchospasm or mucosal edema in medium to larger airways. Wheezes do not clear with coughing.

d. Pleural friction rub: A creaking or grating sound as a result of inflamed pleural surfaces rubbing together during breathing.

VI Work of Breathing

During normal breathing the muscles of ventilation consume 5% to 10% of the total oxygen consumed to perform the work of breathing.

The effort required to perform the work of breathing depends on the following:

Normally the ventilatory pattern a patient assumes is that which requires the least work.

1. Figure 18-4, A, relates the components of the work of breathing to the respiratory rate and tidal volume.

2. Resistance work refers to the amount of work necessary to overcome nonelastic resistance to ventilation. If nonelastic resistance were the only force opposing ventilation, the ideal ventilatory pattern would be a slow rate and a large tidal volume.

3. Elastic work refers to the amount of work necessary to overcome elastic resistance. If elastic resistance were the only force opposing ventilation, a rapid ventilatory rate with a small tidal volume would be ideal.

4. Total work refers to actual work expended with varying ventilatory rates and tidal volumes. Note the ideal rate is approximately 12 to 18 breaths/min with an ideal tidal volume of approximately 6 ml/kg predicted body weight.

5. Figure 18-4, B, illustrates the effect an increase in elastic work has on ventilatory pattern.

6. If resistance work were increased, the total work curve in Figure 18-4, A, would shift to the left.

7. Refer to Chapter 5 for quantification of work of breathing.

VII Ventilatory Reserve

Ventilatory reserve is the ability of the organism to respond to increased levels of cardiopulmonary stress.

During normal breathing, the efficiency of the ventilatory muscles is poor. Approximately 90% of the oxygen consumed to perform the work of breathing is lost as heat.

The efficiency of the ventilatory muscles is further reduced with chronic pulmonary disease and with an increased minute ventilation.

Figure 18-5, A, depicts the relationship between minute volume and percentage of oxygen consumed for breathing in patients without chronic pulmonary disease (solid line) and patients with chronic pulmonary disease (dotted line).

Figure 18-5, B, illustrates the relationship between percentage of oxygen consumed for breathing and the percentage of the vital capacity (VC) that is the tidal volume.

VIII Vital Capacity/Maximum Inspiratory Pressure

A normal VC is approximately 70 to 90 ml/kg of ideal body weight.

VCs are frequently used as an estimate of the patient’s ventilatory reserve.

If the VC is >15 ml/kg of ideal body weight, it is assumed that the individual has the capability to respond to increased levels of cardiopulmonary stress.

However, if the VC is <10 ml/kg, prolonged sustained spontaneous ventilation is questionable. This individual has virtually no reserves.

At VCs between 10 and 15 ml/kg, reserves are marginal, and appropriate monitoring should be instituted.

Maximum inspiratory pressure (MIP) is also a parameter used to assess ventilatory reserves.

If the MIP is more negative than -30 cm H2O in a 20-second period and the patient has normal lungs and is recovering from anesthesia, neuromuscular or neurologic disease, or an overdose, sustained spontaneous ventilation is probable.

In patients with chronic pulmonary disease or acute respiratory failure associated with multiorgan system failure, the use of a -30 cm H2O range for MIP becomes less reliable.

During the measurement of MIP, the diaphragm must be at its resting level for maximum performance. Thus the greater the length of the diaphragmatic muscle fibers (smaller lung volume), the greater their contractile force.

IX Rapid Shallow Breathing Index

Normally the relationship between respiratory rate and tidal volume in liters is approximately 20 to 60 in the average adult.

This relationship has been used to determine whether a patient is ready to be discontinued from ventilatory support.

In critically ill patients a rapid shallow breathing index (RSBI) <105 indicates a high probability of being able to breathe unassisted.

An RSBI ≥105 indicates ventilatory support is still required.

It is critical that the RSBI be determined the same way it was studied if it is to be maximally predictive.

Assessment of Peripheral Perfusion

Adequacy of peripheral perfusion can be estimated by:

Sensorium

Urinary output

Capillary refill decreases as peripheral perfusion decreases.

Skin turgor also decreases as peripheral perfusion decreases.

Cyanosis

Thready, faint, or distant peripheral pulses are noted as peripheral perfusion decreases.

XI Physical Signs Associated with Common Abnormal Pulmonary Pathology (Table 18-5)

TABLE 18-5

Physical Signs of Pulmonary Abnormalities

Abnormality Initial Impression Inspection Palpation Percussion Auscultation Possible Causes
Acute airway obstruction Appears acutely ill Use of accessory muscles Reduced expansion Increased resonance Expiratory wheezing Asthma, bronchitis
Chronic airway obstruction Appears chronically ill Increased anteroposterior diameter, use of accessory muscles Reduced expansion Increased resonance Diffuse reduction in breath sounds, early inspiratory crackles Chronic bronchitis, emphysema
Consolidation May appear acutely ill Inspiratory lag Increased fremitus Dull note Bronchial breath sounds, crackles Pneumonia, tumor
Pneumothorax May appear acutely ill Unilateral expansion Decreased fremitus Increased resonance Absent breath sounds Rib fracture, open wound
Pleural effusion May appear acutely ill Unilateral expansion Absent fremitus Dull note Absent breath sounds Congestive heart failure
Left bronchial obstruction Appears acutely ill Unilateral expansion Absent fremitus Dull note Absent breath sounds Mucous plug
Diffuse interstitial fibrosis Often normal Rapid shallow breathing Often normal, increased fremitus Slight decrease in resonance Late inspiratory crackles Chronic exposure to inorganic dust
Acute upper airway obstruction Appears acutely ill Labored breathing Often normal Often normal Inspiratory/expiratory stridor Epiglottitis, croup, foreign body aspiration

image

From Wilkins RL, Krider SJ, Sheldon RL: Clinical Assessment in Respiratory Care, ed 4. St. Louis, Mosby, 2000.

XII Laboratory Assessment in Cardiopulmonary Disease

Hematology

1. Complete red blood cell count (CBC)

2. Anemia: A below-normal quantity of hemoglobin, RBC count, or hematocrit and greatly decreases oxygen-carrying capacity. It may be a result of:

3. Polycythemia: An increase in hemoglobin, RBC count, or hematocrit. It may result from:

4. Abnormalities in WBC count and their differential are a result of infection, allergic reaction, or leukemia.

a. An increase in overall WBC count (i.e., leukocytosis) is normally noted in bacterial infections.

b. A decrease in overall WBC count (i.e., leukopenia) is normally noted in leukemia, radiation therapy, and chemotherapy.

c. Neutrophilia (increased neutrophils): A common response to stress and the body’s first response to:

d. A leftward shift of the neutrophils: Increased levels of bands (immature neutrophils) as a result of stress. The greater the stress, the greater the percent bands.

e. Eosinophilia: An increase in the number of eosinophils, usually a result of:

f. Lymphocytosis: An increase in the number of lymphocytes, usually a result of a viral infection.

g. Monocytosis: An increase in the number of monocytes, usually a result of:

h. Basophilia: An increase in the number of basophiles usually seen in:

5. Platelet count

Coagulation studies

1. Four distinct tests generally are used to evaluate the tendency of blood to clot.

2. Bleeding time: Evaluates the ability of small skin vessels to constrict and evaluates the function of platelets. Normal time is up to 6 minutes.

3. APTT: Evaluates the amount of time it takes plasma to form a fibrin clot once the body’s intrinsic clotting pathways are activated. Normal time is 32 to 51 seconds.

4. PT: Evaluates the amount of time it takes extrinsic blood factors to form a clot once activated. Normal PT is 12 to 15 seconds.

5. All times are lengthened during hemophilia.

6. APTT: Monitored during heparin therapy.

7. PT: Monitored during warfarin sodium (Coumadin) therapy.

Electrolytes (see Chapter 14)

Blood urea nitrogen and creatinine levels and urinalysis (see Chapter 13).

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