Gastric and esophageal, ± duodenal submucosal edema
Best evaluated by CECT
Caustic agents cause intense pylorospasm, so duodenal injury is less common
• Acute: Severe
Pneumoperitoneum (perforation)
• Chronic phase
Gastric outlet obstruction (antral scarring and fibrosis)
Linitis plastica: Small, aperistaltic stomach with effaced folds
Best evaluated by upper GI series
TOP DIFFERENTIAL DIAGNOSES
• Gastric carcinoma (scirrhous type)
• Gastric metastases and lymphoma
• Gastric thermal injury (iced saline)
CLINICAL ISSUES
• Prognosis
Acute mild phase with early treatment: Good
Acute severe and chronic phases: Poor
• Treatment
Conservative treatment for stable patients
– Antibiotics, steroids, parenteral feedings
Gastric outlet obstruction
– Gastroenterostomy or partial gastrectomy
– Complete gastrectomy with colonic interposition
(Left) Axial NECT shows tremendous thickening of the wall of the stomach and ascites. These findings suggest transmural inflammation and a high likelihood of subsequent necrosis and perforation of the stomach.
(Right) Axial NECT of the same patient shows diffuse involvement of the entire stomach . The patient subsequently had gastric perforation and died.
(Left) Upright film from an upper GI series in a 24-year-old man who drank drain cleaner several days prior shows delayed gastric emptying with fluid levels . Note the fixed contraction (linitis plastica) of the body and antrum of the stomach .
(Right) Film from an upper GI series in a 56-year-old man who drank acid several weeks prior demonstrates a linitis plastica appearance of the stomach being small, rigid, nondistensible, and without peristalsis.
Upper GI series shows an extremely small, nondistensible, distorted stomach due to the ingestion of hydrochloric acid.