Acute abdominal pain

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Chapter 7 ACUTE ABDOMINAL PAIN

CAUSES OF ACUTE ABDOMINAL PAIN

Generalised abdominal pain

Constant abdominal pain

In a patient with generalised constant abdominal pain and abdominal rigidity, the working diagnosis is peritonitis. This is always secondary to some other process. Often, but not always, this is due to a perforated viscus. The role of surgery is to provide peritoneal toilet and to prevent ongoing contamination by removing or repairing the perforated viscus. There are a number of conditions that fulfill this criterion. There are other conditions, however, that may demonstrate the signs of peritonitis but not require intervention (see Figure 7.1). Typically acute pancreatitis with generalised inflammation may have tenderness and guarding but does not require a laparotomy. A diagnostic elevated lipase may be very useful in this setting. In females, pelvic inflammatory disease may show generalised signs and does not benefit from laparotomy as there is no ongoing contamination. This should be considered in appropriate clinical contexts. Spontaneous bacterial peritonitis in portal hypertension should also be considered in patients presenting with generalised abdominal pain and portal hypertension with ascites.

Beware of the patient complaining of severe constant generalised abdominal pain who appears to have a relatively soft ‘doughy’ abdomen to palpation. This patient may have ischaemic bowel. Suspicion may be raised if they have bloody diarrhoea and atrial fibrillation. Investigations may reveal an elevated white cell count and a metabolic acidosis. Venous obstruction is often more subtle and slowly progressive.

Colicky abdominal pain

Colicky abdominal pain is episodic pain that comes in waves every 4–5 minutes. It is crescendo decrescendo in nature. When it is generalised the diagnosis is usually between gastroenteritis or a bowel obstruction. Gastroenteritis will often be associated with early vomiting and diarrhoea and abdominal distension will not be a feature. Some infective forms of gastroenteritis may not produce diarrhoea.

In contrast, a bowel obstruction will usually be associated with vomiting and constipation with progressive abdominal distension. The higher the obstruction is anatomically in the intestine, the earlier the vomiting and the later the constipation. The more distal the obstructing lesion, the greater the distension. A supine and erect plain abdominal film will usually confirm the diagnosis. An erect chest X-ray should always be ordered to look for free peritoneal gas. The supine abdominal film is the most useful. With the patient lying flat it will not show the fluid levels of the erect film but it will show gaseous distended bowel down to the level of obstruction. The ileum or small intestine can be distinguished by its central position and valvulae conniventes extending across the intestinal lumen. The jejunum or large intestine is distinguished by its incomplete valves and more peripheral position.

Be very wary of the diagnosis of constipation causing abdominal pain if the rectum is empty or if abdominal tenderness is present.

Surgery is indicated for an obstructed bowel when the cause for the obstruction is unlikely to resolve. The timing is a balance between appropriate resuscitation of the patient and avoiding the development of gut ischaemia.

Small intestine obstruction is more difficult to manage than colonic obstruction as there are no objective diagnostic indicators of early bowel ischaemia. Colonic ischaemia can be predicted if the bowel lumen is distended above a 10 cm diameter, as venous drainage in the bowel wall will be impaired at this level. The caecum is the part of the large intestine usually most susceptible to distension and can be monitored clinically and radiologically.

If constant abdominal pain, worsening tenderness, fever, leucocytosis and acidosis are present, gut ischaemia should be suspected and urgent surgery is indicted.

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